Exact Mass: 804.7318846

TG(16:0/16:0/16:1(9Z))

C51H96O6 (804.7206515999999)

TG(16:0/16:0/16:1(9Z))[iso3] is a dipalmitic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/16:0/16:1(9Z))[iso3], in particular, consists of one chain of palmitic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols. TG(16:0/16:0/16:1(9Z))[iso3] is a dipalmitic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/16:0/16:1(9Z))[iso3], in particular, consists of one chain of palmitic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)

TG(16:0/14:0/18:1(9Z))

C51H96O6 (804.7206515999999)

TG(16:0/14:0/18:1(9Z))[iso6] is a monooleic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/14:0/18:1(9Z))[iso6], in particular, consists of one chain of palmitic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols. TG(16:0/14:0/18:1(9Z))[iso6] is a monooleic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/14:0/18:1(9Z))[iso6], in particular, consists of one chain of palmitic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)

TG(18:0/14:0/16:1(9Z))

C51H96O6 (804.7206515999999)

TG(18:0/14:0/16:1(9Z))[iso6] is a monostearic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:0/14:0/16:1(9Z))[iso6], in particular, consists of one chain of stearic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols. TG(18:0/14:0/16:1(9Z))[iso6] is a monostearic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid tri-esters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:0/14:0/16:1(9Z))[iso6], in particular, consists of one chain of stearic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)

alpha-Amyrin cerotate

C56H100O2 (804.77229)

alpha-Amyrin cerotate is found in herbs and spices. alpha-Amyrin cerotate is a constituent of Rosmarinus officinalis (rosemary). Constituent of Rosmarinus officinalis (rosemary). alpha-Amyrin cerotate is found in herbs and spices.

TG(14:0/14:0/20:1(11Z))

C51H96O6 (804.7206515999999)

TG(14:0/14:0/20:1(11Z)) is a dimyristic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/14:0/20:1(11Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of eicosenoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/16:0/18:1(11Z))

C51H96O6 (804.7206515999999)

TG(14:0/16:0/18:1(11Z)) is a monovaccenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/16:0/18:1(11Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of vaccenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/16:0/18:1(9Z))

C51H96O6 (804.7206515999999)

TG(14:0/16:0/18:1(9Z)) is a monooleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/16:0/18:1(9Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/18:0/16:1(9Z))

C51H96O6 (804.7206515999999)

TG(14:0/18:0/16:1(9Z)) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/18:0/16:1(9Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of stearic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/20:0/14:1(9Z))

C51H96O6 (804.7206515999999)

TG(14:0/20:0/14:1(9Z)) is a monoarachidic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/20:0/14:1(9Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of arachidic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/14:1(9Z)/20:0)

C51H96O6 (804.7206515999999)

TG(14:0/14:1(9Z)/20:0) is a monoarachidic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/14:1(9Z)/20:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of arachidic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/16:1(9Z)/18:0)

C51H96O6 (804.7206515999999)

TG(14:0/16:1(9Z)/18:0) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/16:1(9Z)/18:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of stearic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/18:1(11Z)/16:0)

C51H96O6 (804.7206515999999)

TG(14:0/18:1(11Z)/16:0) is a monovaccenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/18:1(11Z)/16:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of vaccenic acid at the C-2 position and one chain of palmitic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/18:1(9Z)/16:0)

C51H96O6 (804.7206515999999)

TG(14:0/18:1(9Z)/16:0) is a monooleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/18:1(9Z)/16:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of oleic acid at the C-2 position and one chain of palmitic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/20:1(11Z)/14:0)

C51H96O6 (804.7206515999999)

TG(14:0/20:1(11Z)/14:0) is a dimyristic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/20:1(11Z)/14:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of eicosenoic acid at the C-2 position and one chain of myristic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/15:0/18:1(11Z))

C51H96O6 (804.7206515999999)

TG(15:0/15:0/18:1(11Z)) is a dipentadecanoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/15:0/18:1(11Z)), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of vaccenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/15:0/18:1(9Z))

C51H96O6 (804.7206515999999)

TG(15:0/15:0/18:1(9Z)) is a dipentadecanoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/15:0/18:1(9Z)), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/16:1(9Z)/O-18:0)

C52H100O5 (804.757035)

TG(15:0/16:1(9Z)/O-18:0) is a monoStearyl alcohol triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/16:1(9Z)/O-18:0), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of Stearyl alcohol at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/18:1(11Z)/15:0)

C51H96O6 (804.7206515999999)

TG(15:0/18:1(11Z)/15:0) is a dipentadecanoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/18:1(11Z)/15:0), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of vaccenic acid at the C-2 position and one chain of pentadecanoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/18:1(9Z)/15:0)

C51H96O6 (804.7206515999999)

TG(15:0/18:1(9Z)/15:0) is a dipentadecanoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/18:1(9Z)/15:0), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of oleic acid at the C-2 position and one chain of pentadecanoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/O-18:0/16:1(9Z))

C52H100O5 (804.757035)

TG(15:0/O-18:0/16:1(9Z)) is a monoStearyl alcohol triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/O-18:0/16:1(9Z)), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of Stearyl alcohol at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/14:0/18:1(11Z))

C51H96O6 (804.7206515999999)

TG(16:0/14:0/18:1(11Z)) is a monovaccenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/14:0/18:1(11Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of vaccenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/18:0/14:1(9Z))

C51H96O6 (804.7206515999999)

TG(16:0/18:0/14:1(9Z)) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/18:0/14:1(9Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of stearic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/14:1(9Z)/18:0)

C51H96O6 (804.7206515999999)

TG(16:0/14:1(9Z)/18:0) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/14:1(9Z)/18:0), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of stearic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/16:1(9Z)/16:0)

