NCBI Taxonomy: 235719
Cremastosperma (ncbi_taxid: 235719)
found 8 associated metabolites at genus taxonomy rank level.
Ancestor: Malmeeae
Child Taxonomies: Cremastosperma westrae, Cremastosperma brevipes, Cremastosperma pendulum, Cremastosperma oblongum, Cremastosperma napoense, Cremastosperma bullatum, Cremastosperma panamense, Cremastosperma cenepense, Cremastosperma longicuspe, Cremastosperma gracilipes, Cremastosperma peruvianum, Cremastosperma magdalenae, Cremastosperma cauliflorum, Cremastosperma leiophyllum, Cremastosperma monospermum, Cremastosperma macrocarpum, Cremastosperma microcarpum, Cremastosperma stenophyllum, unclassified Cremastosperma, Cremastosperma venezuelanum, Cremastosperma pedunculatum, Cremastosperma megalophyllum, Cremastosperma yamayakatense, Cremastosperma novogranatense
LICARIN A
(-)-Licarin A is a natural product found in Magnolia dodecapetala, Magnolia kachirachirai, and other organisms with data available. Dehydrodiisoeugenol is a natural product found in Myristica fragrans with data available. Dehydrodiisoeugenol is isolated from Myristica fragrans Houtt, shows anti-inflammatory and anti-bacterial actions[1]. Dehydrodiisoeugenol inhibits LPS- stimulated NF-κB activation and cyclooxygenase (COX)-2 gene expression in murine macrophages[2]. Dehydrodiisoeugenol is isolated from Myristica fragrans Houtt, shows anti-inflammatory and anti-bacterial actions[1]. Dehydrodiisoeugenol inhibits LPS- stimulated NF-κB activation and cyclooxygenase (COX)-2 gene expression in murine macrophages[2]. Licarin A ((+)-Licarin A), a neolignan, significantly and dose-dependently reduces TNF-α production (IC50=12.6 μM) in dinitrophenyl-human serum albumin (DNP-HSA)-stimulated RBL-2H3 cells. Anti-allergic effects. Licarin A reduces TNF-α and PGD2 production, and COX-2 expression[1]. Licarin A ((+)-Licarin A), a neolignan, significantly and dose-dependently reduces TNF-α production (IC50=12.6 μM) in dinitrophenyl-human serum albumin (DNP-HSA)-stimulated RBL-2H3 cells. Anti-allergic effects. Licarin A reduces TNF-α and PGD2 production, and COX-2 expression[1].