Chemical Formula: C14H20O
Chemical Formula C14H20O
Found 83 metabolite its formula value is C14H20O
(6R,7S)-6,7-Epoxy-1,3-tetradecadiyne
(6R,7S)-6,7-Epoxy-1,3-tetradecadiyne is found in tea. (6R,7S)-6,7-Epoxy-1,3-tetradecadiyne is a constituent of Panax quinquefolium (American ginseng)
2-Benzylidene-1-heptanol
2-Benzylidene-1-heptanol is a flavouring ingredien Flavouring ingredient
3-Benzyl-4-heptanone
3-Benzyl-4-heptanone is used in food flavouring (plum/peach). It is used in food flavouring (plum/peach)
(+)-Chamecynenol
5,6,7,8-Tetrahydro-5-isopropyl-3-methyl-2-naphthol
(E)-5-(2,3-dimethyl-3-nortricyclyl)pent-3-en-2-one|1,7-Dimethyl-7-(1-pent-2-en-4-onyl)nortricyclen
(E)-4-(3-octenyl)phenol|E-4-(3-octenyl)phenol|gibbilimbol D
3-[2-Methyl-4-(2-methyl-2-propanyl)phenyl]propanal
3-[3-Methyl-5-(2-methyl-2-propanyl)phenyl]propanal
3-[2-Methyl-5-(2-methyl-2-propanyl)phenyl]propanal
Benzene,1-(1,1-dimethylethyl)-4-[(2-methyl-2-propen-1-yl)oxy]-
Ethanone,1-[4-(1,1-dimethylethyl)-2,6-dimethylphenyl]-
1H-2-Benzopyran,3,4-dihydro-1,1,4,4,7-pentamethyl-(9CI)
1-Isobutyl-9-methylbicyclo(3.2.2)nona-3,6-dien-2-one
Cipepofol
Cipepofol (Ciprofol), a novel 2,6-disubstituted phenol derivative, is a positive allosteric modulator and direct agonist of the GABAA receptor. Cipepofol can cause the central nerve inhibition and promote sleep based on the structural modification of Propofol (HY-B0649). Cipepofol can activate the sirtuin1 (Sirt1)/Nrf2 pathway. Cipepofol protects the heart against Isoproterenol (ISO; HY-B0468)-induced myocardial infarction by reducing cardiac oxidative stress, inflammatory response and cardiomyocyte apoptosis[1][2]. Cipepofol (Ciprofol), a novel 2,6-disubstituted phenol derivative, is a positive allosteric modulator and direct agonist of the GABAA receptor. Cipepofol can cause the central nerve inhibition and promote sleep based on the structural modification of Propofol (HY-B0649). Cipepofol can activate the sirtuin1 (Sirt1)/Nrf2 pathway. Cipepofol protects the heart against Isoproterenol (ISO; HY-B0468)-induced myocardial infarction by reducing cardiac oxidative stress, inflammatory response and cardiomyocyte apoptosis[1][2].