C51H96O6 (804.7206515999999)

TG(16:0/16:1(9Z)/16:0) is a dipalmitic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/16:1(9Z)/16:0), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of palmitic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(18:0/16:0/14:1(9Z))

C51H96O6 (804.7206515999999)

TG(18:0/16:0/14:1(9Z)) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:0/16:0/14:1(9Z)), in particular, consists of one chain of stearic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(20:0/14:0/14:1(9Z))

C51H96O6 (804.7206515999999)

TG(20:0/14:0/14:1(9Z)) is a monoarachidic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(20:0/14:0/14:1(9Z)), in particular, consists of one chain of arachidic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:1(9Z)/15:0/O-18:0)

C52H100O5 (804.757035)

TG(16:1(9Z)/15:0/O-18:0) is a monoStearyl alcohol triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:1(9Z)/15:0/O-18:0), in particular, consists of one chain of palmitoleic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of Stearyl alcohol at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

hancolupenol hexacosanoate|O-Hexacosanoyl-Hancolupenol

C56H100O2 (804.77229)

lup-20(29)-ene-3beta-yl hexacosanoate

C56H100O2 (804.77229)

TG(14:0/14:1(9Z)/20:0)[iso6]

C51H96O6 (804.7206515999999)

TG(14:0/15:0/19:1(9Z))[iso6]

C51H96O6 (804.7206515999999)

TG(14:0/15:1(9Z)/19:0)[iso6]

C51H96O6 (804.7206515999999)

TG(14:0/16:0/18:1(9Z))[iso6]

C51H96O6 (804.7206515999999)

TG(14:0/16:1(9Z)/18:0)[iso6]

C51H96O6 (804.7206515999999)

TG(14:0/17:0/17:1(9Z))[iso6]

C51H96O6 (804.7206515999999)

TG(14:1(9Z)/15:0/19:0)[iso6]

C51H96O6 (804.7206515999999)

TG(14:1(9Z)/16:0/18:0)[iso6]

C51H96O6 (804.7206515999999)

TG(15:0/15:1(9Z)/18:0)[iso6]

C51H96O6 (804.7206515999999)

TG(15:0/16:0/17:1(9Z))[iso6]

C51H96O6 (804.7206515999999)

TG(15:0/16:1(9Z)/17:0)[iso6]

C51H96O6 (804.7206515999999)

TG(15:1(9Z)/16:0/17:0)[iso6]

C51H96O6 (804.7206515999999)

TG(16:0/16:0/16:1)[iso3]

C51H96O6 (804.7206515999999)

Triglyceride

C51H96O6 (804.7206515999999)

alpha-Amyrin cerotate

C56H100O2 (804.77229)

TG(13:0/13:0/22:1(11Z))[iso3]

C51H96O6 (804.7206515999999)

TG(14:0/14:0/20:1(11Z))[iso3]

C51H96O6 (804.7206515999999)

TG(14:1(9Z)/17:0/17:0)[iso3]

C51H96O6 (804.7206515999999)

TG(15:0/15:0/18:1(9Z))[iso3]

C51H96O6 (804.7206515999999)

TG(12:0/14:0/22:1(11Z))[iso6]

C51H96O6 (804.7206515999999)

TG(12:0/14:1(9Z)/22:0)[iso6]

C51H96O6 (804.7206515999999)

TG(12:0/15:1(9Z)/21:0)[iso6]

C51H96O6 (804.7206515999999)

TG(12:0/16:0/20:1(11Z))[iso6]

C51H96O6 (804.7206515999999)

TG(12:0/16:1(9Z)/20:0)[iso6]

C51H96O6 (804.7206515999999)

TG(12:0/17:0/19:1(9Z))[iso6]

C51H96O6 (804.7206515999999)

TG(12:0/17:1(9Z)/19:0)[iso6]

C51H96O6 (804.7206515999999)

TG(12:0/18:0/18:1(9Z))[iso6]

C51H96O6 (804.7206515999999)

TG(13:0/14:1(9Z)/21:0)[iso6]

C51H96O6 (804.7206515999999)

TG(13:0/15:0/20:1(11Z))[iso6]

C51H96O6 (804.7206515999999)

TG(13:0/15:1(9Z)/20:0)[iso6]

C51H96O6 (804.7206515999999)

TG(13:0/16:0/19:1(9Z))[iso6]

C51H96O6 (804.7206515999999)

TG(13:0/16:1(9Z)/19:0)[iso6]

C51H96O6 (804.7206515999999)

TG(13:0/17:0/18:1(9Z))[iso6]

C51H96O6 (804.7206515999999)

TG(13:0/17:1(9Z)/18:0)[iso6]

C51H96O6 (804.7206515999999)

TG 48:1

C51H96O6 (804.7206515999999)

dioctadecyl [[3,5-bis(1,1-dimethylethyl)-4-hydroxyphenyl]methyl]phosphonate

C51H97O4P (804.7124091999999)

9-Hexadecenoic acid, 2-[(1-oxohexadecyl)oxy]-1-[[(1-oxohexadecyl)oxy]methyl]ethyl ester, (9Z)-

C51H96O6 (804.7206515999999)

1,3-dipentadecanoyl-2-(9Z-octadecenoyl)-glycerol (d5)

C51H96O6 (804.7206515999999)

1-Myristoyl-2-myristoleoyl-3-arachidonyl-glycerol

C51H96O6 (804.7206515999999)

1-Myristoyl-2-palmitoyl-3-oleoyl-glycerol

C51H96O6 (804.7206515999999)

1-Palmitoyl-2-myristoyl-3-oleoyl-glycerol

C51H96O6 (804.7206515999999)

