Exact Mass: 814.681505
Exact Mass Matches: 814.681505
Found 500 metabolites which its exact mass value is equals to given mass value 814.681505,
within given mass tolerance error 0.05 dalton. Try search metabolite list with more accurate mass tolerance error
0.01 dalton.
SM(d18:1/24:0)
Sphingomyelin (d18:1/24:0) or SM(d18:1/24:0) is a type of sphingolipid found in animal cell membranes, especially in the membranous myelin sheath which surrounds some nerve cell axons. It usually consists of phosphorylcholine and ceramide. SM(d18:1/24:0) consists of a sphingosine backbone and a lignoceric acid chain. In humans, sphingomyelin is the only membrane phospholipid not derived from glycerol. Like all sphingolipids, SM has a ceramide core (sphingosine bonded to a fatty acid via an amide linkage). In addition, it contains one polar head group, which is either phosphocholine or phosphoethanolamine. The plasma membrane of cells is highly enriched in sphingomyelin and is considered largely to be found in the exoplasmic leaflet of the cell membrane. However, there is some evidence that there may also be a sphingomyelin pool in the inner leaflet of the membrane. Moreover, neutral sphingomyelinase-2, an enzyme that breaks down sphingomyelin into ceramide, has been found to localize exclusively to the inner leaflet further suggesting that there may be sphingomyelin present there. Sphingomyelin can accumulate in a rare hereditary disease called Niemann-Pick Disease, types A and B. Niemann-Pick disease is a genetically-inherited disease caused by a deficiency in the enzyme sphingomyelinase, which causes the accumulation of sphingomyelin in spleen, liver, lungs, bone marrow, and the brain, causing irreversible neurological damage. SMs play a role in signal transduction. Sphingomyelins are synthesized by the transfer of phosphorylcholine from phosphatidylcholine to a ceramide in a reaction catalyzed by sphingomyelin synthase.
Sitoindoside I
Sitoindoside I is found in fruits. Sitoindoside I is a constituent of fruits of banana (Musa paradisiaca) Constituent of fruits of banana (Musa paradisiaca). Sitoindoside I is found in spearmint and fruits.
SM(d18:0/24:1(15Z))
Sphingomyelin (d18:0/24:1(15Z)) or SM(d18:0/24:1(15Z)) is a type of sphingolipid found in animal cell membranes, especially in the membranous myelin sheath which surrounds some nerve cell axons. It usually consists of phosphorylcholine and ceramide. SM(d18:0/24:1(15Z)) consists of a sphinganine backbone and a 15Z-tetracosenoic acid chain. In humans, sphingomyelin is the only membrane phospholipid not derived from glycerol. Like all sphingolipids, SM has a ceramide core (sphingosine bonded to a fatty acid via an amide linkage). In addition, it contains one polar head group, which is either phosphocholine or phosphoethanolamine. The plasma membrane of cells is highly enriched in sphingomyelin and is considered largely to be found in the exoplasmic leaflet of the cell membrane. However, there is some evidence that there may also be a sphingomyelin pool in the inner leaflet of the membrane. Moreover, neutral sphingomyelinase-2, an enzyme that breaks down sphingomyelin into ceramide, has been found to localize exclusively to the inner leaflet further suggesting that there may be sphingomyelin present there. Sphingomyelin can accumulate in a rare hereditary disease called Niemann-Pick Disease, types A and B. Niemann-Pick disease is a genetically-inherited disease caused by a deficiency in the enzyme sphingomyelinase, which causes the accumulation of sphingomyelin in spleen, liver, lungs, bone marrow, and the brain, causing irreversible neurological damage. SMs play a role in signal transduction. Sphingomyelins are synthesized by the transfer of phosphorylcholine from phosphatidylcholine to a ceramide in a reaction catalyzed by sphingomyelin synthase. Sphingomyelin (d18:0/24:1(15Z)) or SM(d18:0/24:1(15Z)) is a type of sphingolipid found in animal cell membranes, especially in the membranous myelin sheath which surrounds some nerve cell axons. It usually consists of phosphorylcholine and ceramide. In humans, sphingomyelin is the only membrane phospholipid not derived from glycerol. Like all sphingolipids, SPH has a ceramide core (sphingosine bonded to a fatty acid via an amide linkage). In addition it contains one polar head group, which is either phosphocholine or phosphoethanolamine. The plasma membrane of cells is highly enriched in sphingomyelin and is considered largely to be found in the exoplasmic leaflet of the cell membrane. However, there is some evidence that there may also be a sphingomyelin pool in the inner leaflet of the membrane. Moreover, neutral sphingomyelinase-2 - an enzyme that breaks down sphingomyelin into ceramide has been found to localise exclusively to the inner leaflet further suggesting that there may be sphingomyelin present there. Sphingomyelin can accumulate in a rare hereditary disease called Niemann-Pick Disease, types A and B. Niemann-Pick disease is a genetically-inherited disease caused by a deficiency in the enzyme Sphingomyelinase, which causes the accumulation of Sphingomyelin in spleen, liver, lungs, bone marrow, and the brain, causing irreversible neurological damage. SMs play a role in signal transduction.
