Exact Mass: 830.7151723999999

Glycerol 1,3-dihexadecanoate 2-(9Z,12Z-octadecadienoate)

C53H98O6 (830.7363008)

Glycerol 1,3-dihexadecanoate 2-(9Z,12Z-octadecadienoate) is found in cocoa and cocoa products. Glycerol 1,3-dihexadecanoate 2-(9Z,12Z-octadecadienoate) is a minor component of sunflower and other vegetable oil Minor component of sunflower and other vegetable oils. Glycerol 1,3-dihexadecanoate 2-(9Z,12Z-octadecadienoate) is found in cocoa and cocoa products and fats and oils.

TG(16:0/16:0/18:2(9Z,12Z))

C53H98O6 (830.7363008)

TG(16:0/16:0/18:2(9Z,12Z))[iso3] is a dipalmitic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/16:0/18:2(9Z,12Z))[iso3], in particular, consists of one chain of palmitic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of linoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols. TG(16:0/16:0/18:2(9Z,12Z))[iso3] is a dipalmitic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/16:0/18:2(9Z,12Z))[iso3], in particular, consists of one chain of palmitic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of linoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)

TG(16:0/16:1(9Z)/18:1(9Z))

C53H98O6 (830.7363008)

TG(16:0/16:1(9Z)/18:1(9Z))[iso6] is a monooleic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/16:1(9Z)/18:1(9Z))[iso6], in particular, consists of one chain of palmitic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols. TG(16:0/16:1(9Z)/18:1(9Z))[iso6] is a monooleic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/16:1(9Z)/18:1(9Z))[iso6], in particular, consists of one chain of palmitic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)

TG(16:1(9Z)/16:1(9Z)/18:0)

C53H98O6 (830.7363008)

TG(16:1(9Z)/16:1(9Z)/18:0)[iso3] is a dipalmitoleic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:1(9Z)/16:1(9Z)/18:0)[iso3], in particular, consists of one chain of palmitoleic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of stearic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:1(9Z)/16:0/18:1(11Z))

C53H98O6 (830.7363008)

TG(16:1(9Z)/16:0/18:1(11Z))[iso6] is a monovaccenic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:1(9Z)/16:0/18:1(11Z))[iso6], in particular, consists of one chain of palmitoleic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of vaccenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols. TG(16:1(9Z)/16:0/18:1(11Z))[iso6] is a monovaccenic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:1(9Z)/16:0/18:1(11Z))[iso6], in particular, consists of one chain of palmitoleic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of vaccenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)

TG(18:0/14:0/18:2(9Z,12Z))

C53H98O6 (830.7363008)

TG(18:0/14:0/18:2(9Z,12Z))[iso6] is a monostearic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:0/14:0/18:2(9Z,12Z))[iso6], in particular, consists of one chain of stearic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of linoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(18:1(11Z)/14:0/18:1(11Z))

C53H98O6 (830.7363008)

TG(18:1(11Z)/14:0/18:1(11Z))[iso3] is a divaccenic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:1(11Z)/14:0/18:1(11Z))[iso3], in particular, consists of one chain of vaccenic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of vaccenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(18:1(11Z)/14:0/18:1(9Z))

C53H98O6 (830.7363008)

TG(18:1(11Z)/14:0/18:1(9Z))[iso6] is a monovaccenic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:1(11Z)/14:0/18:1(9Z))[iso6], in particular, consists of one chain of vaccenic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols. TG(18:1(11Z)/14:0/18:1(9Z))[iso6] is a monovaccenic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:1(11Z)/14:0/18:1(9Z))[iso6], in particular, consists of one chain of vaccenic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)

TG(18:1(9Z)/14:0/18:1(9Z))

C53H98O6 (830.7363008)

TG(18:1(9Z)/14:0/18:1(9Z))[iso3] is a dioleic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:1(9Z)/14:0/18:1(9Z))[iso3], in particular, consists of one chain of oleic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

Glycerol 1,2-di-(9Z-octadecenoate) 3-tetradecanoate

C53H98O6 (830.7363008)

Glycerol 1,2-di-(9Z-octadecenoate) 3-tetradecanoate is found in fats and oils. Glycerol 1,2-di-(9Z-octadecenoate) 3-tetradecanoate is a minor component of palm oil and other vegetable oil Minor component of palm oil and other vegetable oils. Glycerol 1,2-di-(9Z-octadecenoate) 3-tetradecanoate is found in fats and oils.

Glycerol 1,2-dihexadecanoate 3-(9Z,12Z-octadecadienoate)

C53H98O6 (830.7363008)

Glycerol 1,2-dihexadecanoate 3-(9Z,12Z-octadecadienoate) is found in fats and oils. Glycerol 1,2-dihexadecanoate 3-(9Z,12Z-octadecadienoate) is a minor component of sunflower oil and other vegetable oil Minor component of sunflower oil and other vegetable oils. Glycerol 1,2-dihexadecanoate 3-(9Z,12Z-octadecadienoate) is found in fats and oils.

TG(14:0/14:0/22:2(13Z,16Z))

C53H98O6 (830.7363008)

TG(14:0/14:0/22:2(13Z,16Z)) is a dimyristic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/14:0/22:2(13Z,16Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of docosadienoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/16:0/20:2n6)

C53H98O6 (830.7363008)

TG(14:0/16:0/20:2n6) is a monoeicosadienoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/16:0/20:2n6), in particular, consists of one chain of myristic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of eicosadienoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/18:0/18:2(9Z,12Z))

C53H98O6 (830.7363008)

TG(14:0/18:0/18:2(9Z,12Z)) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/18:0/18:2(9Z,12Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of stearic acid at the C-2 position and one chain of linoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/14:1(9Z)/22:1(13Z))

C53H98O6 (830.7363008)

TG(14:0/14:1(9Z)/22:1(13Z)) is a monoerucic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/14:1(9Z)/22:1(13Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of erucic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/16:1(9Z)/20:1(11Z))

C53H98O6 (830.7363008)

TG(14:0/16:1(9Z)/20:1(11Z)) is a monoeicosenoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/16:1(9Z)/20:1(11Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of eicosenoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/18:1(11Z)/18:1(11Z))

C53H98O6 (830.7363008)

TG(14:0/18:1(11Z)/18:1(11Z)) is a divaccenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/18:1(11Z)/18:1(11Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of vaccenic acid at the C-2 position and one chain of vaccenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/18:1(11Z)/18:1(9Z))

C53H98O6 (830.7363008)

TG(14:0/18:1(11Z)/18:1(9Z)) is a monovaccenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/18:1(11Z)/18:1(9Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of vaccenic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/18:1(9Z)/18:1(11Z))

C53H98O6 (830.7363008)

TG(14:0/18:1(9Z)/18:1(11Z)) is a monooleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/18:1(9Z)/18:1(11Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of oleic acid at the C-2 position and one chain of vaccenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/18:1(9Z)/18:1(9Z))

C53H98O6 (830.7363008)

TG(14:0/18:1(9Z)/18:1(9Z)) is a dioleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/18:1(9Z)/18:1(9Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of oleic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/20:1(11Z)/16:1(9Z))

C53H98O6 (830.7363008)

TG(14:0/20:1(11Z)/16:1(9Z)) is a monoeicosenoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/20:1(11Z)/16:1(9Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of eicosenoic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/22:1(13Z)/14:1(9Z))

C53H98O6 (830.7363008)

TG(14:0/22:1(13Z)/14:1(9Z)) is a monoerucic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/22:1(13Z)/14:1(9Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of erucic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/18:2(9Z,12Z)/18:0)

C53H98O6 (830.7363008)

TG(14:0/18:2(9Z,12Z)/18:0) is a monolinoleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/18:2(9Z,12Z)/18:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of linoleic acid at the C-2 position and one chain of stearic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/20:2n6/16:0)

C53H98O6 (830.7363008)

TG(14:0/20:2n6/16:0) is a monoeicosadienoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/20:2n6/16:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of eicosadienoic acid at the C-2 position and one chain of palmitic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/22:2(13Z,16Z)/14:0)

C53H98O6 (830.7363008)

TG(14:0/22:2(13Z,16Z)/14:0) is a dimyristic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/22:2(13Z,16Z)/14:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of docosadienoic acid at the C-2 position and one chain of myristic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/15:0/20:2n6)

C53H98O6 (830.7363008)

TG(15:0/15:0/20:2n6) is a dipentadecanoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/15:0/20:2n6), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of eicosadienoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/20:2n6/15:0)