1-Stearoyl-2-myristoyl-3-palmitoleoyl-glycerol

C51H96O6 (804.7206515999999)

1-Palmitoyl-2-palmitoyl-3-palmitoleoyl-glycerol

C51H96O6 (804.7206515999999)

2-[[(2R)-2-docosanoyloxy-3-hexadecoxypropoxy]-hydroxyphosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[hydroxy-[(2R)-2-icosanoyloxy-3-octadecoxypropoxy]phosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

1-Palmitoleoyl-2,3-dipalmitoylglycerol

C51H96O6 (804.7206515999999)

NAOrn 23:0/22:1

C50H96N2O5 (804.7318846)

NAOrn 24:0/21:1

C50H96N2O5 (804.7318846)

NAOrn 22:1/23:0

C50H96N2O5 (804.7318846)

NAOrn 21:0/24:1

C50H96N2O5 (804.7318846)

NAOrn 21:1/24:0

C50H96N2O5 (804.7318846)

NAOrn 24:1/21:0

C50H96N2O5 (804.7318846)

NAOrn 25:0/20:1

C50H96N2O5 (804.7318846)

NAOrn 20:1/25:0

C50H96N2O5 (804.7318846)

NAOrn 19:0/26:1

C50H96N2O5 (804.7318846)

NAOrn 26:1/19:0

C50H96N2O5 (804.7318846)

NAOrn 19:1/26:0

C50H96N2O5 (804.7318846)

NAOrn 26:0/19:1

C50H96N2O5 (804.7318846)

(1-hydroxy-3-nonanoyloxypropan-2-yl) (Z)-tetracont-29-enoate

C52H100O5 (804.757035)

[2-[(Z)-henicos-11-enoyl]oxy-3-hydroxypropyl] octacosanoate

C52H100O5 (804.757035)

[3-hydroxy-2-[(Z)-tridec-9-enoyl]oxypropyl] hexatriacontanoate

C52H100O5 (804.757035)

[3-hydroxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] pentatriacontanoate

C52H100O5 (804.757035)

(1-heptadecanoyloxy-3-hydroxypropan-2-yl) (Z)-dotriacont-21-enoate

C52H100O5 (804.757035)

(1-hydroxy-3-tridecanoyloxypropan-2-yl) (Z)-hexatriacont-25-enoate

C52H100O5 (804.757035)

[3-hydroxy-2-[(Z)-nonadec-9-enoyl]oxypropyl] triacontanoate

C52H100O5 (804.757035)

(1-hydroxy-3-nonadecanoyloxypropan-2-yl) (Z)-triacont-19-enoate

C52H100O5 (804.757035)

(1-hydroxy-3-undecanoyloxypropan-2-yl) (Z)-octatriacont-27-enoate

C52H100O5 (804.757035)

[3-hydroxy-2-[(Z)-octadec-9-enoyl]oxypropyl] hentriacontanoate

C52H100O5 (804.757035)

[3-hydroxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] tetratriacontanoate

C52H100O5 (804.757035)

[2-[(Z)-hexadec-9-enoyl]oxy-3-hydroxypropyl] tritriacontanoate

C52H100O5 (804.757035)

(1-henicosanoyloxy-3-hydroxypropan-2-yl) (Z)-octacos-17-enoate

C52H100O5 (804.757035)

[3-hydroxy-2-[(Z)-icos-11-enoyl]oxypropyl] nonacosanoate

C52H100O5 (804.757035)

(1-hydroxy-3-pentadecanoyloxypropan-2-yl) (Z)-tetratriacont-23-enoate

C52H100O5 (804.757035)

[2-[(Z)-heptadec-9-enoyl]oxy-3-hydroxypropyl] dotriacontanoate

C52H100O5 (804.757035)

2,3-di(octanoyloxy)propyl (Z)-dotriacont-21-enoate

C51H96O6 (804.7206515999999)

2,3-di(nonanoyloxy)propyl (Z)-triacont-19-enoate

C51H96O6 (804.7206515999999)

(2-heptadecanoyloxy-3-nonanoyloxypropyl) (Z)-docos-13-enoate

C51H96O6 (804.7206515999999)

(2-hexadecanoyloxy-3-octanoyloxypropyl) (Z)-tetracos-13-enoate

C51H96O6 (804.7206515999999)

(2-nonadecanoyloxy-3-octanoyloxypropyl) (Z)-henicos-11-enoate

C51H96O6 (804.7206515999999)

[2-[(Z)-hexadec-9-enoyl]oxy-3-nonanoyloxypropyl] tricosanoate

C51H96O6 (804.7206515999999)

[2-[(Z)-heptadec-9-enoyl]oxy-3-octanoyloxypropyl] tricosanoate

C51H96O6 (804.7206515999999)

(3-nonanoyloxy-2-undecanoyloxypropyl) (Z)-octacos-17-enoate

C51H96O6 (804.7206515999999)

(2-decanoyloxy-3-octanoyloxypropyl) (Z)-triacont-19-enoate

C51H96O6 (804.7206515999999)

[2-[(Z)-hexadec-9-enoyl]oxy-3-octanoyloxypropyl] tetracosanoate

C51H96O6 (804.7206515999999)

[1-[(Z)-icos-11-enoyl]oxy-3-octanoyloxypropan-2-yl] icosanoate

C51H96O6 (804.7206515999999)

(3-nonanoyloxy-2-pentadecanoyloxypropyl) (Z)-tetracos-13-enoate

C51H96O6 (804.7206515999999)

[2-[(Z)-octadec-9-enoyl]oxy-3-octanoyloxypropyl] docosanoate

C51H96O6 (804.7206515999999)