TG(14:0/15:0/20:3(5Z,8Z,11Z))
TG(14:0/15:0/20:3(5Z,8Z,11Z)) is a monomead acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/15:0/20:3(5Z,8Z,11Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of mead acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(14:0/15:0/20:3n6)
TG(14:0/15:0/20:3n6) is a monohomo-g-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/15:0/20:3n6), in particular, consists of one chain of myristic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of homo-g-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(14:0/20:3(5Z,8Z,11Z)/15:0)
TG(14:0/20:3(5Z,8Z,11Z)/15:0) is a monomead acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/20:3(5Z,8Z,11Z)/15:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of mead acid at the C-2 position and one chain of pentadecanoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(14:0/20:3n6/15:0)
TG(14:0/20:3n6/15:0) is a monohomo-g-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/20:3n6/15:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of homo-g-linolenic acid at the C-2 position and one chain of pentadecanoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(15:0/14:0/20:3(5Z,8Z,11Z))
TG(15:0/14:0/20:3(5Z,8Z,11Z)) is a monomead acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/14:0/20:3(5Z,8Z,11Z)), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of mead acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(15:0/14:0/20:3n6)
TG(15:0/14:0/20:3n6) is a monohomo-g-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/14:0/20:3n6), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of homo-g-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(15:0/16:0/18:3(6Z,9Z,12Z))
TG(15:0/16:0/18:3(6Z,9Z,12Z)) is a monog-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/16:0/18:3(6Z,9Z,12Z)), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of g-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(15:0/16:0/18:3(9Z,12Z,15Z))
TG(15:0/16:0/18:3(9Z,12Z,15Z)) is a monoa-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/16:0/18:3(9Z,12Z,15Z)), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of a-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(15:0/14:1(9Z)/20:2n6)
TG(15:0/14:1(9Z)/20:2n6) is a monoeicosadienoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/14:1(9Z)/20:2n6), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of eicosadienoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(15:0/16:1(9Z)/18:2(9Z,12Z))
TG(15:0/16:1(9Z)/18:2(9Z,12Z)) is a monolinoleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/16:1(9Z)/18:2(9Z,12Z)), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of linoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(15:0/18:2(9Z,12Z)/16:1(9Z))
TG(15:0/18:2(9Z,12Z)/16:1(9Z)) is a monolinoleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/18:2(9Z,12Z)/16:1(9Z)), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of linoleic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(15:0/18:3(6Z,9Z,12Z)/16:0)
TG(15:0/18:3(6Z,9Z,12Z)/16:0) is a monog-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/18:3(6Z,9Z,12Z)/16:0), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of g-linolenic acid at the C-2 position and one chain of palmitic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(15:0/20:2n6/14:1(9Z))
TG(15:0/20:2n6/14:1(9Z)) is a monoeicosadienoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/20:2n6/14:1(9Z)), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of eicosadienoic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(15:0/18:3(9Z,12Z,15Z)/16:0)
TG(15:0/18:3(9Z,12Z,15Z)/16:0) is a monoa-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/18:3(9Z,12Z,15Z)/16:0), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of a-linolenic acid at the C-2 position and one chain of palmitic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(16:0/15:0/18:3(6Z,9Z,12Z))
TG(16:0/15:0/18:3(6Z,9Z,12Z)) is a monog-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/15:0/18:3(6Z,9Z,12Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of g-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(16:0/15:0/18:3(9Z,12Z,15Z))
TG(16:0/15:0/18:3(9Z,12Z,15Z)) is a monoa-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/15:0/18:3(9Z,12Z,15Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of a-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(14:1(9Z)/15:0/20:2n6)
TG(14:1(9Z)/15:0/20:2n6) is a monoeicosadienoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/15:0/20:2n6), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of eicosadienoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
TG(16:1(9Z)/15:0/18:2(9Z,12Z))
TG(16:1(9Z)/15:0/18:2(9Z,12Z)) is a monolinoleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:1(9Z)/15:0/18:2(9Z,12Z)), in particular, consists of one chain of palmitoleic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of linoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.
Sitosterol 3-O-(6'-O-palmitoyl-beta-D-glucoside)
Sitosterol 3-o-(6-o-palmitoyl-beta-d-glucoside) is practically insoluble (in water) and a very weakly acidic compound (based on its pKa). Sitosterol 3-o-(6-o-palmitoyl-beta-d-glucoside) can be found in fig, which makes sitosterol 3-o-(6-o-palmitoyl-beta-d-glucoside) a potential biomarker for the consumption of this food product.
SM d42:1
Found in mouse lung; TwoDicalId=16; MgfFile=160901_Lung_AA_Neg_20; MgfId=1683 Found in mouse kidney; TwoDicalId=5; MgfFile=160827_Kidney_DHA_Neg_14; MgfId=2091 Found in mouse lung; TwoDicalId=402; MgfFile=160901_Lung_normal_Neg_02; MgfId=1534 Found in mouse spleen; TwoDicalId=892; MgfFile=160729_spleen_normal_05_Neg; MgfId=1617
beta-sitosteryl-3beta-glucopyranoside-6-O-palmitate
TG(14:0/15:0/20:3(8Z,11Z,14Z))[iso6]
TG(14:0/15:1(9Z)/20:2(11Z,14Z))[iso6]
TG(14:0/17:0/18:3(6Z,9Z,12Z))[iso6]
TG(14:0/17:0/18:3(9Z,12Z,15Z))[iso6]
TG(14:0/17:1(9Z)/18:2(9Z,12Z))[iso6]
TG(14:0/17:2(9Z,12Z)/18:1(9Z))[iso6]
TG(14:1(9Z)/15:0/20:2(11Z,14Z))[iso6]
TG(14:1(9Z)/15:1(9Z)/20:1(11Z))[iso6]
TG(14:1(9Z)/16:1(9Z)/19:1(9Z))[iso6]
TG(14:1(9Z)/17:0/18:2(9Z,12Z))[iso6]
TG(14:1(9Z)/17:1(9Z)/18:1(9Z))[iso6]
TG(14:1(9Z)/17:2(9Z,12Z)/18:0)[iso6]
TG(15:0/16:0/18:3(6Z,9Z,12Z))[iso6]
TG(15:0/16:0/18:3(9Z,12Z,15Z))[iso6]
TG(15:0/16:1(9Z)/18:2(9Z,12Z))[iso6]
TG(15:0/17:1(9Z)/17:2(9Z,12Z))[iso6]
TG(15:1(9Z)/16:0/18:2(9Z,12Z))[iso6]
TG(15:1(9Z)/16:1(9Z)/18:1(9Z))[iso6]
TG(15:1(9Z)/17:0/17:2(9Z,12Z))[iso6]
Sitoindoside I
A steroid saponin that is sitosterol attached to a 6-O-hexadecanoyl-beta-D-glucopyranosyl residue at position 3 via a glycosidic linkage. It has been isolated from Breynia fruticosa.
TG(12:0/15:0/22:3(10Z,13Z,16Z))[iso6]
TG(12:0/15:1(9Z)/22:2(13Z,16Z))[iso6]
TG(12:0/17:0/20:3(8Z,11Z,14Z))[iso6]
TG(12:0/17:1(9Z)/20:2(11Z,14Z))[iso6]
TG(12:0/17:2(9Z,12Z)/20:1(11Z))[iso6]
TG(12:0/18:2(9Z,12Z)/19:1(9Z))[iso6]
TG(12:0/18:3(6Z,9Z,12Z)/19:0)[iso6]
TG(12:0/18:3(9Z,12Z,15Z)/19:0)[iso6]
TG(13:0/14:0/22:3(10Z,13Z,16Z))[iso6]
TG(13:0/14:1(9Z)/22:2(13Z,16Z))[iso6]
TG(13:0/16:0/20:3(8Z,11Z,14Z))[iso6]
TG(13:0/16:1(9Z)/20:2(11Z,14Z))[iso6]
TG(13:0/17:2(9Z,12Z)/19:1(9Z))[iso6]
TG(13:0/18:0/18:3(6Z,9Z,12Z))[iso6]
TG(13:0/18:0/18:3(9Z,12Z,15Z))[iso6]
TG(13:0/18:1(9Z)/18:2(9Z,12Z))[iso6]
[3-hydroxy-2-[[(Z)-tetracos-11-enoyl]amino]octadecyl] 2-(trimethylazaniumyl)ethyl phosphate
N-lignoceroyl-D-erythro-sphingosylphosphorylcholine
1-Pentadecanoyl-2-eicosadienoyl-3-myristoleoyl-glycerol
1-Pentadecanoyl-2-myristoyl-3-homo-g-linolenoyl-glycerol
N-pentacosanoyl-14-methylhexadecasphingosine-1-phosphocholine
A sphingomyelin 42:1 obtained by formal condensation of the carboxy group of pentacosanoic acid with the amino group of 14-methylhexadecasphingosine-1-phosphocholine. It is a metabolite of the nematode Caenorhabditis elegans.