C53H98O6 (830.7363008)

TG(15:0/20:2n6/15:0) is a dipentadecanoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/20:2n6/15:0), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of eicosadienoic acid at the C-2 position and one chain of pentadecanoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/14:0/20:2n6)

C53H98O6 (830.7363008)

TG(16:0/14:0/20:2n6) is a monoeicosadienoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/14:0/20:2n6), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of eicosadienoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/14:1(9Z)/20:1(11Z))

C53H98O6 (830.7363008)

TG(16:0/14:1(9Z)/20:1(11Z)) is a monoeicosenoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/14:1(9Z)/20:1(11Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of eicosenoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/16:1(9Z)/18:1(11Z))

C53H98O6 (830.7363008)

TG(16:0/16:1(9Z)/18:1(11Z)) is a monovaccenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/16:1(9Z)/18:1(11Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of vaccenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/18:1(11Z)/16:1(9Z))

C53H98O6 (830.7363008)

TG(16:0/18:1(11Z)/16:1(9Z)) is a monovaccenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/18:1(11Z)/16:1(9Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of vaccenic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/18:1(9Z)/16:1(9Z))

C53H98O6 (830.7363008)

TG(16:0/18:1(9Z)/16:1(9Z)) is a monooleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/18:1(9Z)/16:1(9Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of oleic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/20:1(11Z)/14:1(9Z))

C53H98O6 (830.7363008)

TG(16:0/20:1(11Z)/14:1(9Z)) is a monoeicosenoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/20:1(11Z)/14:1(9Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of eicosenoic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/18:2(9Z,12Z)/16:0)

C53H98O6 (830.7363008)

TG(16:0/18:2(9Z,12Z)/16:0) is a dipalmitic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/18:2(9Z,12Z)/16:0), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of linoleic acid at the C-2 position and one chain of palmitic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(18:0/14:1(9Z)/18:1(11Z))

C53H98O6 (830.7363008)

TG(18:0/14:1(9Z)/18:1(11Z)) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:0/14:1(9Z)/18:1(11Z)), in particular, consists of one chain of stearic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of vaccenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(18:0/14:1(9Z)/18:1(9Z))

C53H98O6 (830.7363008)

TG(18:0/14:1(9Z)/18:1(9Z)) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:0/14:1(9Z)/18:1(9Z)), in particular, consists of one chain of stearic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(18:0/16:1(9Z)/16:1(9Z))

C53H98O6 (830.7363008)

TG(18:0/16:1(9Z)/16:1(9Z)) is a dipalmitoleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:0/16:1(9Z)/16:1(9Z)), in particular, consists of one chain of stearic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols. TG(16:1(9Z)/16:1(9Z)/18:0)[iso3] is a dipalmitoleic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:1(9Z)/16:1(9Z)/18:0)[iso3], in particular, consists of one chain of palmitoleic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of stearic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)

TG(18:0/18:1(11Z)/14:1(9Z))

C53H98O6 (830.7363008)

TG(18:0/18:1(11Z)/14:1(9Z)) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:0/18:1(11Z)/14:1(9Z)), in particular, consists of one chain of stearic acid at the C-1 position, one chain of vaccenic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(18:0/18:1(9Z)/14:1(9Z))

C53H98O6 (830.7363008)

TG(18:0/18:1(9Z)/14:1(9Z)) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:0/18:1(9Z)/14:1(9Z)), in particular, consists of one chain of stearic acid at the C-1 position, one chain of oleic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(20:0/14:1(9Z)/16:1(9Z))

C53H98O6 (830.7363008)

TG(20:0/14:1(9Z)/16:1(9Z)) is a monoarachidic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(20:0/14:1(9Z)/16:1(9Z)), in particular, consists of one chain of arachidic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(20:0/16:1(9Z)/14:1(9Z))

C53H98O6 (830.7363008)

TG(20:0/16:1(9Z)/14:1(9Z)) is a monoarachidic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(20:0/16:1(9Z)/14:1(9Z)), in particular, consists of one chain of arachidic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(22:0/14:1(9Z)/14:1(9Z))

C53H98O6 (830.7363008)

TG(22:0/14:1(9Z)/14:1(9Z)) is a dimyristoleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(22:0/14:1(9Z)/14:1(9Z)), in particular, consists of one chain of behenic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:1(9Z)/14:0/22:1(13Z))

C53H98O6 (830.7363008)

TG(14:1(9Z)/14:0/22:1(13Z)) is a monoerucic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/14:0/22:1(13Z)), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of erucic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:1(9Z)/16:0/20:1(11Z))

C53H98O6 (830.7363008)

TG(14:1(9Z)/16:0/20:1(11Z)) is a monoeicosenoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/16:0/20:1(11Z)), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of eicosenoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:1(9Z)/18:0/18:1(11Z))

C53H98O6 (830.7363008)

TG(14:1(9Z)/18:0/18:1(11Z)) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/18:0/18:1(11Z)), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of stearic acid at the C-2 position and one chain of vaccenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:1(9Z)/18:0/18:1(9Z))

C53H98O6 (830.7363008)

TG(14:1(9Z)/18:0/18:1(9Z)) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/18:0/18:1(9Z)), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of stearic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:1(9Z)/20:0/16:1(9Z))

C53H98O6 (830.7363008)

TG(14:1(9Z)/20:0/16:1(9Z)) is a monoarachidic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/20:0/16:1(9Z)), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of arachidic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:1(9Z)/22:0/14:1(9Z))

C53H98O6 (830.7363008)

TG(14:1(9Z)/22:0/14:1(9Z)) is a dimyristoleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/22:0/14:1(9Z)), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of behenic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:1(9Z)/14:0/20:1(11Z))

C53H98O6 (830.7363008)

TG(16:1(9Z)/14:0/20:1(11Z)) is a monoeicosenoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:1(9Z)/14:0/20:1(11Z)), in particular, consists of one chain of palmitoleic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of eicosenoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:1(9Z)/16:0/18:1(9Z))

C53H98O6 (830.7363008)

TG(16:1(9Z)/16:0/18:1(9Z)) is a monooleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:1(9Z)/16:0/18:1(9Z)), in particular, consists of one chain of palmitoleic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:1(9Z)/18:0/16:1(9Z))

C53H98O6 (830.7363008)

TG(16:1(9Z)/18:0/16:1(9Z)) is a dipalmitoleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:1(9Z)/18:0/16:1(9Z)), in particular, consists of one chain of palmitoleic acid at the C-1 position, one chain of stearic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

1-tetradecanoyl-2,3-dioleoyl-sn-glycerol

C53H98O6 (830.7363008)

1-tetradecanoyl-2,3-dioleoyl-sn-glycerol is considered to be practically insoluble (in water) and basic. 1-tetradecanoyl-2,3-dioleoyl-sn-glycerol is a triradylglycerol lipid molecule

3,3,4,4-Tetrahydro-2,2,7,7,8,8-hexamethyl-2,2-bis(4,8,12-trimethyltridecyl)[5,6-oxybis(2H-1-benzopyran)]-6-ol

C56H94O4 (830.7151723999999)

C47H94N2O9

C47H94N2O9 (830.6958954)

TG(14:0/14:1(9Z)/22:1(11Z))[iso6]

C53H98O6 (830.7363008)

TG(14:0/16:0/20:2(11Z,14Z))[iso6]

C53H98O6 (830.7363008)

TG(14:0/16:1(9Z)/20:1(11Z))[iso6]

C53H98O6 (830.7363008)

TG(14:0/17:1(9Z)/19:1(9Z))[iso6]

C53H98O6 (830.7363008)

TG(14:0/17:2(9Z,12Z)/19:0)[iso6]

C53H98O6 (830.7363008)

TG(14:0/18:0/18:2(9Z,12Z))[iso6]

C53H98O6 (830.7363008)

TG(14:1(9Z)/15:1(9Z)/21:0)[iso6]

C53H98O6 (830.7363008)

TG(14:1(9Z)/16:0/20:1(11Z))[iso6]

C53H98O6 (830.7363008)

TG(14:1(9Z)/16:1(9Z)/20:0)[iso6]

C53H98O6 (830.7363008)

TG(14:1(9Z)/17:0/19:1(9Z))[iso6]

C53H98O6 (830.7363008)

TG(14:1(9Z)/17:1(9Z)/19:0)[iso6]

C53H98O6 (830.7363008)

TG(14:1(9Z)/18:0/18:1(9Z))[iso6]