(2-dodecanoyloxy-3-octanoyloxypropyl) (Z)-octacos-17-enoate

C51H96O6 (804.7206515999999)

[3-nonanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] tetracosanoate

C51H96O6 (804.7206515999999)

[3-octanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] hexacosanoate

C51H96O6 (804.7206515999999)

[2-[(Z)-heptadec-9-enoyl]oxy-3-nonanoyloxypropyl] docosanoate

C51H96O6 (804.7206515999999)

(3-nonanoyloxy-2-octadecanoyloxypropyl) (Z)-henicos-11-enoate

C51H96O6 (804.7206515999999)

[3-octanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] heptacosanoate

C51H96O6 (804.7206515999999)

[2-[(Z)-nonadec-9-enoyl]oxy-3-octanoyloxypropyl] henicosanoate

C51H96O6 (804.7206515999999)

[3-nonanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] hexacosanoate

C51H96O6 (804.7206515999999)

(3-octanoyloxy-2-tetradecanoyloxypropyl) (Z)-hexacos-15-enoate

C51H96O6 (804.7206515999999)

[3-nonanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] pentacosanoate

C51H96O6 (804.7206515999999)

[3-nonanoyloxy-2-[(Z)-octadec-9-enoyl]oxypropyl] henicosanoate

C51H96O6 (804.7206515999999)

(2-octadecanoyloxy-3-octanoyloxypropyl) (Z)-docos-13-enoate

C51H96O6 (804.7206515999999)

[2-[(Z)-nonadec-9-enoyl]oxy-3-nonanoyloxypropyl] icosanoate

C51H96O6 (804.7206515999999)

1,3-di(pentadecanoyloxy)propan-2-yl (Z)-16,16,17,17,18,18,18-heptadeuteriooctadec-9-enoate

C51H96O6 (804.7206515999999)

(2-nonadecanoyloxy-3-nonanoyloxypropyl) (Z)-icos-11-enoate

C51H96O6 (804.7206515999999)

[3-octanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] pentacosanoate

C51H96O6 (804.7206515999999)

(3-nonanoyloxy-2-tridecanoyloxypropyl) (Z)-hexacos-15-enoate

C51H96O6 (804.7206515999999)

(2-hexadecanoyloxy-3-tetradecanoyloxypropyl) (Z)-octadec-9-enoate

C51H96O6 (804.7206515999999)

(3-decanoyloxy-2-hexadecanoyloxypropyl) (Z)-docos-13-enoate

C51H96O6 (804.7206515999999)

[2-heptadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] octadecanoate

C51H96O6 (804.7206515999999)

[2-[(Z)-hexadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] octadecanoate

C51H96O6 (804.7206515999999)

(2-tetradecanoyloxy-3-tridecanoyloxypropyl) (Z)-henicos-11-enoate

C51H96O6 (804.7206515999999)

[3-decanoyloxy-2-[(Z)-hexadec-9-enoyl]oxypropyl] docosanoate

C51H96O6 (804.7206515999999)

[2-[(Z)-heptadec-9-enoyl]oxy-3-tridecanoyloxypropyl] octadecanoate

C51H96O6 (804.7206515999999)

(2-heptadecanoyloxy-3-tridecanoyloxypropyl) (Z)-octadec-9-enoate

C51H96O6 (804.7206515999999)

[3-decanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] tricosanoate

C51H96O6 (804.7206515999999)

[3-tridecanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] docosanoate

C51H96O6 (804.7206515999999)

[2-[(Z)-hexadec-9-enoyl]oxy-3-undecanoyloxypropyl] henicosanoate

C51H96O6 (804.7206515999999)

[3-dodecanoyloxy-2-[(Z)-heptadec-9-enoyl]oxypropyl] nonadecanoate

C51H96O6 (804.7206515999999)

(3-decanoyloxy-2-octadecanoyloxypropyl) (Z)-icos-11-enoate

C51H96O6 (804.7206515999999)

(2-octadecanoyloxy-3-undecanoyloxypropyl) (Z)-nonadec-9-enoate

C51H96O6 (804.7206515999999)

(3-dodecanoyloxy-2-pentadecanoyloxypropyl) (Z)-henicos-11-enoate

C51H96O6 (804.7206515999999)

[1-decanoyloxy-3-[(Z)-nonadec-9-enoyl]oxypropan-2-yl] nonadecanoate

C51H96O6 (804.7206515999999)

(3-decanoyloxy-2-tetradecanoyloxypropyl) (Z)-tetracos-13-enoate

C51H96O6 (804.7206515999999)

[2-[(Z)-heptadec-9-enoyl]oxy-3-undecanoyloxypropyl] icosanoate

C51H96O6 (804.7206515999999)

[3-dodecanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] tricosanoate

C51H96O6 (804.7206515999999)

[3-dodecanoyloxy-2-[(Z)-hexadec-9-enoyl]oxypropyl] icosanoate

C51H96O6 (804.7206515999999)

[2-[(Z)-tridec-9-enoyl]oxy-3-undecanoyloxypropyl] tetracosanoate

C51H96O6 (804.7206515999999)

2,3-di(dodecanoyloxy)propyl (Z)-tetracos-13-enoate

C51H96O6 (804.7206515999999)

[3-decanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] pentacosanoate

C51H96O6 (804.7206515999999)

[2-[(Z)-tetradec-9-enoyl]oxy-3-undecanoyloxypropyl] tricosanoate

C51H96O6 (804.7206515999999)

[1-[(Z)-heptadec-9-enoyl]oxy-3-tetradecanoyloxypropan-2-yl] heptadecanoate

C51H96O6 (804.7206515999999)