N-pentacosanoyl-15-methylhexadecasphing-4-enine-1-phosphocholine
[1-hydroxy-3-[(16Z,19Z,22Z,25Z)-octacosa-16,19,22,25-tetraenoxy]propan-2-yl] (6Z,9Z,12Z,15Z,18Z,21Z)-tetracosa-6,9,12,15,18,21-hexaenoate
[(E)-2-(heptanoylamino)-3-hydroxypentatriacont-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[1-hydroxy-3-[(10Z,13Z,16Z)-tetracosa-10,13,16-trienoxy]propan-2-yl] (7Z,10Z,13Z,16Z,19Z,22Z,25Z)-octacosa-7,10,13,16,19,22,25-heptaenoate
[(E)-3-hydroxy-2-(pentanoylamino)heptatriacont-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[1-[(5Z,8Z,11Z,14Z,17Z,20Z,23Z)-hexacosa-5,8,11,14,17,20,23-heptaenoxy]-3-hydroxypropan-2-yl] (12Z,15Z,18Z)-hexacosa-12,15,18-trienoate
[1-hydroxy-3-[(13Z,16Z,19Z,22Z,25Z)-octacosa-13,16,19,22,25-pentaenoxy]propan-2-yl] (9Z,12Z,15Z,18Z,21Z)-tetracosa-9,12,15,18,21-pentaenoate
[1-hydroxy-3-[(12Z,15Z,18Z,21Z)-tetracosa-12,15,18,21-tetraenoxy]propan-2-yl] (10Z,13Z,16Z,19Z,22Z,25Z)-octacosa-10,13,16,19,22,25-hexaenoate
[(E)-3-hydroxy-2-(tetratriacontanoylamino)oct-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-3-hydroxy-2-(nonanoylamino)tritriacont-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[1-[(8Z,11Z,14Z,17Z,20Z,23Z)-hexacosa-8,11,14,17,20,23-hexaenoxy]-3-hydroxypropan-2-yl] (14Z,17Z,20Z,23Z)-hexacosa-14,17,20,23-tetraenoate
[1-[(12Z,15Z,18Z)-hexacosa-12,15,18-trienoxy]-3-hydroxypropan-2-yl] (5Z,8Z,11Z,14Z,17Z,20Z,23Z)-hexacosa-5,8,11,14,17,20,23-heptaenoate
[1-hydroxy-3-[(10Z,13Z,16Z,19Z,22Z,25Z)-octacosa-10,13,16,19,22,25-hexaenoxy]propan-2-yl] (12Z,15Z,18Z,21Z)-tetracosa-12,15,18,21-tetraenoate
[(E)-2-(hexanoylamino)-3-hydroxyhexatriacont-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-3-hydroxy-2-(tritriacontanoylamino)non-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[1-[(11Z,14Z,17Z,20Z,23Z)-hexacosa-11,14,17,20,23-pentaenoxy]-3-hydroxypropan-2-yl] (11Z,14Z,17Z,20Z,23Z)-hexacosa-11,14,17,20,23-pentaenoate
[1-hydroxy-3-[(7Z,10Z,13Z,16Z,19Z,22Z,25Z)-octacosa-7,10,13,16,19,22,25-heptaenoxy]propan-2-yl] (10Z,13Z,16Z)-tetracosa-10,13,16-trienoate
[1-hydroxy-3-[(6Z,9Z,12Z,15Z,18Z,21Z)-tetracosa-6,9,12,15,18,21-hexaenoxy]propan-2-yl] (16Z,19Z,22Z,25Z)-octacosa-16,19,22,25-tetraenoate
[1-[(14Z,17Z,20Z,23Z)-hexacosa-14,17,20,23-tetraenoxy]-3-hydroxypropan-2-yl] (8Z,11Z,14Z,17Z,20Z,23Z)-hexacosa-8,11,14,17,20,23-hexaenoate
[(E)-3-hydroxy-2-(octanoylamino)tetratriacont-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[1-hydroxy-3-[(9Z,12Z,15Z,18Z,21Z)-tetracosa-9,12,15,18,21-pentaenoxy]propan-2-yl] (13Z,16Z,19Z,22Z,25Z)-octacosa-13,16,19,22,25-pentaenoate
[(E)-2-(dotriacontanoylamino)-3-hydroxydec-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[3-hydroxy-2-[[(Z)-tetracos-13-enoyl]amino]octadecyl] 2-(trimethylazaniumyl)ethyl phosphate
[2-[[(Z)-henicos-11-enoyl]amino]-3-hydroxyhenicosyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-2-(decanoylamino)-3-hydroxydotriacont-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[2-[[(Z)-docos-13-enoyl]amino]-3-hydroxyicosyl] 2-(trimethylazaniumyl)ethyl phosphate
[2-[[(Z)-dotriacont-21-enoyl]amino]-3-hydroxydecyl] 2-(trimethylazaniumyl)ethyl phosphate
[3-hydroxy-2-[[(Z)-triacont-19-enoyl]amino]dodecyl] 2-(trimethylazaniumyl)ethyl phosphate
[3-hydroxy-2-[[(Z)-tridec-9-enoyl]amino]nonacosyl] 2-(trimethylazaniumyl)ethyl phosphate
[2-[[(Z)-hexacos-15-enoyl]amino]-3-hydroxyhexadecyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-3-hydroxy-2-(undecanoylamino)hentriacont-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[3-hydroxy-2-[[(Z)-octadec-9-enoyl]amino]tetracosyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-3-hydroxy-2-(triacontanoylamino)dodec-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[2-[[(Z)-hexadec-9-enoyl]amino]-3-hydroxyhexacosyl] 2-(trimethylazaniumyl)ethyl phosphate
[3-hydroxy-2-[[(Z)-octacos-17-enoyl]amino]tetradecyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-3-hydroxy-2-(nonacosanoylamino)tridec-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-2-(hentriacontanoylamino)-3-hydroxyundec-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[2-[[(Z)-heptadec-9-enoyl]amino]-3-hydroxypentacosyl] 2-(trimethylazaniumyl)ethyl phosphate
(2-nonanoyloxy-3-octanoyloxypropyl) (18Z,21Z,24Z)-dotriaconta-18,21,24-trienoate
(2-dodecanoyloxy-3-nonanoyloxypropyl) (14Z,17Z,20Z)-octacosa-14,17,20-trienoate
(2-hexadecanoyloxy-3-nonanoyloxypropyl) (10Z,13Z,16Z)-tetracosa-10,13,16-trienoate
[1-[(11Z,14Z)-icosa-11,14-dienoyl]oxy-3-nonanoyloxypropan-2-yl] (Z)-icos-11-enoate
[3-nonanoyloxy-2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxypropyl] (Z)-docos-13-enoate
(2-decanoyloxy-3-nonanoyloxypropyl) (16Z,19Z,22Z)-triaconta-16,19,22-trienoate
[2-[(Z)-heptadec-9-enoyl]oxy-3-octanoyloxypropyl] (13Z,16Z)-tetracosa-13,16-dienoate
[2-[(11Z,14Z)-icosa-11,14-dienoyl]oxy-3-octanoyloxypropyl] (Z)-henicos-11-enoate
(2-heptadecanoyloxy-3-octanoyloxypropyl) (10Z,13Z,16Z)-tetracosa-10,13,16-trienoate
[2-[(Z)-hexadec-9-enoyl]oxy-3-nonanoyloxypropyl] (13Z,16Z)-tetracosa-13,16-dienoate
[3-nonanoyloxy-2-[(Z)-octadec-9-enoyl]oxypropyl] (13Z,16Z)-docosa-13,16-dienoate
[2-[(9Z,12Z)-nonadeca-9,12-dienoyl]oxy-3-nonanoyloxypropyl] (Z)-henicos-11-enoate
[2-[(Z)-nonadec-9-enoyl]oxy-3-nonanoyloxypropyl] (11Z,14Z)-henicosa-11,14-dienoate