C53H98O6 (830.7363008)

TG(15:0/15:1(9Z)/20:1(11Z))[iso6]

C53H98O6 (830.7363008)

TG(15:0/16:1(9Z)/19:1(9Z))[iso6]

C53H98O6 (830.7363008)

TG(15:0/17:0/18:2(9Z,12Z))[iso6]

C53H98O6 (830.7363008)

TG(15:0/17:1(9Z)/18:1(9Z))[iso6]

C53H98O6 (830.7363008)

TG(15:0/17:2(9Z,12Z)/18:0)[iso6]

C53H98O6 (830.7363008)

TG(15:1(9Z)/16:0/19:1(9Z))[iso6]

C53H98O6 (830.7363008)

TG(15:1(9Z)/16:1(9Z)/19:0)[iso6]

C53H98O6 (830.7363008)

TG(15:1(9Z)/17:0/18:1(9Z))[iso6]

C53H98O6 (830.7363008)

TG(15:1(9Z)/17:1(9Z)/18:0)[iso6]

C53H98O6 (830.7363008)

TG(16:0/17:1/17:1)[iso3]

C53H98O6 (830.7363008)

TG(16:1/17:0/17:1)[iso6]

C53H98O6 (830.7363008)

TG(16:0/17:0/17:2)[iso6]

C53H98O6 (830.7363008)

TG(16:1/16:1/18:0)[iso3]

C53H98O6 (830.7363008)

TG(16:0/16:1/18:1)[iso6]

C53H98O6 (830.7363008)

TG(16:0/16:0/18:2)[iso3]

C53H98O6 (830.7363008)

Triglyceride

C53H98O6 (830.7363008)

a,b-Dioleomyristin

C53H98O6 (830.7363008)

a,b-Dipalmitolinolein

C53H98O6 (830.7363008)

a,a'-Dipalmitolinolein

C53H98O6 (830.7363008)

1-dodecanoyl-2,3-di9Z-nonadecenoyl-sn-glycerol

C53H98O6 (830.7363008)

TG(14:0/14:0/22:2(13Z,16Z))[iso3]

C53H98O6 (830.7363008)

TG(14:0/18:1(9Z)/18:1(9Z))[iso3]

C53H98O6 (830.7363008)

TG(14:1(9Z)/14:1(9Z)/22:0)[iso3]

C53H98O6 (830.7363008)

TG(15:0/15:0/20:2(11Z,14Z))[iso3]

C53H98O6 (830.7363008)

TG(15:1(9Z)/15:1(9Z)/20:0)[iso3]

C53H98O6 (830.7363008)

TG(12:0/16:0/22:2(13Z,16Z))[iso6]

C53H98O6 (830.7363008)

TG(12:0/16:1(9Z)/22:1(11Z))[iso6]

C53H98O6 (830.7363008)

TG(12:0/17:2(9Z,12Z)/21:0)[iso6]

C53H98O6 (830.7363008)

TG(12:0/18:0/20:2(11Z,14Z))[iso6]

C53H98O6 (830.7363008)

TG(12:0/18:1(9Z)/20:1(11Z))[iso6]

C53H98O6 (830.7363008)

TG(12:0/18:2(9Z,12Z)/20:0)[iso6]

C53H98O6 (830.7363008)

TG(13:0/15:0/22:2(13Z,16Z))[iso6]

C53H98O6 (830.7363008)

TG(13:0/15:1(9Z)/22:1(11Z))[iso6]

C53H98O6 (830.7363008)

TG(13:0/17:0/20:2(11Z,14Z))[iso6]

C53H98O6 (830.7363008)

TG(13:0/17:1(9Z)/20:1(11Z))[iso6]

C53H98O6 (830.7363008)

TG(13:0/17:2(9Z,12Z)/20:0)[iso6]

C53H98O6 (830.7363008)

TG(13:0/18:1(9Z)/19:1(9Z))[iso6]

C53H98O6 (830.7363008)

TG(13:0/18:2(9Z,12Z)/19:0)[iso6]

C53H98O6 (830.7363008)

TG 50:2

C53H98O6 (830.7363008)

TG(18:1(9Z)/14:0/18:1(9Z))[iso3]

C53H98O6 (830.7363008)

1-tetradecanoyl-2,3-dioleoyl-sn-glycerol

C53H98O6 (830.7363008)

A triacyl-sn-glycerol in which the 1-acyl group is tetradecanoyl while the 2- and 3-acyl groups are oleoyl.

1-Palmitoyl-2-palmitoyl-3-linoleoyl-glycerol

C53H98O6 (830.7363008)

1-Palmitoleoyl-2-palmitoyl-3-vaccenoyl-glycerol

C53H98O6 (830.7363008)

1-Vaccenoyl-2-myristoyl-3-oleoyl-glycerol

C53H98O6 (830.7363008)

1-Palmitoyl-2-palmitoleoyl-3-oleoyl-glycerol

C53H98O6 (830.7363008)

1-Palmitoleoyl-2-palmitoleoyl-3-stearoyl-glycerol

C53H98O6 (830.7363008)

2-[[3-docosanoyloxy-2-[(Z)-octadec-1-enoxy]propoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[2-[(Z)-hexadec-1-enoxy]-3-tetracosanoyloxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[3-[(Z)-hexadec-1-enoxy]-2-tetracosanoyloxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[2-docosanoyloxy-3-[(Z)-octadec-1-enoxy]propoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[(2R)-2-docosanoyloxy-3-[(Z)-octadec-9-enoxy]propoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

TG(18:0/14:0/18:2(9Z,12Z))[iso6]

C53H98O6 (830.7363008)

1-Linoleoyl-2-isoheptadecanoyl-3-isopentadecanoyl-sn-glycerol

C53H98O6 (830.7363008)

A triacylglycerol 50:2 in which the acyl groups at positions 1, 2 and 3 are specified as linoleoyl, isoheptadecanoyl and isopentadecanoyl respectively. It is a metabolite of the nematode Caenorhabditis elegans.

N-hexacosanoyl-14-methylhexadecasphinganine-1-phosphocholine

C48H99N2O6P (830.7240363999999)

A sphingomyelin obtained by formal condensation of the carboxy group of hexacosanoic acid with the amino group of 14-methylhexadecasphinganine-1-phosphocholine. It is a metabolite of the nematode Caenorhabditis elegans.

2-[[(2R)-2-[(Z)-docos-11-enoyl]oxy-3-octadecoxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

9Z,12Z-octadecadienoic acid, 2,3-bis[(1-oxohexadecyl)oxy]propyl ester

C53H98O6 (830.7363008)

[3-hexadecanoyloxy-2-[(Z)-hexadec-9-enoyl]oxypropyl] (Z)-octadec-9-enoate

C53H98O6 (830.7363008)

[2-[(Z)-octadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] (Z)-octadec-9-enoate

C53H98O6 (830.7363008)

[10,13-dimethyl-17-(6-methylheptan-2-yl)-2,3,4,7,8,9,11,12,14,15,16,17-dodecahydro-1H-cyclopenta[a]phenanthren-3-yl] (5Z,8Z,11Z,14Z,17Z,20Z,23Z,26Z,29Z)-dotriaconta-5,8,11,14,17,20,23,26,29-nonaenoate

C59H90O2 (830.694044)

PE-Cer 21:0;2O/25:0

C48H99N2O6P (830.7240363999999)

PE-Cer 26:0;2O/20:0

C48H99N2O6P (830.7240363999999)

PE-Cer 22:0;2O/24:0

C48H99N2O6P (830.7240363999999)

PE-Cer 25:0;2O/21:0

C48H99N2O6P (830.7240363999999)

PE-Cer 23:0;2O/23:0

C48H99N2O6P (830.7240363999999)

PE-Cer 20:0;2O/26:0

C48H99N2O6P (830.7240363999999)

PE-Cer 24:0;2O/22:0

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(octanoylamino)pentatriacontyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(nonanoylamino)tetratriacontyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[2-(Heptanoylamino)-3-hydroxyhexatriacontyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[2-(Hexanoylamino)-3-hydroxyheptatriacontyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(tetratriacontanoylamino)nonyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(pentanoylamino)octatriacontyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[2-(Decanoylamino)-3-hydroxytritriacontyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[2-(Dodecanoylamino)-3-hydroxyhentriacontyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[2-(Dotriacontanoylamino)-3-hydroxyundecyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[2-(Hentriacontanoylamino)-3-hydroxydodecyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(triacontanoylamino)tridecyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(tritriacontanoylamino)decyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(undecanoylamino)dotriacontyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

