Exact Mass: 563.3012
Exact Mass Matches: 563.3012
Found 424 metabolites which its exact mass value is equals to given mass value 563.3012
,
within given mass tolerance error 0.05 dalton. Try search metabolite list with more accurate mass tolerance error
0.01 dalton.
Taurolithocholate 3-sulfate
Taurolithocholic acid 3-sulfate is a sulfated bile acid. Under normal circumstances, bile acid sulfation is a minor pathway. However in the presence of cholestasis, the fraction of the bile acid pool which is sulfated increases. Sulfation of bile acids increases the aqueous solubility of the amphipathic compounds and results in more efficient renal clearance as well as in decreased reabsorption from the intestinal lumen. Bile acids are steroid acids found predominantly in the bile of mammals. The distinction between different bile acids is minute, depending only on the presence or absence of hydroxyl groups on positions 3, 7, and 12. Bile acids are physiological detergents that facilitate excretion, absorption, and transport of fats and sterols in the intestine and liver. Bile acids are also steroidal amphipathic molecules derived from the catabolism of cholesterol. They modulate bile flow and lipid secretion, are essential for the absorption of dietary fats and vitamins, and have been implicated in the regulation of all the key enzymes involved in cholesterol homeostasis. Bile acids recirculate through the liver, bile ducts, small intestine and portal vein to form an enterohepatic circuit. They exist as anions at physiological pH and, consequently, require a carrier for transport across the membranes of the enterohepatic tissues. The unique detergent properties of bile acids are essential for the digestion and intestinal absorption of hydrophobic nutrients. Bile acids have potent toxic properties (e.g. membrane disruption) and there are a plethora of mechanisms to limit their accumulation in blood and tissues (PMID: 11316487, 16037564, 12576301, 11907135). Taurolithocholic acid 3-sulfate is a sulfated bile acid. Under normal circumstances, bile acid sulfation is a minor pathway. However in the presence of cholestasis, the fraction of the bile acid pool which is sulfated increases. Sulfation of bile acids increases the aqueous solubility of the amphipathic compounds and results in more efficient renal clearance as well as in decreased reabsorption from the intestinal lumen. Bile acids are steroid acids found predominantly in bile of mammals. The distinction between different bile acids is minute, depends only on presence or absence of hydroxyl groups on positions 3, 7, and 12. Bile acids are physiological detergents that facilitate excretion, absorption, and transport of fats and sterols in the intestine and liver. Bile acids are also steroidal amphipathic molecules derived from the catabolism of cholesterol. They modulate bile flow and lipid secretion, are essential for the absorption of dietary fats and vitamins, and have been implicated in the regulation of all the key enzymes involved in cholesterol homeostasis. Bile acids recirculate through the liver, bile ducts, small intestine and portal vein to form an enterohepatic circuit. They exist as anions at physiological pH and, consequently, require a carrier for transport across the membranes of the enterohepatic tissues. The unique detergent properties of bile acids are essential for the digestion and intestinal absorption of hydrophobic nutrients. Bile acids have potent toxic properties (e.g., membrane disruption) and there are a plethora of mechanisms to limit their accumulation in blood and tissues. (PMID: 11316487, 16037564, 12576301, 11907135) [HMDB] D005765 - Gastrointestinal Agents > D001647 - Bile Acids and Salts D005765 - Gastrointestinal Agents > D002793 - Cholic Acids KEIO_ID T072
Fosinopril
Fosinopril is a phosphinic acid-containing ester prodrug that belongs to the angiotensin-converting enzyme (ACE) inhibitor class of medications. It is rapidly hydrolyzed to fosinoprilat, its principle active metabolite. Fosinoprilat inhibits ACE, the enzyme responsible for the conversion of angiotensin I (ATI) to angiotensin II (ATII). ATII regulates blood pressure and is a key component of the renin-angiotensin-aldosterone system (RAAS). Fosinopril may be used to treat mild to moderate hypertension, as an adjunct in the treatment of congestive heart failure, and to slow the rate of progression of renal disease in hypertensive individuals with diabetes mellitus and microalbuminuria or overt nephropathy. C - Cardiovascular system > C09 - Agents acting on the renin-angiotensin system > C09A - Ace inhibitors, plain > C09AA - Ace inhibitors, plain D004791 - Enzyme Inhibitors > D011480 - Protease Inhibitors > D000806 - Angiotensin-Converting Enzyme Inhibitors D002317 - Cardiovascular Agents > D000959 - Antihypertensive Agents CONFIDENCE standard compound; EAWAG_UCHEM_ID 3324
