Glutaric acid (BioDeep_00000001619)

 

Secondary id: BioDeep_00000400407, BioDeep_00000415752

natural product human metabolite PANOMIX_OTCML-2023 Endogenous blood metabolite BioNovoGene_Lab2019


代谢物信息卡片


1,3-Propanedicarboxylic acid

化学式: C5H8O4 (132.0423)
中文名称: 戊二酸
谱图信息: 最多检出来源 Homo sapiens(plant) 13.51%

Reviewed

Last reviewed on 2024-07-12.

Cite this Page

Glutaric acid. BioDeep Database v3. PANOMIX ltd, a top metabolomics service provider from China. https://query.biodeep.cn/s/glutaric_acid (retrieved 2024-12-22) (BioDeep RN: BioDeep_00000001619). Licensed under the Attribution-Noncommercial 4.0 International License (CC BY-NC 4.0).

分子结构信息

SMILES: C(CC(=O)O)CC(=O)O
InChI: InChI=1S/C5H8O4/c6-4(7)2-1-3-5(8)9/h1-3H2,(H,6,7)(H,8,9)

描述信息

Glutaric acid is a simple five-carbon linear dicarboxylic acid. Glutaric acid is naturally produced in the body during the metabolism of some amino acids, including lysine and tryptophan. Glutaric acid may cause irritation to the skin and eyes. When present in sufficiently high levels, glutaric acid can act as an acidogen and a metabotoxin. An acidogen is an acidic compound that induces acidosis, which has multiple adverse effects on many organ systems. A metabotoxin is an endogenously produced metabolite that causes adverse health effects at chronically high levels. Chronically high levels of glutaric acid are associated with at least three inborn errors of metabolism, including glutaric aciduria type I, malonyl-CoA decarboxylase deficiency, and glutaric aciduria type III. Glutaric aciduria type I (glutaric acidemia type I, glutaryl-CoA dehydrogenase deficiency, GA1, or GAT1) is an inherited disorder in which the body is unable to completely break down the amino acids lysine, hydroxylysine, and tryptophan due to a deficiency of mitochondrial glutaryl-CoA dehydrogenase (EC 1.3.99.7, GCDH). Excessive levels of their intermediate breakdown products (e.g. glutaric acid, glutaryl-CoA, 3-hydroxyglutaric acid, glutaconic acid) can accumulate and cause damage to the brain (and also other organs). Babies with glutaric acidemia type I are often born with unusually large heads (macrocephaly). Macrocephaly is amongst the earliest signs of GA1. GA1 also causes secondary carnitine deficiency because glutaric acid, like other organic acids, is detoxified by carnitine. Abnormally high levels of organic acids in the blood (organic acidemia), urine (organic aciduria), the brain, and other tissues lead to general metabolic acidosis. Acidosis typically occurs when arterial pH falls below 7.35. In infants with acidosis, the initial symptoms include poor feeding, vomiting, loss of appetite, weak muscle tone (hypotonia), and lack of energy (lethargy). These can progress to heart, liver, and kidney abnormalities, seizures, coma, and possibly death. These are also the characteristic symptoms of untreated glutaric aciduria. Many affected children with organic acidemias experience intellectual disability or delayed development. In adults, acidosis or acidemia is characterized by headaches, confusion, feeling tired, tremors, sleepiness, and seizures. Treatment of glutaric aciduria is mainly based on the restriction of lysine intake, supplementation of carnitine, and an intensification of therapy during intercurrent illnesses. The major principle of dietary treatment is to reduce the production of glutaric acid and 3-hydroxyglutaric acid by restriction of natural protein, in general, and of lysine, in particular (PMID: 17465389, 15505398). Glutaric acid has also been found in Escherichia (PMID: 30143200).
Isolated from basidiomycete fungi and fruits of Prunus cerasus (CCD). Glutaric acid is found in many foods, some of which are red beetroot, common beet, soy bean, and tamarind.

Glutaric acid, C5 dicarboxylic acid, is an intermediate during the catabolic pathways of lysine and tryptophan. Glutaric acid affects pericyte contractility and migration. Glutaric acid is an indicator of glutaric aciduria type I[1][2][3].
Glutaric acid, C5 dicarboxylic acid, is an intermediate during the catabolic pathways of lysine and tryptophan. Glutaric acid affects pericyte contractility and migration. Glutaric acid is an indicator of glutaric aciduria type I[1][2][3].

同义名列表

14 个代谢物同义名

1,3-Propanedicarboxylic acid; 1,3-Propanedicarboxylate; 1,5-Pentanedioic acid; 1,5-Pentanedioate; Pentanedioic acid; Pentandioic acid; GLUTARIC ACID; Pentanedioate; Pentandioate; Glutarsaeure; Glutarate; Glutaric acid; Glutaric acid; Glutarate



数据库引用编号

36 个数据库交叉引用编号

分类词条

相关代谢途径

Reactome(0)

BioCyc(0)

PlantCyc(0)

代谢反应

2 个相关的代谢反应过程信息。

Reactome(0)

BioCyc(0)

WikiPathways(1)

Plant Reactome(0)

INOH(0)

PlantCyc(0)

COVID-19 Disease Map(0)

PathBank(1)

  • Lysine Metabolism: Hydrogen Ion + meso-diaminopimelate ⟶ Carbon dioxide + L-Lysine

PharmGKB(0)