2,3-di(octanoyloxy)propyl (23Z,26Z)-tetratriaconta-23,26-dienoate

C53H98O6 (830.7363008)

2,3-di(nonanoyloxy)propyl (21Z,24Z)-dotriaconta-21,24-dienoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-nonanoyloxypropyl] pentacosanoate

C53H98O6 (830.7363008)

[2-[(Z)-nonadec-9-enoyl]oxy-3-nonanoyloxypropyl] (Z)-docos-13-enoate

C53H98O6 (830.7363008)

[2-[(Z)-octadec-9-enoyl]oxy-3-octanoyloxypropyl] (Z)-tetracos-13-enoate

C53H98O6 (830.7363008)

(2-icosanoyloxy-3-octanoyloxypropyl) (13Z,16Z)-docosa-13,16-dienoate

C53H98O6 (830.7363008)

(3-nonanoyloxy-2-pentadecanoyloxypropyl) (15Z,18Z)-hexacosa-15,18-dienoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-octanoyloxypropyl] hexacosanoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-nonanoyloxypropyl] tetracosanoate

C53H98O6 (830.7363008)

(2-hexadecanoyloxy-3-octanoyloxypropyl) (15Z,18Z)-hexacosa-15,18-dienoate

C53H98O6 (830.7363008)

(3-nonanoyloxy-2-tridecanoyloxypropyl) (17Z,20Z)-octacosa-17,20-dienoate

C53H98O6 (830.7363008)

[3-nonanoyloxy-2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxypropyl] tricosanoate

C53H98O6 (830.7363008)

(3-nonanoyloxy-2-undecanoyloxypropyl) (19Z,22Z)-triaconta-19,22-dienoate

C53H98O6 (830.7363008)

[2-[(Z)-icos-11-enoyl]oxy-3-octanoyloxypropyl] (Z)-docos-13-enoate

C53H98O6 (830.7363008)

(2-decanoyloxy-3-octanoyloxypropyl) (21Z,24Z)-dotriaconta-21,24-dienoate

C53H98O6 (830.7363008)

[3-nonanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] (Z)-octacos-17-enoate

C53H98O6 (830.7363008)

(2-heptadecanoyloxy-3-nonanoyloxypropyl) (13Z,16Z)-tetracosa-13,16-dienoate

C53H98O6 (830.7363008)

(2-icosanoyloxy-3-nonanoyloxypropyl) (11Z,14Z)-henicosa-11,14-dienoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-octanoyloxypropyl] pentacosanoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-nonadeca-9,12-dienoyl]oxy-3-nonanoyloxypropyl] docosanoate

C53H98O6 (830.7363008)

(3-octanoyloxy-2-tetradecanoyloxypropyl) (17Z,20Z)-octacosa-17,20-dienoate

C53H98O6 (830.7363008)

[2-[(11Z,14Z)-icosa-11,14-dienoyl]oxy-3-nonanoyloxypropyl] henicosanoate

C53H98O6 (830.7363008)

[3-octanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-octacos-17-enoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-nonadeca-9,12-dienoyl]oxy-3-octanoyloxypropyl] tricosanoate

C53H98O6 (830.7363008)

[2-[(11Z,14Z)-icosa-11,14-dienoyl]oxy-3-octanoyloxypropyl] docosanoate

C53H98O6 (830.7363008)

(2-nonadecanoyloxy-3-nonanoyloxypropyl) (13Z,16Z)-docosa-13,16-dienoate

C53H98O6 (830.7363008)

[2-[(Z)-hexadec-9-enoyl]oxy-3-octanoyloxypropyl] (Z)-hexacos-15-enoate

C53H98O6 (830.7363008)

[2-[(Z)-icos-11-enoyl]oxy-3-nonanoyloxypropyl] (Z)-henicos-11-enoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-9-enoyl]oxy-3-nonanoyloxypropyl] (Z)-tetracos-13-enoate

C53H98O6 (830.7363008)

(2-octadecanoyloxy-3-octanoyloxypropyl) (13Z,16Z)-tetracosa-13,16-dienoate

C53H98O6 (830.7363008)

[1-[(11Z,14Z)-henicosa-11,14-dienoyl]oxy-3-octanoyloxypropan-2-yl] henicosanoate

C53H98O6 (830.7363008)

[3-nonanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] (Z)-hexacos-15-enoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxy-3-octanoyloxypropyl] tetracosanoate

C53H98O6 (830.7363008)

(2-dodecanoyloxy-3-octanoyloxypropyl) (19Z,22Z)-triaconta-19,22-dienoate

C53H98O6 (830.7363008)

[2-[(Z)-henicos-11-enoyl]oxy-3-octanoyloxypropyl] (Z)-henicos-11-enoate

C53H98O6 (830.7363008)

2,3-di(tridecanoyloxy)propyl (13Z,16Z)-tetracosa-13,16-dienoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-tridecanoyloxypropyl] henicosanoate

C53H98O6 (830.7363008)

[2-heptadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (Z)-icos-11-enoate

C53H98O6 (830.7363008)

[2-[(Z)-pentadec-9-enoyl]oxy-3-tridecanoyloxypropyl] (Z)-docos-13-enoate

C53H98O6 (830.7363008)

(2-octadecanoyloxy-3-undecanoyloxypropyl) (11Z,14Z)-henicosa-11,14-dienoate

C53H98O6 (830.7363008)

[3-decanoyloxy-2-[(Z)-icos-11-enoyl]oxypropyl] (Z)-icos-11-enoate

C53H98O6 (830.7363008)

[2-[(Z)-octadec-9-enoyl]oxy-3-tridecanoyloxypropyl] (Z)-nonadec-9-enoate

C53H98O6 (830.7363008)

[3-decanoyloxy-2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxypropyl] tricosanoate

C53H98O6 (830.7363008)

[2-[(Z)-hexadec-9-enoyl]oxy-3-tridecanoyloxypropyl] (Z)-henicos-11-enoate

C53H98O6 (830.7363008)

[3-dodecanoyloxy-2-[(Z)-hexadec-9-enoyl]oxypropyl] (Z)-docos-13-enoate

C53H98O6 (830.7363008)

[1-[(Z)-octadec-9-enoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropan-2-yl] octadecanoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-tridecanoyloxypropyl] icosanoate

C53H98O6 (830.7363008)

[1-dodecanoyloxy-3-[(9Z,12Z)-nonadeca-9,12-dienoyl]oxypropan-2-yl] nonadecanoate

C53H98O6 (830.7363008)

[2-pentadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-henicos-11-enoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxy-3-undecanoyloxypropyl] henicosanoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-9-enoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] icosanoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-undecanoyloxypropyl] docosanoate

C53H98O6 (830.7363008)

(2-pentadecanoyloxy-3-undecanoyloxypropyl) (13Z,16Z)-tetracosa-13,16-dienoate

C53H98O6 (830.7363008)

[2-[(Z)-octadec-9-enoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] nonadecanoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-tetradecanoyloxypropyl] icosanoate

C53H98O6 (830.7363008)

[2-[(Z)-tridec-9-enoyl]oxy-3-undecanoyloxypropyl] (Z)-hexacos-15-enoate

C53H98O6 (830.7363008)

[2-[(Z)-hexadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] (Z)-icos-11-enoate

C53H98O6 (830.7363008)

(3-dodecanoyloxy-2-heptadecanoyloxypropyl) (11Z,14Z)-henicosa-11,14-dienoate

C53H98O6 (830.7363008)

2,3-di(undecanoyloxy)propyl (17Z,20Z)-octacosa-17,20-dienoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-9-enoyl]oxy-3-tridecanoyloxypropyl] (Z)-icos-11-enoate

C53H98O6 (830.7363008)

[2-octadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (Z)-nonadec-9-enoate

C53H98O6 (830.7363008)

[2-[(Z)-hexadec-9-enoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] icosanoate

C53H98O6 (830.7363008)

[2-[(Z)-pentadec-9-enoyl]oxy-3-undecanoyloxypropyl] (Z)-tetracos-13-enoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-9-enoyl]oxy-3-undecanoyloxypropyl] (Z)-docos-13-enoate

C53H98O6 (830.7363008)

[3-decanoyloxy-2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxypropyl] docosanoate

C53H98O6 (830.7363008)

[3-dodecanoyloxy-2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxypropyl] icosanoate

C53H98O6 (830.7363008)