[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-octanoyloxypropyl] (Z)-tetracos-13-enoate
[3-octanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] (15Z,18Z)-hexacosa-15,18-dienoate
[2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxy-3-nonanoyloxypropyl] tetracosanoate
[1-[(11Z,14Z,17Z)-icosa-11,14,17-trienoyl]oxy-3-nonanoyloxypropan-2-yl] icosanoate
[3-nonanoyloxy-2-[(9Z,12Z,15Z)-octadeca-9,12,15-trienoyl]oxypropyl] docosanoate
(2-nonadecanoyloxy-3-octanoyloxypropyl) (10Z,13Z,16Z)-docosa-10,13,16-trienoate
(3-octanoyloxy-2-undecanoyloxypropyl) (16Z,19Z,22Z)-triaconta-16,19,22-trienoate
(3-octanoyloxy-2-tridecanoyloxypropyl) (14Z,17Z,20Z)-octacosa-14,17,20-trienoate
[2-[(Z)-nonadec-9-enoyl]oxy-3-octanoyloxypropyl] (13Z,16Z)-docosa-13,16-dienoate
[2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxy-3-octanoyloxypropyl] pentacosanoate
(3-nonanoyloxy-2-tetradecanoyloxypropyl) (12Z,15Z,18Z)-hexacosa-12,15,18-trienoate
[2-[(Z)-icos-11-enoyl]oxy-3-octanoyloxypropyl] (11Z,14Z)-henicosa-11,14-dienoate
[2-[(9Z,12Z,15Z)-octadeca-9,12,15-trienoyl]oxy-3-octanoyloxypropyl] tricosanoate
[3-nonanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (15Z,18Z)-hexacosa-15,18-dienoate
[2-[(9Z,12Z)-nonadeca-9,12-dienoyl]oxy-3-octanoyloxypropyl] (Z)-docos-13-enoate
[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-nonanoyloxypropyl] (Z)-tetracos-13-enoate
(3-octanoyloxy-2-pentadecanoyloxypropyl) (12Z,15Z,18Z)-hexacosa-12,15,18-trienoate
[3-octanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] (17Z,20Z)-octacosa-17,20-dienoate
[2-[(11Z,14Z,17Z)-icosa-11,14,17-trienoyl]oxy-3-octanoyloxypropyl] henicosanoate
(3-nonanoyloxy-2-octadecanoyloxypropyl) (10Z,13Z,16Z)-docosa-10,13,16-trienoate
[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] icosanoate
[3-dodecanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] (13Z,16Z)-docosa-13,16-dienoate
[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] nonadecanoate
[3-decanoyloxy-2-[(9Z,12Z,15Z)-octadeca-9,12,15-trienoyl]oxypropyl] henicosanoate
[1-[(9Z,12Z)-octadeca-9,12-dienoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropan-2-yl] octadecanoate
(2-heptadecanoyloxy-3-tetradecanoyloxypropyl) (9Z,12Z,15Z)-octadeca-9,12,15-trienoate
[2-heptadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (9Z,12Z)-nonadeca-9,12-dienoate
(3-dodecanoyloxy-2-heptadecanoyloxypropyl) (11Z,14Z,17Z)-icosa-11,14,17-trienoate
[3-dodecanoyloxy-2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxypropyl] (Z)-nonadec-9-enoate
[3-decanoyloxy-2-[(Z)-heptadec-9-enoyl]oxypropyl] (13Z,16Z)-docosa-13,16-dienoate
[2-[(9Z,12Z,15Z)-octadeca-9,12,15-trienoyl]oxy-3-undecanoyloxypropyl] icosanoate
[2-[(Z)-heptadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] (9Z,12Z)-octadeca-9,12-dienoate
[3-decanoyloxy-2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxypropyl] (Z)-docos-13-enoate
(2-hexadecanoyloxy-3-tridecanoyloxypropyl) (11Z,14Z,17Z)-icosa-11,14,17-trienoate
(3-dodecanoyloxy-2-pentadecanoyloxypropyl) (10Z,13Z,16Z)-docosa-10,13,16-trienoate
(2-octadecanoyloxy-3-undecanoyloxypropyl) (11Z,14Z,17Z)-icosa-11,14,17-trienoate
[3-dodecanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] (13Z,16Z)-tetracosa-13,16-dienoate
[3-dodecanoyloxy-2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxypropyl] henicosanoate
[1-[(9Z,12Z)-nonadeca-9,12-dienoyl]oxy-3-undecanoyloxypropan-2-yl] (Z)-nonadec-9-enoate
[2-[(Z)-tetradec-9-enoyl]oxy-3-undecanoyloxypropyl] (13Z,16Z)-tetracosa-13,16-dienoate
[2-[(Z)-heptadec-9-enoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-octadec-9-enoate
[2-[(Z)-heptadec-9-enoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (Z)-nonadec-9-enoate
(2-tetradecanoyloxy-3-tridecanoyloxypropyl) (10Z,13Z,16Z)-docosa-10,13,16-trienoate
[3-dodecanoyloxy-2-[(9Z,12Z,15Z)-octadeca-9,12,15-trienoyl]oxypropyl] nonadecanoate
[3-decanoyloxy-2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxypropyl] (Z)-henicos-11-enoate
[2-hexadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (11Z,14Z)-icosa-11,14-dienoate
[2-[(Z)-heptadec-9-enoyl]oxy-3-undecanoyloxypropyl] (11Z,14Z)-henicosa-11,14-dienoate
[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-tridecanoyloxypropyl] (Z)-nonadec-9-enoate
[3-dodecanoyloxy-2-[(Z)-heptadec-9-enoyl]oxypropyl] (11Z,14Z)-icosa-11,14-dienoate
[1-[(9Z,12Z)-octadeca-9,12-dienoyl]oxy-3-tridecanoyloxypropan-2-yl] (Z)-octadec-9-enoate
(3-decanoyloxy-2-nonadecanoyloxypropyl) (11Z,14Z,17Z)-icosa-11,14,17-trienoate
[2-[(Z)-pentadec-9-enoyl]oxy-3-tridecanoyloxypropyl] (11Z,14Z)-henicosa-11,14-dienoate
[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-tridecanoyloxypropyl] (Z)-icos-11-enoate
[3-decanoyloxy-2-[(9Z,12Z)-nonadeca-9,12-dienoyl]oxypropyl] (Z)-icos-11-enoate
[2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxy-3-tetradecanoyloxypropyl] nonadecanoate
[2-[(Z)-pentadec-9-enoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (Z)-henicos-11-enoate
[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-undecanoyloxypropyl] (Z)-henicos-11-enoate
(3-decanoyloxy-2-pentadecanoyloxypropyl) (10Z,13Z,16Z)-tetracosa-10,13,16-trienoate
(3-decanoyloxy-2-heptadecanoyloxypropyl) (10Z,13Z,16Z)-docosa-10,13,16-trienoate
[2-[(Z)-hexadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] (9Z,12Z)-nonadeca-9,12-dienoate