[2-tetradecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] henicosanoate

C51H96O6 (804.7206515999999)

(2-heptadecanoyloxy-3-undecanoyloxypropyl) (Z)-icos-11-enoate

C51H96O6 (804.7206515999999)

[2-[(Z)-hexadec-9-enoyl]oxy-3-tridecanoyloxypropyl] nonadecanoate

C51H96O6 (804.7206515999999)

(2-hexadecanoyloxy-3-undecanoyloxypropyl) (Z)-henicos-11-enoate

C51H96O6 (804.7206515999999)

[3-decanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] tetracosanoate

C51H96O6 (804.7206515999999)

2,3-di(decanoyloxy)propyl (Z)-octacos-17-enoate

C51H96O6 (804.7206515999999)

(2-pentadecanoyloxy-3-undecanoyloxypropyl) (Z)-docos-13-enoate

C51H96O6 (804.7206515999999)

[2-hexadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] nonadecanoate

C51H96O6 (804.7206515999999)

(3-decanoyloxy-2-heptadecanoyloxypropyl) (Z)-henicos-11-enoate

C51H96O6 (804.7206515999999)

2,3-di(tridecanoyloxy)propyl (Z)-docos-13-enoate

C51H96O6 (804.7206515999999)

[2-[(Z)-octadec-9-enoyl]oxy-3-undecanoyloxypropyl] nonadecanoate

C51H96O6 (804.7206515999999)

[3-decanoyloxy-2-[(Z)-heptadec-9-enoyl]oxypropyl] henicosanoate

C51H96O6 (804.7206515999999)

[1-dodecanoyloxy-3-[(Z)-octadec-9-enoyl]oxypropan-2-yl] octadecanoate

C51H96O6 (804.7206515999999)

[2-pentadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] icosanoate

C51H96O6 (804.7206515999999)

(2-tridecanoyloxy-3-undecanoyloxypropyl) (Z)-tetracos-13-enoate

C51H96O6 (804.7206515999999)

(3-dodecanoyloxy-2-tetradecanoyloxypropyl) (Z)-docos-13-enoate

C51H96O6 (804.7206515999999)

2,3-di(undecanoyloxy)propyl (Z)-hexacos-15-enoate

C51H96O6 (804.7206515999999)

[2-[(Z)-pentadec-9-enoyl]oxy-3-undecanoyloxypropyl] docosanoate

C51H96O6 (804.7206515999999)

[3-decanoyloxy-2-[(Z)-octadec-9-enoyl]oxypropyl] icosanoate

C51H96O6 (804.7206515999999)

(3-decanoyloxy-2-dodecanoyloxypropyl) (Z)-hexacos-15-enoate

C51H96O6 (804.7206515999999)

[2-[(Z)-hexadec-9-enoyl]oxy-3-pentadecanoyloxypropyl] heptadecanoate

C51H96O6 (804.7206515999999)

2,3-di(pentadecanoyloxy)propyl (Z)-octadec-9-enoate

C51H96O6 (804.7206515999999)

(2-hexadecanoyloxy-3-pentadecanoyloxypropyl) (Z)-heptadec-9-enoate

C51H96O6 (804.7206515999999)

(1-hydroxy-3-tricosanoyloxypropan-2-yl) (Z)-hexacos-15-enoate

C52H100O5 (804.757035)

[3-hydroxy-2-[(Z)-tetracos-13-enoyl]oxypropyl] pentacosanoate

C52H100O5 (804.757035)

[2-[(Z)-docos-13-enoyl]oxy-3-hydroxypropyl] heptacosanoate

C52H100O5 (804.757035)

[3-pentadecanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] octadecanoate

C51H96O6 (804.7206515999999)

[2-pentadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] nonadecanoate

C51H96O6 (804.7206515999999)

[2-[(Z)-pentadec-9-enoyl]oxy-3-tridecanoyloxypropyl] icosanoate

C51H96O6 (804.7206515999999)

[2-hexadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] octadecanoate

C51H96O6 (804.7206515999999)

(2-pentadecanoyloxy-3-tetradecanoyloxypropyl) (Z)-nonadec-9-enoate

C51H96O6 (804.7206515999999)

[2-heptadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] heptadecanoate

C51H96O6 (804.7206515999999)

2,3-di(tetradecanoyloxy)propyl (Z)-icos-11-enoate

C51H96O6 (804.7206515999999)

[3-dodecanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] henicosanoate

C51H96O6 (804.7206515999999)

(3-dodecanoyloxy-2-hexadecanoyloxypropyl) (Z)-icos-11-enoate

C51H96O6 (804.7206515999999)

[2-[(Z)-tetradec-9-enoyl]oxy-3-tridecanoyloxypropyl] henicosanoate

C51H96O6 (804.7206515999999)

(3-dodecanoyloxy-2-heptadecanoyloxypropyl) (Z)-nonadec-9-enoate

C51H96O6 (804.7206515999999)

[2-hexadecanoyloxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] heptadecanoate

C51H96O6 (804.7206515999999)

(2-hexadecanoyloxy-3-tridecanoyloxypropyl) (Z)-nonadec-9-enoate

C51H96O6 (804.7206515999999)

(2-pentadecanoyloxy-3-tridecanoyloxypropyl) (Z)-icos-11-enoate

C51H96O6 (804.7206515999999)

[2-[(Z)-pentadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] nonadecanoate

C51H96O6 (804.7206515999999)

[3-tetradecanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] icosanoate

C51H96O6 (804.7206515999999)

[3-dodecanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] docosanoate

C51H96O6 (804.7206515999999)