[3-decanoyloxy-2-[(Z)-octadec-9-enoyl]oxypropyl] (Z)-docos-13-enoate

C53H98O6 (830.7363008)

[2-[(Z)-tetradec-9-enoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] tricosanoate

C53H98O6 (830.7363008)

(3-dodecanoyloxy-2-tetradecanoyloxypropyl) (13Z,16Z)-tetracosa-13,16-dienoate

C53H98O6 (830.7363008)

[2-[(Z)-pentadec-9-enoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] docosanoate

C53H98O6 (830.7363008)

[3-decanoyloxy-2-[(9Z,12Z)-nonadeca-9,12-dienoyl]oxypropyl] henicosanoate

C53H98O6 (830.7363008)

[2-[(Z)-nonadec-9-enoyl]oxy-3-undecanoyloxypropyl] (Z)-icos-11-enoate

C53H98O6 (830.7363008)

(2-nonadecanoyloxy-3-undecanoyloxypropyl) (11Z,14Z)-icosa-11,14-dienoate

C53H98O6 (830.7363008)

(3-dodecanoyloxy-2-hexadecanoyloxypropyl) (13Z,16Z)-docosa-13,16-dienoate

C53H98O6 (830.7363008)

2,3-di(decanoyloxy)propyl (19Z,22Z)-triaconta-19,22-dienoate

C53H98O6 (830.7363008)

(3-decanoyloxy-2-hexadecanoyloxypropyl) (13Z,16Z)-tetracosa-13,16-dienoate

C53H98O6 (830.7363008)

(2-tridecanoyloxy-3-undecanoyloxypropyl) (15Z,18Z)-hexacosa-15,18-dienoate

C53H98O6 (830.7363008)

[2-[(Z)-pentadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] (Z)-henicos-11-enoate

C53H98O6 (830.7363008)

(3-decanoyloxy-2-tetradecanoyloxypropyl) (15Z,18Z)-hexacosa-15,18-dienoate

C53H98O6 (830.7363008)

[3-tridecanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] (Z)-tetracos-13-enoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxy-3-tridecanoyloxypropyl] nonadecanoate

C53H98O6 (830.7363008)

(2-pentadecanoyloxy-3-tridecanoyloxypropyl) (13Z,16Z)-docosa-13,16-dienoate

C53H98O6 (830.7363008)

[1-[(9Z,12Z)-octadeca-9,12-dienoyl]oxy-3-tetradecanoyloxypropan-2-yl] octadecanoate

C53H98O6 (830.7363008)

[3-decanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-hexacos-15-enoate

C53H98O6 (830.7363008)

(3-decanoyloxy-2-dodecanoyloxypropyl) (17Z,20Z)-octacosa-17,20-dienoate

C53H98O6 (830.7363008)

2,3-bis[[(Z)-tridec-9-enoyl]oxy]propyl tetracosanoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-undecanoyloxypropyl] tricosanoate

C53H98O6 (830.7363008)

[3-dodecanoyloxy-2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxypropyl] henicosanoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-9-enoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] nonadecanoate

C53H98O6 (830.7363008)

[2-[(Z)-octadec-9-enoyl]oxy-3-undecanoyloxypropyl] (Z)-henicos-11-enoate

C53H98O6 (830.7363008)

[2-[(Z)-hexadec-9-enoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] henicosanoate

C53H98O6 (830.7363008)

2,3-di(tetradecanoyloxy)propyl (13Z,16Z)-docosa-13,16-dienoate

C53H98O6 (830.7363008)

[3-dodecanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-tetracos-13-enoate

C53H98O6 (830.7363008)

(2-heptadecanoyloxy-3-undecanoyloxypropyl) (13Z,16Z)-docosa-13,16-dienoate

C53H98O6 (830.7363008)

(2-octadecanoyloxy-3-tridecanoyloxypropyl) (9Z,12Z)-nonadeca-9,12-dienoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-nonadeca-9,12-dienoyl]oxy-3-undecanoyloxypropyl] icosanoate

C53H98O6 (830.7363008)

[3-dodecanoyloxy-2-[(Z)-heptadec-9-enoyl]oxypropyl] (Z)-henicos-11-enoate

C53H98O6 (830.7363008)

[1-decanoyloxy-3-[(11Z,14Z)-icosa-11,14-dienoyl]oxypropan-2-yl] icosanoate

C53H98O6 (830.7363008)

[3-decanoyloxy-2-[(Z)-hexadec-9-enoyl]oxypropyl] (Z)-tetracos-13-enoate

C53H98O6 (830.7363008)

(3-decanoyloxy-2-octadecanoyloxypropyl) (13Z,16Z)-docosa-13,16-dienoate

C53H98O6 (830.7363008)

[3-decanoyloxy-2-[(Z)-nonadec-9-enoyl]oxypropyl] (Z)-henicos-11-enoate

C53H98O6 (830.7363008)

[3-tetradecanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-docos-13-enoate

C53H98O6 (830.7363008)

[3-dodecanoyloxy-2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxypropyl] docosanoate

C53H98O6 (830.7363008)

[2-hexadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (Z)-henicos-11-enoate

C53H98O6 (830.7363008)

(2-heptadecanoyloxy-3-tetradecanoyloxypropyl) (9Z,12Z)-nonadeca-9,12-dienoate

C53H98O6 (830.7363008)

(2-hexadecanoyloxy-3-tridecanoyloxypropyl) (11Z,14Z)-henicosa-11,14-dienoate

C53H98O6 (830.7363008)

(2-pentadecanoyloxy-3-tetradecanoyloxypropyl) (11Z,14Z)-henicosa-11,14-dienoate

C53H98O6 (830.7363008)

[2-pentadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (Z)-docos-13-enoate

C53H98O6 (830.7363008)

[3-dodecanoyloxy-2-[(Z)-octadec-9-enoyl]oxypropyl] (Z)-icos-11-enoate

C53H98O6 (830.7363008)

(3-decanoyloxy-2-nonadecanoyloxypropyl) (11Z,14Z)-henicosa-11,14-dienoate

C53H98O6 (830.7363008)

2,3-di(dodecanoyloxy)propyl (15Z,18Z)-hexacosa-15,18-dienoate

C53H98O6 (830.7363008)

[3-decanoyloxy-2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxypropyl] tetracosanoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] (Z)-nonadec-9-enoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-tetradecanoyloxypropyl] nonadecanoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-9-enoyl]oxy-3-hexadecanoyloxypropyl] (Z)-heptadec-9-enoate

C53H98O6 (830.7363008)

(2-hexadecanoyloxy-3-pentadecanoyloxypropyl) (9Z,12Z)-nonadeca-9,12-dienoate

C53H98O6 (830.7363008)

[2-heptadecanoyloxy-3-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxypropyl] heptadecanoate

C53H98O6 (830.7363008)

[1-[(Z)-heptadec-9-enoyl]oxy-3-[(Z)-hexadec-9-enoyl]oxypropan-2-yl] heptadecanoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-pentadecanoyloxypropyl] octadecanoate

C53H98O6 (830.7363008)

(2-heptadecanoyloxy-3-pentadecanoyloxypropyl) (9Z,12Z)-octadeca-9,12-dienoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-9-enoyl]oxy-3-pentadecanoyloxypropyl] (Z)-octadec-9-enoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-pentadecanoyloxypropyl] nonadecanoate

C53H98O6 (830.7363008)

[2-[(Z)-hexadec-9-enoyl]oxy-3-pentadecanoyloxypropyl] (Z)-nonadec-9-enoate

C53H98O6 (830.7363008)

2,3-bis[[(Z)-hexadec-9-enoyl]oxy]propyl octadecanoate

C53H98O6 (830.7363008)

[2-heptadecanoyloxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] (Z)-octadec-9-enoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-9-enoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] octadecanoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-hexadecanoyloxypropyl] octadecanoate

C53H98O6 (830.7363008)

[2-[(Z)-hexadec-9-enoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] nonadecanoate

C53H98O6 (830.7363008)

[1-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-hexadecanoyloxypropan-2-yl] heptadecanoate

C53H98O6 (830.7363008)