Alpha-Hydroxy-tamoxifen-O-glucuronide
Alpha-Hydroxy-tamoxifen-O-glucuronide is a metabolite of tamoxifen. Tamoxifen is an antagonist of the estrogen receptor in breast tissue via its active metabolite, hydroxytamoxifen. In other tissues such as the endometrium, it behaves as an agonist, and thus may be characterized as a mixed agonist/antagonist. Tamoxifen is the usual endocrine therapy for hormone receptor-positive breast cancer in pre-menopausal women, and is also a standard in post-menopausal women although aromatase inhibitors are also frequently used in that setting. (Wikipedia)
4-Hydroxytamoxifen-O-glucuronide
4-Hydroxytamoxifen-O-glucuronide is a metabolite of tamoxifen. Tamoxifen is an antagonist of the estrogen receptor in breast tissue via its active metabolite, hydroxytamoxifen. In other tissues such as the endometrium, it behaves as an agonist, and thus may be characterized as a mixed agonist/antagonist. Tamoxifen is the usual endocrine therapy for hormone receptor-positive breast cancer in pre-menopausal women, and is also a standard in post-menopausal women although aromatase inhibitors are also frequently used in that setting. (Wikipedia)
(E)-3-Hydroxy Tamoxifen O-|A-D-Glucuronide
Dihydroergocornine
D018377 - Neurotransmitter Agents > D015259 - Dopamine Agents > D018491 - Dopamine Agonists
Fosenopril
Phe Ile Arg Glu
Phe Glu Ile Arg
Asn Arg Phe Lys
fosinopril
C - Cardiovascular system > C09 - Agents acting on the renin-angiotensin system > C09A - Ace inhibitors, plain > C09AA - Ace inhibitors, plain D004791 - Enzyme Inhibitors > D011480 - Protease Inhibitors > D000806 - Angiotensin-Converting Enzyme Inhibitors D002317 - Cardiovascular Agents > D000959 - Antihypertensive Agents CONFIDENCE standard compound; INTERNAL_ID 2247
Tamoxifen citrate
D006730 - Hormones, Hormone Substitutes, and Hormone Antagonists > D006727 - Hormone Antagonists > D020847 - Estrogen Receptor Modulators D006730 - Hormones, Hormone Substitutes, and Hormone Antagonists > D006727 - Hormone Antagonists > D004965 - Estrogen Antagonists C274 - Antineoplastic Agent > C163758 - Targeted Therapy Agent > C1821 - Selective Estrogen Receptor Modulator C471 - Enzyme Inhibitor > C1404 - Protein Kinase Inhibitor > C61074 - Serine/Threonine Kinase Inhibitor C274 - Antineoplastic Agent > C129818 - Antineoplastic Hormonal/Endocrine Agent > C481 - Antiestrogen C147908 - Hormone Therapy Agent > C548 - Therapeutic Hormone > C483 - Therapeutic Estrogen C147908 - Hormone Therapy Agent > C547 - Hormone Antagonist D050071 - Bone Density Conservation Agents D000970 - Antineoplastic Agents C1892 - Chemopreventive Agent
3alpha-Sulfatolithocholyltaurine
BA-149-150. In-source decay; 1 microL of the bile acid in MeOH solution was flow injected. Sampling interval was 1 Hz.; This record was created by the financial support of MEXT/JSPS KAKENHI Grant Number 17HP8021 (2017) to the MassBank database committee of the Mass Spectrometry Society of Japan. BA-149-120. In-source decay; 1 microL of the bile acid in MeOH solution was flow injected. Sampling interval was 1 Hz.; This record was created by the financial support of MEXT/JSPS KAKENHI Grant Number 17HP8021 (2017) to the MassBank database committee of the Mass Spectrometry Society of Japan. BA-149-90. In-source decay; 1 microL of the bile acid in MeOH solution was flow injected. Sampling interval was 1 Hz.; This record was created by the financial support of MEXT/JSPS KAKENHI Grant Number 17HP8021 (2017) to the MassBank database committee of the Mass Spectrometry Society of Japan. BA-149-60. In-source decay; 1 microL of the bile acid in MeOH solution was flow injected. Sampling interval was 1 Hz.; This record was created by the financial support of MEXT/JSPS KAKENHI Grant Number 17HP8021 (2017) to the MassBank database committee of the Mass Spectrometry Society of Japan. BA-149-30. In-source decay; 1 microL of the bile acid in MeOH solution was flow injected. Sampling interval was 1 Hz.; This record was created by the financial support of MEXT/JSPS KAKENHI Grant Number 17HP8021 (2017) to the MassBank database committee of the Mass Spectrometry Society of Japan.