(2-tetradecanoyloxy-3-tridecanoyloxypropyl) (Z)-henicos-9-enoate

C51H96O6 (804.7206515999999)

[2-tetradecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] henicosanoate

C51H96O6 (804.7206515999999)

[2-[(Z)-hexadec-7-enoyl]oxy-3-tetradecanoyloxypropyl] octadecanoate

C51H96O6 (804.7206515999999)

[2-[(Z)-heptadec-7-enoyl]oxy-3-tridecanoyloxypropyl] octadecanoate

C51H96O6 (804.7206515999999)

(2-heptadecanoyloxy-3-tridecanoyloxypropyl) (Z)-octadec-11-enoate

C51H96O6 (804.7206515999999)

(3-dodecanoyloxy-2-tetradecanoyloxypropyl) (Z)-docos-11-enoate

C51H96O6 (804.7206515999999)

[3-tridecanoyloxy-2-[(Z)-tridec-8-enoyl]oxypropyl] docosanoate

C51H96O6 (804.7206515999999)

[3-[(Z)-dodec-5-enoyl]oxy-2-tetradecanoyloxypropyl] docosanoate

C51H96O6 (804.7206515999999)

[2-[(Z)-hexadec-7-enoyl]oxy-3-tridecanoyloxypropyl] nonadecanoate

C51H96O6 (804.7206515999999)

2,3-di(tridecanoyloxy)propyl (Z)-docos-11-enoate

C51H96O6 (804.7206515999999)

[3-dodecanoyloxy-2-[(Z)-heptadec-7-enoyl]oxypropyl] nonadecanoate

C51H96O6 (804.7206515999999)

[3-[(Z)-dodec-5-enoyl]oxy-2-hexadecanoyloxypropyl] icosanoate

C51H96O6 (804.7206515999999)

[2-hexadecanoyloxy-3-[(Z)-hexadec-7-enoyl]oxypropyl] hexadecanoate

C51H96O6 (804.7206515999999)

2,3-di(pentadecanoyloxy)propyl (Z)-octadec-11-enoate

C51H96O6 (804.7206515999999)

[3-dodecanoyloxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] icosanoate

C51H96O6 (804.7206515999999)

(2-hexadecanoyloxy-3-pentadecanoyloxypropyl) (Z)-heptadec-7-enoate

C51H96O6 (804.7206515999999)

[2-hexadecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] nonadecanoate

C51H96O6 (804.7206515999999)

[2-pentadecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] icosanoate

C51H96O6 (804.7206515999999)

[2-heptadecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] octadecanoate

C51H96O6 (804.7206515999999)

(3-dodecanoyloxy-2-pentadecanoyloxypropyl) (Z)-henicos-9-enoate

C51H96O6 (804.7206515999999)

[3-[(Z)-dodec-5-enoyl]oxy-2-octadecanoyloxypropyl] octadecanoate

C51H96O6 (804.7206515999999)

(2-hexadecanoyloxy-3-tetradecanoyloxypropyl) (Z)-octadec-11-enoate

C51H96O6 (804.7206515999999)

[1-dodecanoyloxy-3-[(Z)-octadec-11-enoyl]oxypropan-2-yl] octadecanoate

C51H96O6 (804.7206515999999)

[2-[(Z)-hexadec-7-enoyl]oxy-3-pentadecanoyloxypropyl] heptadecanoate

C51H96O6 (804.7206515999999)

[1-[(Z)-heptadec-7-enoyl]oxy-3-tetradecanoyloxypropan-2-yl] heptadecanoate

C51H96O6 (804.7206515999999)

[3-[(Z)-dodec-5-enoyl]oxy-2-heptadecanoyloxypropyl] nonadecanoate

C51H96O6 (804.7206515999999)

[3-[(Z)-dodec-5-enoyl]oxy-2-pentadecanoyloxypropyl] henicosanoate

C51H96O6 (804.7206515999999)

2-[(2-Docosanoyloxy-3-hexadecoxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[Hydroxy-(2-icosanoyloxy-3-octadecoxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[Hydroxy-(3-icosoxy-2-octadecanoyloxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

[(2R)-2-[(E)-hexadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] octadecanoate

C51H96O6 (804.7206515999999)

[(2S)-1-hydroxy-3-tricosanoyloxypropan-2-yl] (E)-hexacos-5-enoate

C52H100O5 (804.757035)

1,3-di(pentadecanoyloxy)propan-2-yl (E)-octadec-11-enoate

C51H96O6 (804.7206515999999)

[(2R)-2-hexadecanoyloxy-3-[(E)-hexadec-9-enoyl]oxypropyl] hexadecanoate

C51H96O6 (804.7206515999999)

[(2S)-1-hydroxy-3-[(E)-tetracos-15-enoyl]oxypropan-2-yl] pentacosanoate

C52H100O5 (804.757035)

[(2S)-3-hydroxy-2-[(E)-tetracos-15-enoyl]oxypropyl] pentacosanoate

C52H100O5 (804.757035)

[(2R)-2-hexadecanoyloxy-3-tetradecanoyloxypropyl] (E)-octadec-11-enoate

C51H96O6 (804.7206515999999)

[(2S)-3-hydroxy-2-tricosanoyloxypropyl] (E)-hexacos-5-enoate

C52H100O5 (804.757035)