[3-Hydroxy-2-(pentatriacontanoylamino)octyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[2-(Hexacosanoylamino)-3-hydroxyheptadecyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(pentacosanoylamino)octadecyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(tetracosanoylamino)nonadecyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(tricosanoylamino)icosyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[2-(Docosanoylamino)-3-hydroxyhenicosyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[2-(Henicosanoylamino)-3-hydroxydocosyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(octacosanoylamino)pentadecyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[2-(Hexadecanoylamino)-3-hydroxyheptacosyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(tetradecanoylamino)nonacosyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[2-(Heptacosanoylamino)-3-hydroxyhexadecyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[2-(Heptadecanoylamino)-3-hydroxyhexacosyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(icosanoylamino)tricosyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(nonacosanoylamino)tetradecyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(pentadecanoylamino)octacosyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(octadecanoylamino)pentacosyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(nonadecanoylamino)tetracosyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[3-Hydroxy-2-(tridecanoylamino)triacontyl] 2-(trimethylazaniumyl)ethyl phosphate

C48H99N2O6P (830.7240363999999)

[2-hexadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-icos-11-enoate

C53H98O6 (830.7363008)

[2-hexadecanoyloxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] (Z)-nonadec-9-enoate

C53H98O6 (830.7363008)

[2-heptadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-nonadec-9-enoate

C53H98O6 (830.7363008)

[2-[(Z)-pentadec-9-enoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] henicosanoate

C53H98O6 (830.7363008)

[3-dodecanoyloxy-2-[(Z)-nonadec-9-enoyl]oxypropyl] (Z)-nonadec-9-enoate

C53H98O6 (830.7363008)

(2-hexadecanoyloxy-3-tetradecanoyloxypropyl) (11Z,14Z)-icosa-11,14-dienoate

C53H98O6 (830.7363008)

2,3-bis[[(Z)-tetradec-9-enoyl]oxy]propyl docosanoate

C53H98O6 (830.7363008)

[3-pentadecanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] (Z)-icos-11-enoate

C53H98O6 (830.7363008)

2,3-di(pentadecanoyloxy)propyl (11Z,14Z)-icosa-11,14-dienoate

C53H98O6 (830.7363008)

(2-heptadecanoyloxy-3-tridecanoyloxypropyl) (11Z,14Z)-icosa-11,14-dienoate

C53H98O6 (830.7363008)

(3-dodecanoyloxy-2-octadecanoyloxypropyl) (11Z,14Z)-icosa-11,14-dienoate

C53H98O6 (830.7363008)

2,3-bis[[(Z)-pentadec-9-enoyl]oxy]propyl icosanoate

C53H98O6 (830.7363008)

[2-[(Z)-hexadec-7-enoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] icosanoate

C53H98O6 (830.7363008)

[3-dodecanoyloxy-2-[(Z)-octadec-11-enoyl]oxypropyl] (Z)-icos-11-enoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxy-3-pentadecanoyloxypropyl] icosanoate

C53H98O6 (830.7363008)

[2-hexadecanoyloxy-3-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] icosanoate

C53H98O6 (830.7363008)

[2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxy-3-pentadecanoyloxypropyl] nonadecanoate

C53H98O6 (830.7363008)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-nonadecanoyloxypropyl] nonadecanoate

C53H98O6 (830.7363008)

[2-heptadecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (Z)-icos-11-enoate

C53H98O6 (830.7363008)

[2-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxy-3-tridecanoyloxypropyl] icosanoate

C53H98O6 (830.7363008)

(2-octadecanoyloxy-3-tridecanoyloxypropyl) (7Z,9Z)-nonadeca-7,9-dienoate

C53H98O6 (830.7363008)

[2-octadecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (Z)-nonadec-9-enoate

C53H98O6 (830.7363008)

[2-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxy-3-tetradecanoyloxypropyl] nonadecanoate

C53H98O6 (830.7363008)

[3-[(Z)-dodec-5-enoyl]oxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] docosanoate

C53H98O6 (830.7363008)

[2-hexadecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (Z)-henicos-9-enoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-7-enoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] octadecanoate

C53H98O6 (830.7363008)

[3-[(Z)-dodec-5-enoyl]oxy-2-octadecanoyloxypropyl] (Z)-icos-11-enoate

C53H98O6 (830.7363008)

[3-dodecanoyloxy-2-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxypropyl] henicosanoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxy-3-tridecanoyloxypropyl] docosanoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-7-enoyl]oxy-3-tetradecanoyloxypropyl] (Z)-nonadec-9-enoate

C53H98O6 (830.7363008)

[2-[(Z)-octadec-11-enoyl]oxy-3-tridecanoyloxypropyl] (Z)-nonadec-9-enoate

C53H98O6 (830.7363008)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-octadecanoyloxypropyl] icosanoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-7-enoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] nonadecanoate

C53H98O6 (830.7363008)

[3-dodecanoyloxy-2-[(10Z,12Z)-octadeca-10,12-dienoyl]oxypropyl] icosanoate

C53H98O6 (830.7363008)

[2-[(Z)-hexadec-7-enoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] nonadecanoate

C53H98O6 (830.7363008)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-hexadecanoyloxypropyl] docosanoate

C53H98O6 (830.7363008)

(2-pentadecanoyloxy-3-tetradecanoyloxypropyl) (9Z,11Z)-henicosa-9,11-dienoate

C53H98O6 (830.7363008)

[2-octadecanoyloxy-3-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] octadecanoate

C53H98O6 (830.7363008)

[1-[(Z)-dodec-5-enoyl]oxy-3-[(Z)-nonadec-9-enoyl]oxypropan-2-yl] nonadecanoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-7-enoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] icosanoate

C53H98O6 (830.7363008)

[2-[(Z)-pentadec-9-enoyl]oxy-3-tridecanoyloxypropyl] (Z)-docos-11-enoate

C53H98O6 (830.7363008)

[1-[(Z)-heptadec-7-enoyl]oxy-3-[(Z)-hexadec-7-enoyl]oxypropan-2-yl] heptadecanoate

C53H98O6 (830.7363008)

[3-[(Z)-dodec-5-enoyl]oxy-2-hexadecanoyloxypropyl] (Z)-docos-11-enoate

C53H98O6 (830.7363008)

(3-dodecanoyloxy-2-hexadecanoyloxypropyl) (14Z,16Z)-docosa-14,16-dienoate

C53H98O6 (830.7363008)

[2-[(Z)-octadec-11-enoyl]oxy-3-tetradecanoyloxypropyl] (Z)-octadec-11-enoate

C53H98O6 (830.7363008)

[2-[(Z)-hexadec-7-enoyl]oxy-3-tridecanoyloxypropyl] (Z)-henicos-9-enoate

C53H98O6 (830.7363008)

2,3-di(hexadecanoyloxy)propyl (10Z,12Z)-octadeca-10,12-dienoate

C53H98O6 (830.7363008)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-heptadecanoyloxypropyl] henicosanoate

C53H98O6 (830.7363008)

[3-dodecanoyloxy-2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxypropyl] docosanoate

C53H98O6 (830.7363008)

[1-[(10Z,12Z)-octadeca-10,12-dienoyl]oxy-3-tetradecanoyloxypropan-2-yl] octadecanoate

C53H98O6 (830.7363008)

[2-pentadecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (Z)-docos-11-enoate

C53H98O6 (830.7363008)

[1-[(Z)-octadec-11-enoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropan-2-yl] octadecanoate

C53H98O6 (830.7363008)

2,3-di(tetradecanoyloxy)propyl (14Z,16Z)-docosa-14,16-dienoate

C53H98O6 (830.7363008)

(2-hexadecanoyloxy-3-pentadecanoyloxypropyl) (7Z,9Z)-nonadeca-7,9-dienoate

C53H98O6 (830.7363008)

[2-[(Z)-hexadec-7-enoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] henicosanoate

C53H98O6 (830.7363008)

[3-[(Z)-dodec-5-enoyl]oxy-2-heptadecanoyloxypropyl] (Z)-henicos-9-enoate

C53H98O6 (830.7363008)

[2-heptadecanoyloxy-3-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxypropyl] heptadecanoate

C53H98O6 (830.7363008)

[3-dodecanoyloxy-2-[(Z)-heptadec-7-enoyl]oxypropyl] (Z)-henicos-9-enoate

C53H98O6 (830.7363008)

[2-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxy-3-pentadecanoyloxypropyl] octadecanoate

C53H98O6 (830.7363008)

(2-heptadecanoyloxy-3-pentadecanoyloxypropyl) (10Z,12Z)-octadeca-10,12-dienoate

C53H98O6 (830.7363008)

[3-[(Z)-dodec-5-enoyl]oxy-2-[(Z)-heptadec-7-enoyl]oxypropyl] henicosanoate

C53H98O6 (830.7363008)

