Exact Mass: 571.161335

Exact Mass Matches: 571.161335

Found 8 metabolites which its exact mass value is equals to given mass value 571.161335, within given mass tolerance error 0.01 dalton. Try search metabolite list with more accurate mass tolerance error 0.001 dalton.

Cyclochlorotine

(3S,7R,9S,13S,16R)-17,18-dichloro-13-ethyl-3,9-bis(hydroxymethyl)-7-phenyl-1,4,8,11,14-pentazabicyclo[14.3.0]nonadecane-2,5,10,12,15-pentone

C24H31Cl2N5O7 (571.1600436)


   

Islanditoxin

17,18-dichloro-7-ethyl-1,5,8,11-tetrahydroxy-10,13-bis(hydroxymethyl)-3-phenyl-3H,4H,7H,10H,13H,14H,16H,17H,18H,18aH-pyrrolo[2,1-c]1,4,7,10,13-pentaazacyclohexadecan-14-one

C24H31Cl2N5O7 (571.1600436)


Islanditoxin is a mycotoxin produced by the common food storage mould Penicillium islandicu

   

Cyclochlorotine

17,18-dichloro-3-ethyl-6,13-bis(hydroxymethyl)-9-phenyl-octadecahydropyrrolo[1,2-d]1,4,7,10,13-pentaazacyclohexadecane-1,4,7,11,14-pentone

C24H31Cl2N5O7 (571.1600436)


Cyclochlorotine is a mycotoxin produced by the common food storage mould Penicillium islandicu

   

Telaglenastat

2-(pyridin-2-yl)-N-{5-[4-(6-{2-[3-(trifluoromethoxy)phenyl]acetamido}pyridazin-3-yl)butyl]-1,3,4-thiadiazol-2-yl}acetamide

C26H24F3N7O3S (571.161335)


C471 - Enzyme Inhibitor Telaglenastat (CB-839) is a first-in-class, selective, reversible and orally active glutaminase 1 (GLS1) inhibitor. Telaglenastat selectively inhibits GLS1 splice variants KGA (kidney-type glutaminase) and GAC (glutaminase C) compared to GLS2. The IC50s are 23 nM and 28 nM for endogenous glutaminase in mouse kidney and brain, respectively. Telaglenastat inuduces autophagy and has antitumor activity[1].

   

Islanditoxin

17,18-dichloro-7-ethyl-10,13-bis(hydroxymethyl)-3-phenyl-octadecahydropyrrolo[2,1-c]1,4,7,10,13-pentaazacyclohexadecane-1,5,8,11,14-pentone

C24H31Cl2N5O7 (571.1600436)


   

Telaglenastat

Telaglenastat

C26H24F3N7O3S (571.161335)


C471 - Enzyme Inhibitor Telaglenastat (CB-839) is a first-in-class, selective, reversible and orally active glutaminase 1 (GLS1) inhibitor. Telaglenastat selectively inhibits GLS1 splice variants KGA (kidney-type glutaminase) and GAC (glutaminase C) compared to GLS2. The IC50s are 23 nM and 28 nM for endogenous glutaminase in mouse kidney and brain, respectively. Telaglenastat inuduces autophagy and has antitumor activity[1].

   

(3S,7R,9S,13S,16R)-17,18-dichloro-13-ethyl-3,9-bis(hydroxymethyl)-7-phenyl-1,4,8,11,14-pentazabicyclo[14.3.0]nonadecane-2,5,10,12,15-pentone

(3S,7R,9S,13S,16R)-17,18-dichloro-13-ethyl-3,9-bis(hydroxymethyl)-7-phenyl-1,4,8,11,14-pentazabicyclo[14.3.0]nonadecane-2,5,10,12,15-pentone

C24H31Cl2N5O7 (571.1600436)


   

17,18-Dichloro-9-ethyl-3,6-bis(hydroxymethyl)-13-phenyl-1,4,7,10,14-pentazabicyclo[14.3.0]nonadecane-2,5,8,11,15-pentone

17,18-Dichloro-9-ethyl-3,6-bis(hydroxymethyl)-13-phenyl-1,4,7,10,14-pentazabicyclo[14.3.0]nonadecane-2,5,8,11,15-pentone

C24H31Cl2N5O7 (571.1600436)