2-[Hydroxy-(2-tetracosanoyloxy-3-tetradecoxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[(3-Decoxy-2-octacosanoyloxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[Hydroxy-(2-hydroxy-3-octatriacontanoyloxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[(3-Dodecoxy-2-hexacosanoyloxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[(3-Heptacosoxy-2-undecanoyloxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[Hydroxy-(2-pentacosanoyloxy-3-tridecoxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[Hydroxy-(3-pentacosoxy-2-tridecanoyloxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[(2-Henicosanoyloxy-3-heptadecoxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[Hydroxy-(2-nonadecanoyloxy-3-nonadecoxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[Hydroxy-(3-pentadecoxy-2-tricosanoyloxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[(3-Docosoxy-2-hexadecanoyloxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[Hydroxy-(3-tetracosoxy-2-tetradecanoyloxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[Hydroxy-(2-pentadecanoyloxy-3-tricosoxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[(2-Heptacosanoyloxy-3-undecoxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[(2-Decanoyloxy-3-octacosoxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[(2-Dodecanoyloxy-3-hexacosoxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

2-[(3-Henicosoxy-2-heptadecanoyloxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C46H95NO7P+ (804.684579)

1-(9Z-tetradecenoyl)-2-hexadecanoyl-3-octadecanoyl-sn-glycerol

C51H96O6 (804.7206515999999)

1-Myristoyl-2-oleoyl-3-palmitoyl-glycerol

C51H96O6 (804.7206515999999)

1-Myristoyl-2-palmitoyl-3-vaccenoyl-glycerol

C51H96O6 (804.7206515999999)

1-Myristoyl-2-stearoyl-3-palmitoleoyl-glycerol

C51H96O6 (804.7206515999999)

1-Myristoyl-2-arachidonyl-3-myristoleoyl-glycerol

C51H96O6 (804.7206515999999)

1-Myristoyl-2-vaccenoyl-3-palmitoyl-glycerol

C51H96O6 (804.7206515999999)

1-Myristoyl-2-eicosenoyl-3-myristoyl-glycerol

C51H96O6 (804.7206515999999)

1-Pentadecanoyl-2-pentadecanoyl-3-vaccenoyl-glycerol

C51H96O6 (804.7206515999999)

1-Pentadecanoyl-2-vaccenoyl-3-pentadecanoyl-glycerol

C51H96O6 (804.7206515999999)

1-Pentadecanoyl-2-oleoyl-3-pentadecanoyl-glycerol

C51H96O6 (804.7206515999999)

1-Palmitoyl-2-myristoyl-3-vaccenoyl-glycerol

C51H96O6 (804.7206515999999)

1-Palmitoyl-2-stearoyl-3-myristoleoyl-glycerol

C51H96O6 (804.7206515999999)

1-Palmitoyl-2-myristoleoyl-3-stearoyl-glycerol

C51H96O6 (804.7206515999999)

1-Arachidonyl-2-myristoyl-3-myristoleoyl-glycerol

C51H96O6 (804.7206515999999)

1-Pentadecanoyl-2-palmitoleoyl-3-stearyl-glycerol

C52H100O5 (804.757035)

1,2-dipalmitoyl-3-palmitoleoyl-sn-glycerol

C51H96O6 (804.7206515999999)

A triglyceride in which the acyl groups at positions 1 and 2 are specified as palmitoyl while that at position 3 is specified as palmitoleoyl.

TG(16:0/14:0/18:1(9Z))[iso6]

C51H96O6 (804.7206515999999)

TG(18:0/14:0/16:1(9Z))[iso6]

C51H96O6 (804.7206515999999)

1-tetradecanoyl-2-hexadecanoyl-3-(9Z-octadecenoyl)-sn-glycerol

C51H96O6 (804.7206515999999)

1,2-ditetradecanoyl-3-(11Z-eicosenoyl)-sn-glycerol

C51H96O6 (804.7206515999999)

1,2-dipentadecanoyl-3-(9Z-octadecenoyl)-sn-glycerol

C51H96O6 (804.7206515999999)

1-tetradecanoyl-2-(9Z-tetradecenoyl)-3-eicosanoyl-sn-glycerol

C51H96O6 (804.7206515999999)

1-tetradecanoyl-2-(9Z-hexadecenoyl)-3-octadecanoyl-sn-glycerol

C51H96O6 (804.7206515999999)

triacylglycerol 48:1

C51H96O6 (804.7206515999999)

A triglyceride in which the three acyl groups contain a total of 48 carbons and 1 double bond.

1-tetradecanoyl-2-hexadecanoyl-3-[(9Z)-octadecenoyl]-sn-glycerol

C51H96O6 (804.7206515999999)

A triacyl-sn-glycerol in which the which the acyl groups at positions 1, 2 and 3 are specified as tetradecanoyl, hexadecanoyl and (9Z)-octadecenoyl respectively.

CmE(28:1)

C56H100O2 (804.77229)

Provides by LipidSearch Vendor. © Copyright 2006-2024 Thermo Fisher Scientific Inc. All rights reserved

DG(49:1)

C52H100O5 (804.757035)

Provides by LipidSearch Vendor. © Copyright 2006-2024 Thermo Fisher Scientific Inc. All rights reserved

NA-Orn 45:1

C50H96N2O5 (804.7318846)

FAHFA 54:7;O

C54H92O4 (804.6995231999999)

DG 14:1_35:0

C52H100O5 (804.757035)

DG 15:1_34:0

C52H100O5 (804.757035)

DG 16:1_33:0

C52H100O5 (804.757035)

DG 17:1_32:0

C52H100O5 (804.757035)

DG 18:1_31:0

C52H100O5 (804.757035)

DG 20:1_29:0

C52H100O5 (804.757035)

DG 22:1_27:0

C52H100O5 (804.757035)

DG 23:0_26:1

C52H100O5 (804.757035)

DG 24:1_25:0

C52H100O5 (804.757035)

DG 49:1

C52H100O5 (804.757035)

MeDAG 49:1

C52H100O5 (804.757035)

TG 10:0_12:0_26:1

C51H96O6 (804.7206515999999)