[3-tetradecanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-docos-11-enoate

C53H98O6 (830.7363008)

[2-[(Z)-pentadec-9-enoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] docosanoate

C53H98O6 (830.7363008)

[2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxy-3-tetradecanoyloxypropyl] henicosanoate

C53H98O6 (830.7363008)

[2-pentadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-henicos-9-enoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-7-enoyl]oxy-3-pentadecanoyloxypropyl] (Z)-octadec-11-enoate

C53H98O6 (830.7363008)

[2-[(Z)-hexadec-7-enoyl]oxy-3-tetradecanoyloxypropyl] (Z)-icos-11-enoate

C53H98O6 (830.7363008)

[2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxy-3-tetradecanoyloxypropyl] icosanoate

C53H98O6 (830.7363008)

[2-[(Z)-octadec-11-enoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] nonadecanoate

C53H98O6 (830.7363008)

[1-dodecanoyloxy-3-[(7Z,9Z)-nonadeca-7,9-dienoyl]oxypropan-2-yl] nonadecanoate

C53H98O6 (830.7363008)

[2-heptadecanoyloxy-3-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxypropyl] octadecanoate

C53H98O6 (830.7363008)

[3-dodecanoyloxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] (Z)-docos-11-enoate

C53H98O6 (830.7363008)

[2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxy-3-hexadecanoyloxypropyl] octadecanoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-7-enoyl]oxy-3-hexadecanoyloxypropyl] (Z)-heptadec-7-enoate

C53H98O6 (830.7363008)

[2-hexadecanoyloxy-3-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxypropyl] nonadecanoate

C53H98O6 (830.7363008)

[2-[(Z)-heptadec-7-enoyl]oxy-3-tridecanoyloxypropyl] (Z)-icos-11-enoate

C53H98O6 (830.7363008)

(3-dodecanoyloxy-2-heptadecanoyloxypropyl) (9Z,11Z)-henicosa-9,11-dienoate

C53H98O6 (830.7363008)

[3-[(Z)-dodec-5-enoyl]oxy-2-[(Z)-octadec-11-enoyl]oxypropyl] icosanoate

C53H98O6 (830.7363008)

[3-hexadecanoyloxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] (Z)-octadec-11-enoate

C53H98O6 (830.7363008)

(2-heptadecanoyloxy-3-tetradecanoyloxypropyl) (7Z,9Z)-nonadeca-7,9-dienoate

C53H98O6 (830.7363008)

[2-pentadecanoyloxy-3-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] henicosanoate

C53H98O6 (830.7363008)

[2-heptadecanoyloxy-3-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] nonadecanoate

C53H98O6 (830.7363008)

[2-[(Z)-hexadec-7-enoyl]oxy-3-pentadecanoyloxypropyl] (Z)-nonadec-9-enoate

C53H98O6 (830.7363008)

(2-pentadecanoyloxy-3-tridecanoyloxypropyl) (14Z,16Z)-docosa-14,16-dienoate

C53H98O6 (830.7363008)

[2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxy-3-tridecanoyloxypropyl] henicosanoate

C53H98O6 (830.7363008)

[2-[(10Z,12Z)-octadeca-10,12-dienoyl]oxy-3-tridecanoyloxypropyl] nonadecanoate

C53H98O6 (830.7363008)

[2-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxy-3-tetradecanoyloxypropyl] docosanoate

C53H98O6 (830.7363008)

[2-heptadecanoyloxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] (Z)-octadec-11-enoate

C53H98O6 (830.7363008)

[2-[(Z)-pentadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] (Z)-henicos-9-enoate

C53H98O6 (830.7363008)

2,3-bis[[(Z)-hexadec-7-enoyl]oxy]propyl octadecanoate

C53H98O6 (830.7363008)

[1-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxy-3-hexadecanoyloxypropan-2-yl] heptadecanoate

C53H98O6 (830.7363008)

(2-hexadecanoyloxy-3-tridecanoyloxypropyl) (9Z,11Z)-henicosa-9,11-dienoate

C53H98O6 (830.7363008)

N-(hexacosanoyl)-heptadecasphinganine-1-phosphocholine

C48H99N2O6P (830.7240363999999)

N-(tricosanoyl)-eicosasphinganine-1-phosphocholine

C48H99N2O6P (830.7240363999999)

N-(pentacosanoyl)-sphinganine-1-phosphocholine

C48H99N2O6P (830.7240363999999)

N-(heneicosanoyl)-docosasphinganine-1-phosphocholine

C48H99N2O6P (830.7240363999999)

N-(docosanoyl)-heneicosasphinganine-1-phosphocholine

C48H99N2O6P (830.7240363999999)

N-(tetracosanoyl)-nonadecasphinganine-1-phosphocholine

C48H99N2O6P (830.7240363999999)

1-O,2-O-Dipalmitoyl-3-O-linoleoyl-L-glycerol

C53H98O6 (830.7363008)

[(2R)-3-hexadecanoyloxy-2-[(E)-hexadec-9-enoyl]oxypropyl] (E)-octadec-11-enoate

C53H98O6 (830.7363008)

[(2R)-2-[(E)-heptadec-9-enoyl]oxy-3-hexadecanoyloxypropyl] (E)-heptadec-9-enoate

C53H98O6 (830.7363008)

2-[[(2R)-3-[(E)-hexadec-1-enoxy]-2-tetracosanoyloxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

[(2S)-2-[(E)-hexadec-9-enoyl]oxy-3-[(E)-tetradec-9-enoyl]oxypropyl] icosanoate

C53H98O6 (830.7363008)

[(2R)-1-[(E)-heptadec-9-enoyl]oxy-3-[(E)-hexadec-9-enoyl]oxypropan-2-yl] heptadecanoate

C53H98O6 (830.7363008)

[2-heptadecanoyloxy-3-[(4E,7E)-hexadeca-4,7-dienoyl]oxypropyl] heptadecanoate

C53H98O6 (830.7363008)

2-[[(2R)-2-docosanoyloxy-3-[(E)-octadec-1-enoxy]propoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

[(2S)-2,3-bis[[(E)-hexadec-9-enoyl]oxy]propyl] octadecanoate

C53H98O6 (830.7363008)

[2-[(4E,7E)-hexadeca-4,7-dienoyl]oxy-3-hexadecanoyloxypropyl] octadecanoate

C53H98O6 (830.7363008)

2-[hydroxy-[(2R)-2-icosanoyloxy-3-[(E)-icos-1-enoxy]propoxy]phosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

[(2R)-1-[(9E,12E)-heptadeca-9,12-dienoyl]oxy-3-hexadecanoyloxypropan-2-yl] heptadecanoate

C53H98O6 (830.7363008)

2-[[3-hexadecoxy-2-[(Z)-tetracos-13-enoyl]oxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[2-[(Z)-hexacos-15-enoyl]oxy-3-tetradecoxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[2-hexacosanoyloxy-3-[(Z)-tetradec-9-enoxy]propoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[3-[(Z)-hexadec-9-enoxy]-2-tetracosanoyloxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[2-[(Z)-docos-13-enoyl]oxy-3-octadecoxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[2-docosanoyloxy-3-[(Z)-octadec-9-enoxy]propoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[3-dodecoxy-2-[(Z)-octacos-17-enoyl]oxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[hydroxy-[2-hydroxy-3-[(Z)-tetracont-29-enoyl]oxypropoxy]phosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[3-[(Z)-heptadec-9-enoxy]-2-tricosanoyloxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[hydroxy-[2-[(Z)-icos-11-enoyl]oxy-3-icosoxypropoxy]phosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[2-heptacosanoyloxy-3-[(Z)-tridec-9-enoxy]propoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[3-heptacosoxy-2-[(Z)-tridec-9-enoyl]oxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[3-[(Z)-docos-13-enoxy]-2-octadecanoyloxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[2-hexadecanoyloxy-3-[(Z)-tetracos-13-enoxy]propoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[2-[(Z)-hexadec-9-enoyl]oxy-3-tetracosoxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[3-[(Z)-henicos-11-enoxy]-2-nonadecanoyloxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[3-henicosoxy-2-[(Z)-nonadec-9-enoyl]oxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[3-[(Z)-hexacos-15-enoxy]-2-tetradecanoyloxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[3-hexacosoxy-2-[(Z)-tetradec-9-enoyl]oxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[2-[(Z)-henicos-11-enoyl]oxy-3-nonadecoxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[3-docosoxy-2-[(Z)-octadec-9-enoyl]oxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[hydroxy-[3-pentacosoxy-2-[(Z)-pentadec-9-enoyl]oxypropoxy]phosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[hydroxy-[2-pentacosanoyloxy-3-[(Z)-pentadec-9-enoxy]propoxy]phosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[hydroxy-[2-icosanoyloxy-3-[(Z)-icos-11-enoxy]propoxy]phosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[2-dodecanoyloxy-3-[(Z)-octacos-17-enoxy]propoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[2-[(Z)-heptadec-9-enoyl]oxy-3-tricosoxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

2-[[2-henicosanoyloxy-3-[(Z)-nonadec-9-enoxy]propoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C48H97NO7P+ (830.7002281999999)

TG(16:1(9Z)/16:1(9Z)/18:0)[iso3]

C53H98O6 (830.7363008)

1,3-Dipalmitoyl-2-linoleoylglycerol

C53H98O6 (830.7363008)

1,2-Dioleoyl-3-myristoyl-sn-glycerol

C53H98O6 (830.7363008)

A triacyl-sn-glycerol in which the acyl groups at positions 1 and 2 are specified as oleoyl while that at position 3 is specifed as myristoyl.