[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-tetradecanoyloxypropyl] (Z)-nonadec-9-enoate
[3-tetradecanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (11Z,14Z)-henicosa-11,14-dienoate
[2-[(Z)-hexadec-9-enoyl]oxy-3-tridecanoyloxypropyl] (11Z,14Z)-icosa-11,14-dienoate
(2-dodecanoyloxy-3-undecanoyloxypropyl) (12Z,15Z,18Z)-hexacosa-12,15,18-trienoate
[2-[(Z)-octadec-9-enoyl]oxy-3-undecanoyloxypropyl] (11Z,14Z)-icosa-11,14-dienoate
[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-undecanoyloxypropyl] (Z)-docos-13-enoate
[2-heptadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (9Z,12Z)-octadeca-9,12-dienoate
(2-pentadecanoyloxy-3-tetradecanoyloxypropyl) (11Z,14Z,17Z)-icosa-11,14,17-trienoate
[2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxy-3-tridecanoyloxypropyl] icosanoate
[2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxy-3-undecanoyloxypropyl] (Z)-icos-11-enoate
[2-hexadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (9Z,12Z)-nonadeca-9,12-dienoate
[3-decanoyloxy-2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxypropyl] tricosanoate
[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] octadecanoate
[3-decanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] (13Z,16Z)-tetracosa-13,16-dienoate
[2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxy-3-undecanoyloxypropyl] docosanoate
[3-decanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] (15Z,18Z)-hexacosa-15,18-dienoate
[1-[(9Z,12Z,15Z)-octadeca-9,12,15-trienoyl]oxy-3-tridecanoyloxypropan-2-yl] octadecanoate
[2-[(Z)-hexadec-9-enoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (Z)-icos-11-enoate
[2-tetradecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (13Z,16Z)-docosa-13,16-dienoate
[3-dodecanoyloxy-2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxypropyl] (Z)-henicos-11-enoate
(3-decanoyloxy-2-tridecanoyloxypropyl) (12Z,15Z,18Z)-hexacosa-12,15,18-trienoate
[2-[(Z)-hexadec-9-enoyl]oxy-3-undecanoyloxypropyl] (13Z,16Z)-docosa-13,16-dienoate
(2-hexadecanoyloxy-3-undecanoyloxypropyl) (10Z,13Z,16Z)-docosa-10,13,16-trienoate
(3-decanoyloxy-2-undecanoyloxypropyl) (14Z,17Z,20Z)-octacosa-14,17,20-trienoate
[3-dodecanoyloxy-2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxypropyl] (Z)-icos-11-enoate
[3-dodecanoyloxy-2-[(Z)-hexadec-9-enoyl]oxypropyl] (11Z,14Z)-henicosa-11,14-dienoate
[2-[(Z)-octadec-9-enoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (Z)-octadec-9-enoate
[2-[(Z)-tetradec-9-enoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (Z)-docos-13-enoate
[2-[(Z)-tetradec-9-enoyl]oxy-3-tridecanoyloxypropyl] (13Z,16Z)-docosa-13,16-dienoate
[3-decanoyloxy-2-[(Z)-octadec-9-enoyl]oxypropyl] (11Z,14Z)-henicosa-11,14-dienoate
[2-pentadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (11Z,14Z)-henicosa-11,14-dienoate
[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] nonadecanoate
[2-[(Z)-hexadec-9-enoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-nonadec-9-enoate
[2-[(Z)-heptadec-9-enoyl]oxy-3-tridecanoyloxypropyl] (9Z,12Z)-nonadeca-9,12-dienoate
2,3-bis[[(Z)-tetradec-9-enoyl]oxy]propyl (Z)-henicos-11-enoate
(3-dodecanoyloxy-2-tridecanoyloxypropyl) (10Z,13Z,16Z)-tetracosa-10,13,16-trienoate
[3-decanoyloxy-2-[(Z)-nonadec-9-enoyl]oxypropyl] (11Z,14Z)-icosa-11,14-dienoate
[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-tetradecanoyloxypropyl] (Z)-octadec-9-enoate
[3-dodecanoyloxy-2-[(Z)-octadec-9-enoyl]oxypropyl] (9Z,12Z)-nonadeca-9,12-dienoate
(2-tetradecanoyloxy-3-undecanoyloxypropyl) (10Z,13Z,16Z)-tetracosa-10,13,16-trienoate
[3-hexadecanoyloxy-2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxypropyl] heptadecanoate
[2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxy-3-pentadecanoyloxypropyl] octadecanoate
(2-hexadecanoyloxy-3-pentadecanoyloxypropyl) (9Z,12Z,15Z)-octadeca-9,12,15-trienoate
[1-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-pentadecanoyloxypropan-2-yl] (Z)-heptadec-9-enoate
[2-[(Z)-hexadec-9-enoyl]oxy-3-pentadecanoyloxypropyl] (9Z,12Z)-octadeca-9,12-dienoate
[3-pentadecanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] (9Z,12Z)-nonadeca-9,12-dienoate
[1-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropan-2-yl] heptadecanoate
[2-hexadecanoyloxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] (9Z,12Z)-octadeca-9,12-dienoate
[3-hexadecanoyloxy-2-[(Z)-hexadec-9-enoyl]oxypropyl] (9Z,12Z)-heptadeca-9,12-dienoate
[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-[(Z)-hexadec-9-enoyl]oxypropyl] heptadecanoate
[2-[(Z)-heptadec-9-enoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] (Z)-heptadec-9-enoate
2,3-bis[[(Z)-hexadec-9-enoyl]oxy]propyl (Z)-heptadec-9-enoate
[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-pentadecanoyloxypropyl] (Z)-octadec-9-enoate
[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-hexadecanoyloxypropyl] (Z)-heptadec-9-enoate
[2-[(Z)-hexadec-9-enoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] (Z)-octadec-9-enoate
[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] octadecanoate