Cys Lys Lys Trp
Cys Lys Gln Trp
Cys Lys Trp Lys
Cys Lys Trp Gln
Cys Gln Lys Trp
Cys Gln Trp Lys
Cys Trp Lys Lys
Cys Trp Lys Gln
Cys Trp Gln Lys
Asp His His Arg
Asp His Arg His
Asp Arg His His
Glu Phe Ile Arg
Glu Phe Leu Arg
Glu Phe Arg Ile
Glu Phe Arg Leu
Glu Ile Phe Arg
Glu Ile Arg Phe
Glu Leu Phe Arg
Glu Leu Arg Phe
Glu Pro Arg Tyr
Glu Pro Tyr Arg
Glu Arg Phe Ile
Glu Arg Phe Leu
Glu Arg Ile Phe
Glu Arg Leu Phe
Glu Arg Pro Tyr
Glu Arg Tyr Pro
Glu Tyr Pro Arg
Glu Tyr Arg Pro
Phe Glu Leu Arg
Phe Glu Arg Ile
Phe Glu Arg Leu
Phe Ile Glu Arg
Phe Ile Val Trp
Phe Ile Trp Val
Phe Lys Asn Arg
Phe Lys Arg Asn
Phe Leu Glu Arg
Phe Leu Arg Glu
Phe Leu Val Trp
Phe Leu Trp Val
Phe Asn Lys Arg
Phe Asn Gln Arg
Phe Asn Arg Lys
Phe Asn Arg Gln
Phe Gln Asn Arg
Phe Gln Arg Asn
Phe Arg Glu Ile
Phe Arg Glu Leu
Phe Arg Ile Glu
Phe Arg Lys Asn
Phe Arg Leu Glu
Phe Arg Asn Lys
Phe Arg Asn Gln
Phe Arg Gln Asn
Phe Val Ile Trp
Phe Val Leu Trp
Phe Val Trp Ile
Phe Val Trp Leu
Phe Trp Ile Val
Phe Trp Leu Val
Phe Trp Val Ile
Phe Trp Val Leu
His Asp His Arg
His Asp Arg His
His His Asp Arg
His His Arg Asp
His Arg Asp His
His Arg His Asp
Ile Glu Phe Arg
Ile Glu Arg Phe
Ile Phe Glu Arg
Ile Phe Arg Glu
Ile Phe Val Trp
Ile Phe Trp Val
Ile Ile Arg Tyr
Ile Ile Tyr Arg
Ile Leu Arg Tyr
Ile Leu Tyr Arg
Ile Arg Glu Phe
Ile Arg Phe Glu
Ile Arg Ile Tyr
Ile Arg Leu Tyr
Ile Arg Tyr Ile
Ile Arg Tyr Leu
Ile Val Phe Trp
Ile Val Trp Phe
Ile Trp Phe Val
Ile Trp Val Phe
Ile Tyr Ile Arg
Ile Tyr Leu Arg
Ile Tyr Arg Ile
Ile Tyr Arg Leu
Lys Cys Lys Trp
Lys Cys Gln Trp
Lys Cys Trp Lys
Lys Cys Trp Gln
Lys Phe Asn Arg
Lys Phe Arg Asn
Lys Lys Cys Trp
Lys Lys Trp Cys
Lys Asn Phe Arg
Lys Asn Arg Phe
Lys Gln Cys Trp
Lys Gln Trp Cys
Lys Arg Phe Asn
Lys Arg Asn Phe
Lys Trp Cys Lys
Lys Trp Cys Gln
Lys Trp Lys Cys
Lys Trp Gln Cys
Leu Glu Phe Arg
Leu Glu Arg Phe
Leu Phe Glu Arg
Leu Phe Arg Glu
Leu Phe Val Trp
Leu Phe Trp Val
Leu Ile Arg Tyr
Leu Ile Tyr Arg
Leu Leu Arg Tyr
Leu Leu Tyr Arg
Leu Arg Glu Phe
Leu Arg Phe Glu
Leu Arg Ile Tyr
Leu Arg Leu Tyr
Leu Arg Tyr Ile
Leu Arg Tyr Leu
Leu Val Phe Trp
Leu Val Trp Phe
Leu Trp Phe Val