1,2-di-(9Z-tetradecenoyl)-3-docosanoyl-sn-glycerol

C53H98O6 (830.7363008)

TG(18:1(11Z)/14:0/18:1(11Z))[iso3]

C53H98O6 (830.7363008)

1-(9Z-tetradecenoyl)-2-(9Z-hexadecenoyl)-3-eicosanoyl-sn-glycerol

C53H98O6 (830.7363008)

Glycerol 1,3-dihexadecanoate 2-(9Z,12Z-octadecadienoate)

C53H98O6 (830.7363008)

Glycerol 1,2-di-(9Z-octadecenoate) 3-tetradecanoate

C53H98O6 (830.7363008)

1-Palmitoyl-2-oleoyl-3-palmitoleoyl-glycerol

C53H98O6 (830.7363008)

1-Palmitoleoyl-2-palmitoyl-3-oleoyl-glycerol

C53H98O6 (830.7363008)

1-Myristoyl-2-linoleoyl-3-stearoyl-glycerol

C53H98O6 (830.7363008)

1-Stearoyl-2-myristoleoyl-3-oleoyl-glycerol

C53H98O6 (830.7363008)

1-Stearoyl-2-oleoyl-3-myristoleoyl-glycerol

C53H98O6 (830.7363008)

1-Arachidonyl-2-myristoleoyl-3-palmitoleoyl-glycerol

C53H98O6 (830.7363008)

1-Myristoleoyl-2-arachidonyl-3-palmitoleoyl-glycerol

C53H98O6 (830.7363008)

1-Myristoleoyl-2-behenoyl-3-myristoleoyl-glycerol

C53H98O6 (830.7363008)

1-Palmitoleoyl-2-stearoyl-3-palmitoleoyl-glycerol

C53H98O6 (830.7363008)

1-Myristoyl-2-myristoleoyl-3-erucoyl-glycerol

C53H98O6 (830.7363008)

1-Myristoyl-2-vaccenoyl-3-vaccenoyl-glycerol

C53H98O6 (830.7363008)

1-Myristoyl-2-vaccenoyl-3-oleoyl-glycerol

C53H98O6 (830.7363008)

1-Myristoyl-2-oleoyl-3-vaccenoyl-glycerol

C53H98O6 (830.7363008)

1-Myristoyl-2-eicosenoyl-3-palmitoleoyl-glycerol

C53H98O6 (830.7363008)

1-Myristoyl-2-erucoyl-3-myristoleoyl-glycerol

C53H98O6 (830.7363008)

1-Myristoyl-2-docosadienoyl-3-myristoyl-glycerol

C53H98O6 (830.7363008)

1-Palmitoyl-2-myristoleoyl-3-eicosenoyl-glycerol

C53H98O6 (830.7363008)

1-Palmitoyl-2-palmitoleoyl-3-vaccenoyl-glycerol

C53H98O6 (830.7363008)

1-Palmitoyl-2-vaccenoyl-3-palmitoleoyl-glycerol

C53H98O6 (830.7363008)

1-Palmitoyl-2-eicosenoyl-3-myristoleoyl-glycerol

C53H98O6 (830.7363008)

1-Stearoyl-2-myristoleoyl-3-vaccenoyl-glycerol

C53H98O6 (830.7363008)

1-Stearoyl-2-vaccenoyl-3-myristoleoyl-glycerol

C53H98O6 (830.7363008)

1-Myristoleoyl-2-myristoyl-3-erucoyl-glycerol

C53H98O6 (830.7363008)

1-Myristoleoyl-2-stearoyl-3-vaccenoyl-glycerol

C53H98O6 (830.7363008)

1-Palmitoleoyl-2-myristoyl-3-eicosenoyl-glycerol

C53H98O6 (830.7363008)

Glycerol 1,2-dihexadecanoate 3-(9Z,12Z-octadecadienoate)

C53H98O6 (830.7363008)

1-Myristoyl-2-palmitoyl-3-eicosadienoyl-glycerol

C53H98O6 (830.7363008)

1-Myristoyl-2-eicosadienoyl-3-palmitoyl-glycerol

C53H98O6 (830.7363008)

1-Pentadecanoyl-2-pentadecanoyl-3-eicosadienoyl-glycerol

C53H98O6 (830.7363008)

1-Pentadecanoyl-2-eicosadienoyl-3-pentadecanoyl-glycerol

C53H98O6 (830.7363008)

1-Palmitoyl-2-myristoyl-3-eicosadienoyl-glycerol

C53H98O6 (830.7363008)

1-stearoyl-2-myristoyl-3-linoleoyl-glycerol

C53H98O6 (830.7363008)

TG(16:0/16:0/18:2(9Z,12Z))[iso3]

C53H98O6 (830.7363008)

1-hexadecanoyl-2-[(9Z)-hexadecenoyl]-3-[(9Z)-octadecenoyl]-sn-glycerol

C53H98O6 (830.7363008)

A triacyl-sn-glycerol in which the which the acyl groups at positions 1, 2 and 3 are specified as hexadecanoyl, (9Z)-hexadecenoyl and (9Z)-octadecenoyl respectively.

SM(43:0)

C48H99N2O6P (830.7240363999999)

Provides by LipidSearch Vendor. © Copyright 2006-2024 Thermo Fisher Scientific Inc. All rights reserved

FAHFA 56:8;O

C56H94O4 (830.7151723999999)

PA O-14:0/32:0

C49H99O7P (830.7128034)

PA O-16:0/30:0

C49H99O7P (830.7128034)

PA O-18:0/28:0

C49H99O7P (830.7128034)

PA O-20:0/26:0

C49H99O7P (830.7128034)

PA O-22:0/24:0

C49H99O7P (830.7128034)

PA O-46:0

C49H99O7P (830.7128034)

CerPE 14:0;O2/32:0

C48H99N2O6P (830.7240363999999)

CerPE 15:0;O2/31:0

C48H99N2O6P (830.7240363999999)

CerPE 16:0;O2/30:0

C48H99N2O6P (830.7240363999999)

CerPE 17:0;O2/29:0

C48H99N2O6P (830.7240363999999)

CerPE 18:0;O2/28:0

C48H99N2O6P (830.7240363999999)

CerPE 19:0;O2/27:0

C48H99N2O6P (830.7240363999999)

CerPE 20:0;O2/26:0

C48H99N2O6P (830.7240363999999)

CerPE 21:0;O2/25:0

C48H99N2O6P (830.7240363999999)

CerPE 22:0;O2/24:0

C48H99N2O6P (830.7240363999999)

CerPE 46:0;O2

C48H99N2O6P (830.7240363999999)

SM 14:0;O2/29:0

C48H99N2O6P (830.7240363999999)

SM 15:0;O2/28:0

C48H99N2O6P (830.7240363999999)

SM 16:0;O2/27:0

C48H99N2O6P (830.7240363999999)

SM 17:0;O2/26:0

C48H99N2O6P (830.7240363999999)

SM 18:0;O2/25:0

C48H99N2O6P (830.7240363999999)

SM 19:0;O2/24:0

C48H99N2O6P (830.7240363999999)

SM 20:0;O2/23:0

C48H99N2O6P (830.7240363999999)

SM 21:0;O2/22:0

C48H99N2O6P (830.7240363999999)