[3-hydroxy-2-[[(Z)-tetratriacont-23-enoyl]amino]octyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-3-hydroxy-2-(nonadecanoylamino)tricos-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-3-hydroxy-2-(icosanoylamino)docos-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[3-hydroxy-2-[[(Z)-pentadec-9-enoyl]amino]heptacosyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-2-(heptacosanoylamino)-3-hydroxypentadec-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-3-hydroxy-2-(octacosanoylamino)tetradec-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-3-hydroxy-2-(tricosanoylamino)nonadec-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-3-hydroxy-2-(tetradecanoylamino)octacos-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-2-(heptadecanoylamino)-3-hydroxypentacos-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-2-(docosanoylamino)-3-hydroxyicos-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-3-hydroxy-2-(pentacosanoylamino)heptadec-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[3-hydroxy-2-[[(Z)-tetradec-9-enoyl]amino]octacosyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-2-(hexadecanoylamino)-3-hydroxyhexacos-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-2-(henicosanoylamino)-3-hydroxyhenicos-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[3-hydroxy-2-[[(Z)-icos-11-enoyl]amino]docosyl] 2-(trimethylazaniumyl)ethyl phosphate
[3-hydroxy-2-[[(Z)-nonadec-9-enoyl]amino]tricosyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-3-hydroxy-2-(octadecanoylamino)tetracos-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-2-(hexacosanoylamino)-3-hydroxyhexadec-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-2-(dodecanoylamino)-3-hydroxytriacont-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-3-hydroxy-2-(tridecanoylamino)nonacos-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E)-3-hydroxy-2-(pentadecanoylamino)heptacos-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[2-[(Z)-pentadec-9-enoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-icos-11-enoate
[2-[(Z)-pentadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] (11Z,14Z)-icosa-11,14-dienoate
2,3-bis[[(Z)-pentadec-9-enoyl]oxy]propyl (Z)-nonadec-9-enoate
[2-pentadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (11Z,14Z)-icosa-11,14-dienoate
[3-dodecanoyloxy-2-[(9Z,11Z,13Z)-hexadeca-9,11,13-trienoyl]oxypropyl] henicosanoate
(2-pentadecanoyloxy-3-tridecanoyloxypropyl) (9Z,11Z,13Z)-henicosa-9,11,13-trienoate
[2-hexadecanoyloxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] (10Z,12Z)-octadeca-10,12-dienoate
[3-hydroxy-2-[[(Z)-tridec-8-enoyl]amino]nonacosyl] 2-(trimethylazaniumyl)ethyl phosphate
(2-hexadecanoyloxy-3-tridecanoyloxypropyl) (5Z,8Z,11Z)-icosa-5,8,11-trienoate
(3-dodecanoyloxy-2-hexadecanoyloxypropyl) (9Z,11Z,13Z)-henicosa-9,11,13-trienoate
[3-dodecanoyloxy-2-[(8Z,11Z,14Z)-heptadeca-8,11,14-trienoyl]oxypropyl] icosanoate
2,3-bis[[(Z)-tetradec-9-enoyl]oxy]propyl (Z)-henicos-9-enoate
[1-[(10Z,12Z)-octadeca-10,12-dienoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropan-2-yl] octadecanoate
[2-[(9Z,11Z,13Z)-hexadeca-9,11,13-trienoyl]oxy-3-pentadecanoyloxypropyl] octadecanoate
[2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] nonadecanoate
[2-pentadecanoyloxy-3-[(5Z,8Z,11Z)-tetradeca-5,8,11-trienoyl]oxypropyl] icosanoate
[2-[(Z)-hexadec-7-enoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (Z)-icos-11-enoate
[2-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxy-3-tetradecanoyloxypropyl] (Z)-henicos-9-enoate
[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-pentadecanoyloxypropyl] (Z)-docos-11-enoate
[3-dodecanoyloxy-2-[(Z)-octadec-11-enoyl]oxypropyl] (7Z,9Z)-nonadeca-7,9-dienoate
2,3-di(pentadecanoyloxy)propyl (10Z,13Z,16Z)-nonadeca-10,13,16-trienoate
2,3-bis[[(Z)-hexadec-7-enoyl]oxy]propyl (Z)-heptadec-7-enoate
[2-[(8Z,11Z,14Z)-heptadeca-8,11,14-trienoyl]oxy-3-tridecanoyloxypropyl] nonadecanoate
[2-[(Z)-pentadec-9-enoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (Z)-henicos-9-enoate
[2-[(Z)-tetradec-9-enoyl]oxy-3-tridecanoyloxypropyl] (14Z,16Z)-docosa-14,16-dienoate
[2-[[(Z)-dodec-5-enoyl]amino]-3-hydroxytriacontyl] 2-(trimethylazaniumyl)ethyl phosphate
[2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxy-3-[(Z)-hexadec-7-enoyl]oxypropyl] heptadecanoate
[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-heptadecanoyloxypropyl] (Z)-icos-11-enoate
(2-heptadecanoyloxy-3-tetradecanoyloxypropyl) (11Z,13Z,15Z)-octadeca-11,13,15-trienoate
[2-hexadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (7Z,9Z)-nonadeca-7,9-dienoate
[3-dodecanoyloxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] (9Z,11Z)-henicosa-9,11-dienoate
[3-dodecanoyloxy-2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxypropyl] (Z)-docos-11-enoate
[3-[(3Z,6Z,9Z)-dodeca-3,6,9-trienoyl]oxy-2-pentadecanoyloxypropyl] docosanoate
[3-[(Z)-dodec-5-enoyl]oxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] (Z)-henicos-9-enoate
[1-[(Z)-heptadec-7-enoyl]oxy-3-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxypropan-2-yl] heptadecanoate
(3-dodecanoyloxy-2-heptadecanoyloxypropyl) (5Z,8Z,11Z)-icosa-5,8,11-trienoate
[2-[(9Z,11Z,13Z)-hexadeca-9,11,13-trienoyl]oxy-3-tetradecanoyloxypropyl] nonadecanoate
[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-[(Z)-heptadec-7-enoyl]oxypropyl] icosanoate
[3-[(Z)-dodec-5-enoyl]oxy-2-octadecanoyloxypropyl] (7Z,9Z)-nonadeca-7,9-dienoate