Leu Trp Val Phe
Leu Tyr Ile Arg
Leu Tyr Leu Arg
Leu Tyr Arg Ile
Leu Tyr Arg Leu
Asn Phe Lys Arg
Asn Phe Gln Arg
Asn Phe Arg Lys
Asn Phe Arg Gln
Asn Lys Phe Arg
Asn Lys Arg Phe
Asn Gln Phe Arg
Asn Gln Arg Phe
Asn Arg Phe Gln
Asn Arg Lys Phe
Asn Arg Gln Phe
Pro Glu Arg Tyr
Pro Glu Tyr Arg
Pro Arg Glu Tyr
Pro Arg Tyr Glu
Pro Val Trp Tyr
Pro Val Tyr Trp
Pro Trp Val Tyr
Pro Trp Tyr Val
Pro Tyr Glu Arg
Pro Tyr Arg Glu
Pro Tyr Val Trp
Pro Tyr Trp Val
Gln Cys Lys Trp
Gln Cys Trp Lys
Gln Phe Asn Arg
Gln Phe Arg Asn
Gln Lys Cys Trp
Gln Lys Trp Cys
Gln Asn Phe Arg
Gln Asn Arg Phe
Gln Arg Phe Asn
Gln Arg Asn Phe
Gln Trp Cys Lys
Gln Trp Lys Cys
Arg Asp His His
Arg Glu Phe Ile
Arg Glu Phe Leu
Arg Glu Ile Phe
Arg Glu Leu Phe
Arg Glu Pro Tyr
Arg Glu Tyr Pro
Arg Phe Glu Ile
Arg Phe Glu Leu
Arg Phe Ile Glu
Arg Phe Lys Asn
Arg Phe Leu Glu
Arg Phe Asn Lys
Arg Phe Asn Gln
Arg Phe Gln Asn
Arg His Asp His
Arg His His Asp
Arg Ile Glu Phe
Arg Ile Phe Glu
Arg Ile Ile Tyr
Arg Ile Leu Tyr
Arg Ile Tyr Ile
Arg Ile Tyr Leu
Arg Lys Phe Asn
Arg Lys Asn Phe
Arg Leu Glu Phe
Arg Leu Phe Glu
Arg Leu Ile Tyr
Arg Leu Leu Tyr
Arg Leu Tyr Ile
Arg Leu Tyr Leu
Arg Asn Phe Lys
Arg Asn Phe Gln
Arg Asn Lys Phe
Arg Asn Gln Phe
Arg Pro Glu Tyr
Arg Pro Tyr Glu
Arg Gln Phe Asn
Arg Gln Asn Phe
Arg Tyr Glu Pro
Arg Tyr Ile Ile
Arg Tyr Ile Leu
Arg Tyr Leu Ile
Arg Tyr Leu Leu
Arg Tyr Pro Glu
Val Phe Ile Trp
Val Phe Leu Trp
Val Phe Trp Ile
Val Phe Trp Leu
Val Ile Phe Trp
Val Ile Trp Phe
Val Leu Phe Trp
Val Leu Trp Phe
Val Pro Trp Tyr
Val Pro Tyr Trp
Val Trp Phe Ile
Val Trp Phe Leu
Val Trp Ile Phe
Val Trp Leu Phe
Val Trp Pro Tyr
Val Trp Tyr Pro
Val Tyr Pro Trp
Val Tyr Trp Pro
Trp Cys Lys Lys
Trp Cys Lys Gln
Trp Cys Gln Lys
Trp Phe Ile Val
Trp Phe Leu Val
Trp Phe Val Ile
Trp Phe Val Leu
Trp Ile Phe Val
Trp Ile Val Phe
Trp Lys Cys Lys
Trp Lys Cys Gln
Trp Lys Lys Cys
Trp Lys Gln Cys
Trp Leu Phe Val
Trp Leu Val Phe
Trp Pro Val Tyr
Trp Pro Tyr Val
Trp Gln Cys Lys
Trp Gln Lys Cys
Trp Val Phe Ile
Trp Val Phe Leu
Trp Val Ile Phe
Trp Val Leu Phe
Trp Val Pro Tyr
Trp Val Tyr Pro
Trp Tyr Pro Val
Trp Tyr Val Pro
Tyr Glu Pro Arg
Tyr Glu Arg Pro