TG 10:0_14:0_24:1

C51H96O6 (804.7206515999999)

TG 10:0_14:1_24:0

C51H96O6 (804.7206515999999)

TG 10:0_15:1_23:0

C51H96O6 (804.7206515999999)

TG 10:0_16:0_22:1

C51H96O6 (804.7206515999999)

TG 10:0_16:1_22:0

C51H96O6 (804.7206515999999)

TG 10:0_17:1_21:0

C51H96O6 (804.7206515999999)

TG 10:0_18:0_20:1

C51H96O6 (804.7206515999999)

TG 10:0_18:1_20:0

C51H96O6 (804.7206515999999)

TG 11:0_11:0_26:1

C51H96O6 (804.7206515999999)

TG 11:0_13:0_24:1

C51H96O6 (804.7206515999999)

TG 11:0_14:1_23:0

C51H96O6 (804.7206515999999)

TG 11:0_15:0_22:1

C51H96O6 (804.7206515999999)

TG 11:0_15:1_22:0

C51H96O6 (804.7206515999999)

TG 11:0_16:1_21:0

C51H96O6 (804.7206515999999)

TG 11:0_17:0_20:1

C51H96O6 (804.7206515999999)

TG 11:0_17:1_20:0

C51H96O6 (804.7206515999999)

TG 11:0_18:1_19:0

C51H96O6 (804.7206515999999)

TG 12:0_12:0_24:1

C51H96O6 (804.7206515999999)

TG 12:0_14:0_22:1

C51H96O6 (804.7206515999999)

TG 12:0_14:1_22:0

C51H96O6 (804.7206515999999)

TG 12:0_15:1_21:0

C51H96O6 (804.7206515999999)

TG 12:0_16:0_20:1

C51H96O6 (804.7206515999999)

TG 12:0_16:1_20:0

C51H96O6 (804.7206515999999)

TG 12:0_17:1_19:0

C51H96O6 (804.7206515999999)

TG 12:0_18:0_18:1

C51H96O6 (804.7206515999999)

TG 13:0_13:0_22:1

C51H96O6 (804.7206515999999)

TG 13:0_14:1_21:0

C51H96O6 (804.7206515999999)

TG 13:0_15:0_20:1

C51H96O6 (804.7206515999999)

TG 13:0_15:1_20:0

C51H96O6 (804.7206515999999)

TG 13:0_16:1_19:0

C51H96O6 (804.7206515999999)

TG 13:0_17:0_18:1

C51H96O6 (804.7206515999999)

TG 13:0_17:1_18:0

C51H96O6 (804.7206515999999)

TG 14:0_14:0_20:1

C51H96O6 (804.7206515999999)

TG 14:0_14:1_20:0

C51H96O6 (804.7206515999999)

TG 14:0_15:1_19:0

C51H96O6 (804.7206515999999)

TG 14:0_16:0_18:1

C51H96O6 (804.7206515999999)

TG 14:0/16:0/18:1

C51H96O6 (804.7206515999999)

TG 14:0_16:1_18:0

C51H96O6 (804.7206515999999)

TG 14:0_17:0_17:1

C51H96O6 (804.7206515999999)

TG 14:1_15:0_19:0

C51H96O6 (804.7206515999999)

TG 14:1_16:0_18:0

C51H96O6 (804.7206515999999)

TG 14:1_17:0_17:0

C51H96O6 (804.7206515999999)

TG 15:0_15:0_18:1

C51H96O6 (804.7206515999999)

TG 15:0_15:1_18:0

C51H96O6 (804.7206515999999)

TG 15:0_16:0_17:1

C51H96O6 (804.7206515999999)

TG 15:0_16:1_17:0

C51H96O6 (804.7206515999999)

TG 15:1_16:0_17:0

C51H96O6 (804.7206515999999)

TG 16:0/14:0/18:1

C51H96O6 (804.7206515999999)

TG 16:0_16:0_16:1

C51H96O6 (804.7206515999999)

TG 16:0/16:0/16:1

C51H96O6 (804.7206515999999)

TG 16:0/16:0/16:1(9Z)

C51H96O6 (804.7206515999999)

TG 48:1_12:0

C51H96O6 (804.7206515999999)

TG 48:1_14:0

C51H96O6 (804.7206515999999)

TG 48:1_14:1

C51H96O6 (804.7206515999999)

TG 48:1_15:0

C51H96O6 (804.7206515999999)

TG 48:1_16:0

C51H96O6 (804.7206515999999)

TG 48:1_16:1

C51H96O6 (804.7206515999999)

TG 48:1_17:0

C51H96O6 (804.7206515999999)

TG 48:1_18:0

C51H96O6 (804.7206515999999)

TG 48:1_18:1

C51H96O6 (804.7206515999999)

TG 48:1_20:0

C51H96O6 (804.7206515999999)

TG 48:1_20:1

C51H96O6 (804.7206515999999)

TG 48:1_22:1

C51H96O6 (804.7206515999999)

3a,5a,5b,8,8,11a-hexamethyl-1-(prop-1-en-2-yl)-hexadecahydrocyclopenta[a]chrysen-9-yl hexacosanoate

C56H100O2 (804.77229)

(2r)-2,3-bis(hexadecanoyloxy)propyl (9z)-hexadec-9-enoate

C51H96O6 (804.7206515999999)

(1r,3ar,5ar,5br,7ar,9s,11ar,11br,13ar,13br)-3a,5a,5b,8,8,11a-hexamethyl-1-(prop-1-en-2-yl)-hexadecahydrocyclopenta[a]chrysen-9-yl hexacosanoate

C56H100O2 (804.77229)