[3-dodecanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] (14Z,16Z)-docosa-14,16-dienoate
[2-tetradecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (14Z,16Z)-docosa-14,16-dienoate
[2-[[(Z)-hexacos-11-enoyl]amino]-3-hydroxyhexadecyl] 2-(trimethylazaniumyl)ethyl phosphate
[2-hexadecanoyloxy-3-[(5Z,8Z,11Z)-tetradeca-5,8,11-trienoyl]oxypropyl] nonadecanoate
[2-[(Z)-heptadec-7-enoyl]oxy-3-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] octadecanoate
2,3-di(tetradecanoyloxy)propyl (9Z,11Z,13Z)-henicosa-9,11,13-trienoate
[3-[(3Z,6Z,9Z)-dodeca-3,6,9-trienoyl]oxy-2-heptadecanoyloxypropyl] icosanoate
(2-pentadecanoyloxy-3-tetradecanoyloxypropyl) (5Z,8Z,11Z)-icosa-5,8,11-trienoate
[2-[[(Z)-heptacos-12-enoyl]amino]-3-hydroxypentadecyl] 2-(trimethylazaniumyl)ethyl phosphate
[2-[(Z)-hexadec-7-enoyl]oxy-3-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] nonadecanoate
[2-[(Z)-heptadec-7-enoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] (Z)-heptadec-7-enoate
[2-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] octadecanoate
[2-[(Z)-hexadec-7-enoyl]oxy-3-pentadecanoyloxypropyl] (10Z,12Z)-octadeca-10,12-dienoate
[2-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] nonadecanoate
[2-[[(Z)-henicos-9-enoyl]amino]-3-hydroxyhenicosyl] 2-(trimethylazaniumyl)ethyl phosphate
(3-dodecanoyloxy-2-octadecanoyloxypropyl) (10Z,13Z,16Z)-nonadeca-10,13,16-trienoate
[3-dodecanoyloxy-2-[(10Z,12Z)-octadeca-10,12-dienoyl]oxypropyl] (Z)-nonadec-9-enoate
[2-[(Z)-hexadec-7-enoyl]oxy-3-tridecanoyloxypropyl] (11Z,14Z)-icosa-11,14-dienoate
[3-[(3Z,6Z,9Z)-dodeca-3,6,9-trienoyl]oxy-2-octadecanoyloxypropyl] nonadecanoate
[3-dodecanoyloxy-2-[(11Z,13Z,15Z)-octadeca-11,13,15-trienoyl]oxypropyl] nonadecanoate
[2-heptadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (10Z,12Z)-octadeca-10,12-dienoate
[2-[(Z)-pentadec-9-enoyl]oxy-3-tridecanoyloxypropyl] (9Z,11Z)-henicosa-9,11-dienoate
[2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxy-3-pentadecanoyloxypropyl] (Z)-nonadec-9-enoate
[3-hydroxy-2-[[(Z)-octadec-11-enoyl]amino]tetracosyl] 2-(trimethylazaniumyl)ethyl phosphate
[1-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxy-3-pentadecanoyloxypropan-2-yl] (Z)-heptadec-7-enoate
[1-[(11Z,13Z,15Z)-octadeca-11,13,15-trienoyl]oxy-3-tridecanoyloxypropan-2-yl] octadecanoate
[3-hexadecanoyloxy-2-[(9Z,11Z,13Z)-hexadeca-9,11,13-trienoyl]oxypropyl] heptadecanoate
[2-heptadecanoyloxy-3-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] (Z)-octadec-11-enoate
[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] docosanoate
[2-[(Z)-heptadec-7-enoyl]oxy-3-tetradecanoyloxypropyl] (10Z,12Z)-octadeca-10,12-dienoate
(2-tetradecanoyloxy-3-tridecanoyloxypropyl) (13Z,16Z,19Z)-docosa-13,16,19-trienoate
[3-tetradecanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (9Z,11Z)-henicosa-9,11-dienoate
[3-pentadecanoyloxy-2-[(6Z,9Z,12Z)-pentadeca-6,9,12-trienoyl]oxypropyl] nonadecanoate
(2-hexadecanoyloxy-3-pentadecanoyloxypropyl) (11Z,13Z,15Z)-octadeca-11,13,15-trienoate
[1-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropan-2-yl] heptadecanoate
[3-[(Z)-dodec-5-enoyl]oxy-2-hexadecanoyloxypropyl] (9Z,11Z)-henicosa-9,11-dienoate
[1-[(10Z,12Z)-octadeca-10,12-dienoyl]oxy-3-tridecanoyloxypropan-2-yl] (Z)-octadec-11-enoate
[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] henicosanoate
[1-[(8Z,11Z,14Z)-heptadeca-8,11,14-trienoyl]oxy-3-pentadecanoyloxypropan-2-yl] heptadecanoate
[2-pentadecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (9Z,11Z)-henicosa-9,11-dienoate
[2-[(Z)-hexadec-7-enoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-nonadec-9-enoate
[3-dodecanoyloxy-2-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxypropyl] (Z)-icos-11-enoate
[3-hexadecanoyloxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] (11Z,14Z)-heptadeca-11,14-dienoate
[2-[(Z)-heptadec-7-enoyl]oxy-3-tridecanoyloxypropyl] (7Z,9Z)-nonadeca-7,9-dienoate
[2-[(Z)-hexadec-7-enoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] (Z)-octadec-11-enoate
[2-[(Z)-hexadec-7-enoyl]oxy-3-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxypropyl] octadecanoate
[3-hydroxy-2-[[(Z)-pentacos-11-enoyl]amino]heptadecyl] 2-(trimethylazaniumyl)ethyl phosphate
[3-[(Z)-dodec-5-enoyl]oxy-2-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxypropyl] icosanoate
[2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] henicosanoate
[3-[(Z)-dodec-5-enoyl]oxy-2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxypropyl] docosanoate
[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-[(Z)-octadec-11-enoyl]oxypropyl] nonadecanoate
[3-hydroxy-2-[[(Z)-tricos-11-enoyl]amino]nonadecyl] 2-(trimethylazaniumyl)ethyl phosphate
[3-hydroxy-2-[[(Z)-octacos-13-enoyl]amino]tetradecyl] 2-(trimethylazaniumyl)ethyl phosphate
[2-[[(Z)-hexadec-7-enoyl]amino]-3-hydroxyhexacosyl] 2-(trimethylazaniumyl)ethyl phosphate
[2-[[(Z)-docos-11-enoyl]amino]-3-hydroxyicosyl] 2-(trimethylazaniumyl)ethyl phosphate
N-(eicosanoyl)-4E-docosasphingenine-1-phosphocholine
N-(tricosanoyl)-4E-nonadecasphingenine-1-phosphocholine
N-(docosanoyl)-4E-eicosasphingenine-1-phosphocholine
N-(pentacosanoyl)-4E-heptadecasphingenine-1-phosphocholine
N-(heneicosanoyl)-4E-heneicosasphingenine-1-phosphocholine