Tyr Ile Ile Arg
Tyr Ile Leu Arg
Tyr Ile Arg Ile
Tyr Ile Arg Leu
Tyr Leu Ile Arg
Tyr Leu Leu Arg
Tyr Leu Arg Ile
Tyr Leu Arg Leu
Tyr Pro Glu Arg
Tyr Pro Arg Glu
Tyr Pro Val Trp
Tyr Pro Trp Val
Tyr Arg Glu Pro
Tyr Arg Ile Ile
Tyr Arg Ile Leu
Tyr Arg Leu Ile
Tyr Arg Leu Leu
Tyr Arg Pro Glu
Tyr Val Pro Trp
Tyr Val Trp Pro
Tyr Trp Pro Val
Tyr Trp Val Pro
Dihydroergocornine
C78272 - Agent Affecting Nervous System > C29747 - Adrenergic Agent > C72900 - Adrenergic Antagonist D018377 - Neurotransmitter Agents > D015259 - Dopamine Agents > D018491 - Dopamine Agonists Ergocornine in which a single bond replaces the double bond between positions 9 and 10. C78272 - Agent Affecting Nervous System > C66884 - Dopamine Agonist
Ethyl 4-{4-[4-(hydroxydiphenylmethyl)-1-piperidinyl]-1-oxobutyl}-alpha,alpha-dimethylbenzeneacetate hydrochloride
6-[3-[(E)-1-[4-[2-(dimethylamino)ethoxy]phenyl]-2-phenylbut-1-enyl]phenoxy]-3,4,5-trihydroxyoxane-2-carboxylic acid
2-{4-[(1Z)-1,2-diphenylbut-1-en-1-yl]phenoxy}-N,N-dimethylethanamine 2-hydroxypropane-1,2,3-tricarboxylate
N-[(2S,3S)-2-[[[[(3,5-dimethyl-4-isoxazolyl)amino]-oxomethyl]-methylamino]methyl]-5-[(2R)-1-hydroxypropan-2-yl]-3-methyl-6-oxo-3,4-dihydro-2H-1,5-benzoxazocin-8-yl]-2-phenylacetamide
N-(2-aminophenyl)-4-[[[(8R,9S)-6-[(2R)-1-hydroxypropan-2-yl]-8-methyl-5-oxo-10-oxa-1,6,13,14-tetrazabicyclo[10.2.1]pentadeca-12(15),13-dien-9-yl]methyl-methylamino]methyl]benzamide
N-[(4S,7R,8R)-5-(cyclohexylmethyl)-8-methoxy-4,7,10-trimethyl-11-oxo-2-oxa-5,10-diazabicyclo[10.4.0]hexadeca-1(12),13,15-trien-14-yl]-2,2,2-trifluoroethanesulfonamide
N-[(4R,7R,8R)-5-(cyclohexylmethyl)-8-methoxy-4,7,10-trimethyl-11-oxo-2-oxa-5,10-diazabicyclo[10.4.0]hexadeca-1(12),13,15-trien-14-yl]-2,2,2-trifluoroethanesulfonamide
N-[(4R,7S,8S)-5-(cyclohexylmethyl)-8-methoxy-4,7,10-trimethyl-11-oxo-2-oxa-5,10-diazabicyclo[10.4.0]hexadeca-1(12),13,15-trien-14-yl]-2,2,2-trifluoroethanesulfonamide
N-[(4R,7R,8S)-5-(cyclohexylmethyl)-8-methoxy-4,7,10-trimethyl-11-oxo-2-oxa-5,10-diazabicyclo[10.4.0]hexadeca-1(12),13,15-trien-14-yl]-2,2,2-trifluoroethanesulfonamide