41 个相关的物种来源信息

在这里通过桑基图来展示出与当前的这个代谢物在我们的BioDeep知识库中具有相关联信息的其他代谢物。在这里进行关联的信息来源主要有：

  • PubMed: 来源于PubMed文献库中的文献信息，我们通过自然语言数据挖掘得到的在同一篇文献中被同时提及的相关代谢物列表，这个列表按照代谢物同时出现的文献数量降序排序，取前10个代谢物作为相关研究中关联性很高的代谢物集合展示在桑基图中。
  • NCBI Taxonomy: 通过文献数据挖掘，得到的代谢物物种来源信息关联。这个关联信息同样按照出现的次数降序排序，取前10个代谢物作为高关联度的代谢物集合展示在桑吉图上。
  • Chemical Taxonomy: 在物质分类上处于同一个分类集合中的其他代谢物
  • Chemical Reaction: 在化学反应过程中，存在为当前代谢物相关联的生化反应过程中的反应底物或者反应产物的关联代谢物信息。

点击图上的相关代谢物的名称，可以跳转到相关代谢物的信息页面。

亚细胞结构定位 关联基因列表
Cytoplasm 6 ANXA5, CAT, FOLH1, GFAP, GLUL, RBFOX3
Peripheral membrane protein 2 ANXA5, HSD17B6
Nucleus 2 GLUL, RBFOX3
cytosol 7 ANXA5, CAT, GFAP, GLUL, LIPE, PRKCQ, SLC22A7
Cell membrane 5 FOLH1, GLUL, LIPE, SLC22A11, SLC22A7
Lipid-anchor 1 GLUL
Early endosome membrane 1 HSD17B6
Multi-pass membrane protein 6 ABCC3, SLC13A2, SLC22A11, SLC22A6, SLC22A7, SLC22A8
Synapse 1 ALDH5A1
cell surface 1 FOLH1
Golgi membrane 1 INS
sarcolemma 1 ANXA5
Cytoplasm, cytosol 2 GLUL, LIPE
plasma membrane 9 ABCC3, FOLH1, GLUL, PRKCQ, SLC13A2, SLC22A11, SLC22A6, SLC22A7, SLC22A8
Membrane 9 ABCC3, ANXA5, CAT, FOLH1, LIPE, SLC13A2, SLC22A11, SLC22A6, SLC22A7
apical plasma membrane 4 SLC13A2, SLC22A11, SLC22A7, SLC22A8
basolateral plasma membrane 4 ABCC3, SLC22A6, SLC22A7, SLC22A8
caveola 2 LIPE, SLC22A6
extracellular exosome 8 ANXA5, CAT, FOLH1, GLUL, SLC13A2, SLC22A11, SLC22A6, SLC22A8
Lumenal side 1 HSD17B6
endoplasmic reticulum 2 GLUL, HSD17B6
extracellular space 1 INS
mitochondrion 6 ABAT, ALDH5A1, CAT, GCDH, GLUL, SUGCT
protein-containing complex 2 CAT, SLC22A6
intracellular membrane-bounded organelle 2 CAT, HSD17B6
Microsome membrane 1 HSD17B6
Secreted 1 INS
extracellular region 3 ANXA5, CAT, INS
astrocyte end-foot 1 GFAP
Mitochondrion matrix 1 GCDH
mitochondrial matrix 4 ABAT, ALDH5A1, CAT, GCDH
centriolar satellite 1 PRKCQ
external side of plasma membrane 2 ANXA5, SLC22A11
perikaryon 1 RBFOX3
Single-pass type II membrane protein 1 FOLH1
Apical cell membrane 3 SLC13A2, SLC22A11, SLC22A7
focal adhesion 2 ANXA5, CAT
Peroxisome 1 CAT
Peroxisome matrix 1 CAT
peroxisomal matrix 1 CAT
peroxisomal membrane 1 CAT
collagen-containing extracellular matrix 1 ANXA5
intermediate filament 1 GFAP
lateral plasma membrane 1 SLC22A8
Zymogen granule membrane 1 ANXA5
cell projection 1 GFAP
Basolateral cell membrane 4 ABCC3, SLC22A6, SLC22A7, SLC22A8
endosome lumen 1 INS
Lipid droplet 1 LIPE
Membrane, caveola 1 LIPE
cell body 2 GFAP, GLUL
intermediate filament cytoskeleton 1 GFAP
basal plasma membrane 4 ABCC3, SLC22A11, SLC22A6, SLC22A7
Microsome 1 GLUL
ficolin-1-rich granule lumen 1 CAT
secretory granule lumen 2 CAT, INS
Golgi lumen 1 INS
endoplasmic reticulum lumen 1 INS
transport vesicle 1 INS
Endoplasmic reticulum-Golgi intermediate compartment membrane 1 INS
immunological synapse 1 PRKCQ
aggresome 1 PRKCQ
vesicle membrane 1 ANXA5
Basal cell membrane 3 ABCC3, SLC22A11, SLC22A6
[Isoform 1]: Cytoplasm, cytosol 1 SLC22A7
cytoplasmic side of lysosomal membrane 1 GFAP
glial cell projection 1 GLUL
catalase complex 1 CAT
endothelial microparticle 1 ANXA5
4-aminobutyrate transaminase complex 1 ABAT
[Isoform PSMA']: Cytoplasm 1 FOLH1
[Isoform 2]: Basolateral cell membrane 1 SLC22A7


文献列表

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