N-(hexacosanoyl)-4E-hexadecasphingenine-1-phosphocholine
[(E,2S,3R)-3-hydroxy-2-(icosanoylamino)docos-8-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E,2S,3R)-3-hydroxy-2-(pentacosanoylamino)heptadec-8-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(2S,3R)-2-[[(E)-docos-13-enoyl]amino]-3-hydroxyicosyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E,2S,3R)-2-(docosanoylamino)-3-hydroxyicos-8-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E,2S,3R)-2-(hexacosanoylamino)-3-hydroxyhexadec-8-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E,2S,3R)-2-(henicosanoylamino)-3-hydroxyhenicos-8-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(2S,3R)-2-[[(E)-hexacos-17-enoyl]amino]-3-hydroxyhexadecyl] 2-(trimethylazaniumyl)ethyl phosphate
[(2S,3R)-3-hydroxy-2-[[(E)-icos-11-enoyl]amino]docosyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E,2S,3R)-3-hydroxy-2-(tricosanoylamino)nonadec-8-enyl] 2-(trimethylazaniumyl)ethyl phosphate
[(2S,3R)-3-hydroxy-2-[[(E)-tetracos-15-enoyl]amino]octadecyl] 2-(trimethylazaniumyl)ethyl phosphate
[(E,2S,3R)-3-hydroxy-2-(tetracosanoylamino)octadec-8-enyl] 2-(trimethylazaniumyl)ethyl phosphate
2-[[2-[(11Z,14Z)-henicosa-11,14-dienoyl]oxy-3-octadecoxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[2-henicosanoyloxy-3-[(9Z,12Z)-octadeca-9,12-dienoxy]propoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[2-[(Z)-henicos-11-enoyl]oxy-3-[(Z)-octadec-9-enoxy]propoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[3-[(9Z,12Z)-hexadeca-9,12-dienoxy]-2-tricosanoyloxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[hydroxy-[2-[(17Z,20Z)-octacosa-17,20-dienoyl]oxy-3-undecoxypropoxy]phosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[3-henicosoxy-2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[hydroxy-[2-[(Z)-pentadec-9-enoyl]oxy-3-[(Z)-tetracos-13-enoxy]propoxy]phosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[3-[(13Z,16Z)-docosa-13,16-dienoxy]-2-heptadecanoyloxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[hydroxy-[2-[(Z)-icos-11-enoyl]oxy-3-[(Z)-nonadec-9-enoxy]propoxy]phosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[3-[(Z)-henicos-11-enoxy]-2-[(Z)-octadec-9-enoyl]oxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[3-[(Z)-hexacos-15-enoxy]-2-[(Z)-tridec-9-enoyl]oxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-tricosoxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[hydroxy-[2-icosanoyloxy-3-[(9Z,12Z)-nonadeca-9,12-dienoxy]propoxy]phosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[2-[(15Z,18Z)-hexacosa-15,18-dienoyl]oxy-3-tridecoxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[hydroxy-[3-[(17Z,20Z)-octacosa-17,20-dienoxy]-2-undecanoyloxypropoxy]phosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[hydroxy-[3-[(11Z,14Z)-icosa-11,14-dienoxy]-2-nonadecanoyloxypropoxy]phosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[2-[(13Z,16Z)-docosa-13,16-dienoyl]oxy-3-heptadecoxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[3-[(11Z,14Z)-henicosa-11,14-dienoxy]-2-octadecanoyloxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[2-[(Z)-hexacos-15-enoyl]oxy-3-[(Z)-tridec-9-enoxy]propoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[2-[(Z)-docos-13-enoyl]oxy-3-[(Z)-heptadec-9-enoxy]propoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[hydroxy-[3-[(Z)-pentadec-9-enoxy]-2-[(Z)-tetracos-13-enoyl]oxypropoxy]phosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[hydroxy-[3-[(Z)-icos-11-enoxy]-2-[(Z)-nonadec-9-enoyl]oxypropoxy]phosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[2-docosanoyloxy-3-[(9Z,12Z)-heptadeca-9,12-dienoxy]propoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[3-[(15Z,18Z)-hexacosa-15,18-dienoxy]-2-tridecanoyloxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[hydroxy-[2-[(11Z,14Z)-icosa-11,14-dienoyl]oxy-3-nonadecoxypropoxy]phosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[hydroxy-[3-pentadecoxy-2-[(13Z,16Z)-tetracosa-13,16-dienoyl]oxypropoxy]phosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[3-docosoxy-2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[hydroxy-[2-pentadecanoyloxy-3-[(13Z,16Z)-tetracosa-13,16-dienoxy]propoxy]phosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[[3-[(Z)-docos-13-enoxy]-2-[(Z)-heptadec-9-enoyl]oxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
2-[hydroxy-[3-icosoxy-2-[(9Z,12Z)-nonadeca-9,12-dienoyl]oxypropoxy]phosphoryl]oxyethyl-trimethylazanium
C47H93NO7P+ (814.6689297999999)
N-lignoceroylsphingosine-1-phosphocholine
A sphingomyelin d18:1 in which the ceramide N-acyl group is specified as lignoceroyl (tetracosanoyl).
N-(15Z-tetracosenoyl)-sphinganine-1-phosphocholine
sphingomyelin d18:1/24:0
A sphingomyelin d18:1 in which the fatty acyl group contains 24 carbons and is fully saturated.
AcHexSiE(16:0)
Provides by LipidSearch Vendor. © Copyright 2006-2024 Thermo Fisher Scientific Inc. All rights reserved
MGMG(39:0)
Provides by LipidSearch Vendor. © Copyright 2006-2024 Thermo Fisher Scientific Inc. All rights reserved
MGDG(39:0)
Provides by LipidSearch Vendor. © Copyright 2006-2024 Thermo Fisher Scientific Inc. All rights reserved
AcHexChE(18:0)
Provides by LipidSearch Vendor. © Copyright 2006-2024 Thermo Fisher Scientific Inc. All rights reserved