N-[(2R,3S)-2-[[[[(3,5-dimethyl-4-isoxazolyl)amino]-oxomethyl]-methylamino]methyl]-5-[(2S)-1-hydroxypropan-2-yl]-3-methyl-6-oxo-3,4-dihydro-2H-1,5-benzoxazocin-8-yl]-2-phenylacetamide
N-[(2S,3S)-2-[[[[(3,5-dimethyl-4-isoxazolyl)amino]-oxomethyl]-methylamino]methyl]-5-[(2S)-1-hydroxypropan-2-yl]-3-methyl-6-oxo-3,4-dihydro-2H-1,5-benzoxazocin-8-yl]-2-phenylacetamide
N-[(2S,3S)-2-[[1,3-benzodioxol-5-ylmethyl(methyl)amino]methyl]-5-[(2R)-1-hydroxypropan-2-yl]-3-methyl-6-oxo-3,4-dihydro-2H-1,5-benzoxazocin-10-yl]-2,5-dimethyl-3-pyrazolecarboxamide
N-(2-aminophenyl)-4-[[[(8R,9S)-6-[(2R)-1-hydroxypropan-2-yl]-8-methyl-5-oxo-10-oxa-1,6,14,15-tetrazabicyclo[10.3.0]pentadeca-12,14-dien-9-yl]methyl-methylamino]methyl]benzamide
N-(2-aminophenyl)-4-[[[(8S,9R)-6-[(2S)-1-hydroxypropan-2-yl]-8-methyl-5-oxo-10-oxa-1,6,13,14-tetrazabicyclo[10.2.1]pentadeca-12(15),13-dien-9-yl]methyl-methylamino]methyl]benzamide
N-(2-aminophenyl)-4-[[[(8R,9R)-6-[(2S)-1-hydroxypropan-2-yl]-8-methyl-5-oxo-10-oxa-1,6,14,15-tetrazabicyclo[10.3.0]pentadeca-12,14-dien-9-yl]methyl-methylamino]methyl]benzamide
N-(2-aminophenyl)-4-[[[(8S,9S)-6-[(2S)-1-hydroxypropan-2-yl]-8-methyl-5-oxo-10-oxa-1,6,14,15-tetrazabicyclo[10.3.0]pentadeca-12,14-dien-9-yl]methyl-methylamino]methyl]benzamide
N-(2-aminophenyl)-4-[[[(8S,9S)-6-[(2S)-1-hydroxypropan-2-yl]-8-methyl-5-oxo-10-oxa-1,6,13,14-tetrazabicyclo[10.2.1]pentadeca-12(15),13-dien-9-yl]methyl-methylamino]methyl]benzamide
N-[(4S,7R,8S)-5-(cyclohexylmethyl)-8-methoxy-4,7,10-trimethyl-11-oxo-2-oxa-5,10-diazabicyclo[10.4.0]hexadeca-1(12),13,15-trien-14-yl]-2,2,2-trifluoroethanesulfonamide
N-[(4R,7S,8R)-5-(cyclohexylmethyl)-8-methoxy-4,7,10-trimethyl-11-oxo-2-oxa-5,10-diazabicyclo[10.4.0]hexadeca-1(12),13,15-trien-14-yl]-2,2,2-trifluoroethanesulfonamide
(1S)-N1-cyclopentyl-N2-(4-fluorophenyl)-1-(hydroxymethyl)-7-methoxy-9-methylspiro[1,3-dihydropyrido[3,4-b]indole-4,4-piperidine]-1,2-dicarboxamide
N-[(2R,3R)-2-[[[[(3,5-dimethyl-4-isoxazolyl)amino]-oxomethyl]-methylamino]methyl]-5-[(2S)-1-hydroxypropan-2-yl]-3-methyl-6-oxo-3,4-dihydro-2H-1,5-benzoxazocin-8-yl]-2-phenylacetamide
N-[(2S,3R)-2-[[[[(3,5-dimethyl-4-isoxazolyl)amino]-oxomethyl]-methylamino]methyl]-5-[(2R)-1-hydroxypropan-2-yl]-3-methyl-6-oxo-3,4-dihydro-2H-1,5-benzoxazocin-8-yl]-2-phenylacetamide
N-[(2S,3R)-2-[[[[(3,5-dimethyl-4-isoxazolyl)amino]-oxomethyl]-methylamino]methyl]-5-[(2S)-1-hydroxypropan-2-yl]-3-methyl-6-oxo-3,4-dihydro-2H-1,5-benzoxazocin-8-yl]-2-phenylacetamide
N-[(2R,3S)-2-[[[[(3,5-dimethyl-4-isoxazolyl)amino]-oxomethyl]-methylamino]methyl]-5-[(2R)-1-hydroxypropan-2-yl]-3-methyl-6-oxo-3,4-dihydro-2H-1,5-benzoxazocin-8-yl]-2-phenylacetamide
N-[(2R,3R)-2-[[[[(3,5-dimethyl-4-isoxazolyl)amino]-oxomethyl]-methylamino]methyl]-5-[(2R)-1-hydroxypropan-2-yl]-3-methyl-6-oxo-3,4-dihydro-2H-1,5-benzoxazocin-8-yl]-2-phenylacetamide
N-[(2S,3R)-2-[[1,3-benzodioxol-5-ylmethyl(methyl)amino]methyl]-5-[(2S)-1-hydroxypropan-2-yl]-3-methyl-6-oxo-3,4-dihydro-2H-1,5-benzoxazocin-10-yl]-2,5-dimethyl-3-pyrazolecarboxamide
N-[(2S,3R)-2-[[1,3-benzodioxol-5-ylmethyl(methyl)amino]methyl]-5-[(2R)-1-hydroxypropan-2-yl]-3-methyl-6-oxo-3,4-dihydro-2H-1,5-benzoxazocin-10-yl]-2,5-dimethyl-3-pyrazolecarboxamide
N-(2-aminophenyl)-4-[[[(8S,9S)-6-[(2R)-1-hydroxypropan-2-yl]-8-methyl-5-oxo-10-oxa-1,6,13,14-tetrazabicyclo[10.2.1]pentadeca-12(15),13-dien-9-yl]methyl-methylamino]methyl]benzamide
N-(2-aminophenyl)-4-[[[(8S,9R)-6-[(2R)-1-hydroxypropan-2-yl]-8-methyl-5-oxo-10-oxa-1,6,13,14-tetrazabicyclo[10.2.1]pentadeca-12(15),13-dien-9-yl]methyl-methylamino]methyl]benzamide
N-(2-aminophenyl)-4-[[[(8S,9R)-6-[(2R)-1-hydroxypropan-2-yl]-8-methyl-5-oxo-10-oxa-1,6,14,15-tetrazabicyclo[10.3.0]pentadeca-12,14-dien-9-yl]methyl-methylamino]methyl]benzamide
N-(2-aminophenyl)-4-[[[(8R,9R)-6-[(2R)-1-hydroxypropan-2-yl]-8-methyl-5-oxo-10-oxa-1,6,14,15-tetrazabicyclo[10.3.0]pentadeca-12,14-dien-9-yl]methyl-methylamino]methyl]benzamide
N-(2-aminophenyl)-4-[[[(8R,9S)-6-[(2S)-1-hydroxypropan-2-yl]-8-methyl-5-oxo-10-oxa-1,6,14,15-tetrazabicyclo[10.3.0]pentadeca-12,14-dien-9-yl]methyl-methylamino]methyl]benzamide
N-(2-aminophenyl)-4-[[[(8S,9S)-6-[(2R)-1-hydroxypropan-2-yl]-8-methyl-5-oxo-10-oxa-1,6,14,15-tetrazabicyclo[10.3.0]pentadeca-12,14-dien-9-yl]methyl-methylamino]methyl]benzamide
N-(2-aminophenyl)-4-[[[(8R,9R)-6-[(2R)-1-hydroxypropan-2-yl]-8-methyl-5-oxo-10-oxa-1,6,13,14-tetrazabicyclo[10.2.1]pentadeca-12(15),13-dien-9-yl]methyl-methylamino]methyl]benzamide
N-(2-aminophenyl)-4-[[[(8R,9S)-6-[(2S)-1-hydroxypropan-2-yl]-8-methyl-5-oxo-10-oxa-1,6,13,14-tetrazabicyclo[10.2.1]pentadeca-12(15),13-dien-9-yl]methyl-methylamino]methyl]benzamide
N-(2-aminophenyl)-4-[[[(8R,9R)-6-[(2S)-1-hydroxypropan-2-yl]-8-methyl-5-oxo-10-oxa-1,6,13,14-tetrazabicyclo[10.2.1]pentadeca-12(15),13-dien-9-yl]methyl-methylamino]methyl]benzamide
N-[(4S,7S,8R)-5-(cyclohexylmethyl)-8-methoxy-4,7,10-trimethyl-11-oxo-2-oxa-5,10-diazabicyclo[10.4.0]hexadeca-1(12),13,15-trien-14-yl]-2,2,2-trifluoroethanesulfonamide
N-[(4S,7S,8S)-5-(cyclohexylmethyl)-8-methoxy-4,7,10-trimethyl-11-oxo-2-oxa-5,10-diazabicyclo[10.4.0]hexadeca-1(12),13,15-trien-14-yl]-2,2,2-trifluoroethanesulfonamide
(1R)-N1-cyclopentyl-N2-(4-fluorophenyl)-1-(hydroxymethyl)-7-methoxy-9-methylspiro[1,3-dihydropyrido[3,4-b]indole-4,4-piperidine]-1,2-dicarboxamide
2-amino-3-[[3-[(11Z,14Z)-henicosa-11,14-dienoyl]oxy-2-hydroxypropoxy]-hydroxyphosphoryl]oxypropanoic acid
2-amino-3-[[3-butanoyloxy-2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxypropoxy]-hydroxyphosphoryl]oxypropanoic acid
2-amino-3-[[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-propanoyloxypropoxy]-hydroxyphosphoryl]oxypropanoic acid
3-[[3-acetyloxy-2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxypropoxy]-hydroxyphosphoryl]oxy-2-aminopropanoic acid
Taurolithocholic acid 3-sulfate
D005765 - Gastrointestinal Agents > D001647 - Bile Acids and Salts D005765 - Gastrointestinal Agents > D002793 - Cholic Acids
Taurolithocholic acid sulfate
The steroid sulfate of taurolithocholic acid.
Fesoterodine (L-mandelate)
Fesoterodine L-mandelate is an orally active, nonsubtype selective, competitive muscarinic receptor (mAChR) antagonist with pKi values of 8.0, 7.7, 7.4, 7.3, 7.5 for M1, M2, M3, M4, M5 receptors, respectively. Fesoterodine L-mandelate is used for the overactive bladder (OAB)[1][2].