Exact Mass: 818.7475

TG(15:0/18:1(9Z)/16:0)

C52H98O6 (818.7363)

TG(15:0/18:1(9Z)/16:0) is a monooleic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/18:1(9Z)/16:0), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of oleic acid at the C-2 position and one chain of palmitic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols. TG(15:0/18:1(9Z)/16:0) is a monooleic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/18:1(9Z)/16:0), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of oleic acid at the C-2 position and one chain of palmitic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)

TG(15:0/20:0/14:1(9Z))

C52H98O6 (818.7363)

TG(15:0/20:0/14:1(9Z)) is a monoarachidic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/20:0/14:1(9Z)), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of arachidic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/20:1(11Z)/14:0)

C52H98O6 (818.7363)

TG(15:0/20:1(11Z)/14:0) is a monoeicosenoic acid triglyceride. Triglycerides (TGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/20:1(11Z)/14:0), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of eicosenoic acid at the C-2 position and one chain of myristic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/15:0/20:1(11Z))

C52H98O6 (818.7363)

TG(14:0/15:0/20:1(11Z)) is a monoeicosenoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/15:0/20:1(11Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of eicosenoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/18:1(11Z)/O-18:0)

C53H102O5 (818.7727)

TG(14:0/18:1(11Z)/O-18:0) is a monovaccenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/18:1(11Z)/O-18:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of vaccenic acid at the C-2 position and one chain of Stearyl alcohol at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/18:1(9Z)/O-18:0)

C53H102O5 (818.7727)

TG(14:0/18:1(9Z)/O-18:0) is a monooleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/18:1(9Z)/O-18:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of oleic acid at the C-2 position and one chain of Stearyl alcohol at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/20:1(11Z)/15:0)

C52H98O6 (818.7363)

TG(14:0/20:1(11Z)/15:0) is a monoeicosenoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/20:1(11Z)/15:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of eicosenoic acid at the C-2 position and one chain of pentadecanoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/O-18:0/18:1(11Z))

C53H102O5 (818.7727)

TG(14:0/O-18:0/18:1(11Z)) is a monoStearyl alcohol triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/O-18:0/18:1(11Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of Stearyl alcohol at the C-2 position and one chain of vaccenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/O-18:0/18:1(9Z))

C53H102O5 (818.7727)

TG(14:0/O-18:0/18:1(9Z)) is a monoStearyl alcohol triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/O-18:0/18:1(9Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of Stearyl alcohol at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/14:0/20:1(11Z))

C52H98O6 (818.7363)

TG(15:0/14:0/20:1(11Z)) is a monoeicosenoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/14:0/20:1(11Z)), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of eicosenoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/16:0/18:1(11Z))

C52H98O6 (818.7363)

TG(15:0/16:0/18:1(11Z)) is a monovaccenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/16:0/18:1(11Z)), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of vaccenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/16:0/18:1(9Z))

C52H98O6 (818.7363)

TG(15:0/16:0/18:1(9Z)) is a monooleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/16:0/18:1(9Z)), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/18:0/16:1(9Z))

C52H98O6 (818.7363)

TG(15:0/18:0/16:1(9Z)) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/18:0/16:1(9Z)), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of stearic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/14:1(9Z)/20:0)

C52H98O6 (818.7363)

TG(15:0/14:1(9Z)/20:0) is a monoarachidic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/14:1(9Z)/20:0), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of arachidic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/16:1(9Z)/18:0)

C52H98O6 (818.7363)

TG(15:0/16:1(9Z)/18:0) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/16:1(9Z)/18:0), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of stearic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/18:1(11Z)/16:0)

C52H98O6 (818.7363)

TG(15:0/18:1(11Z)/16:0) is a monovaccenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/18:1(11Z)/16:0), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of vaccenic acid at the C-2 position and one chain of palmitic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/15:0/18:1(11Z))

C52H98O6 (818.7363)

TG(16:0/15:0/18:1(11Z)) is a monovaccenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/15:0/18:1(11Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of vaccenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/15:0/18:1(9Z))

C52H98O6 (818.7363)

TG(16:0/15:0/18:1(9Z)) is a monooleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/15:0/18:1(9Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of oleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/16:1(9Z)/O-18:0)

C53H102O5 (818.7727)

TG(16:0/16:1(9Z)/O-18:0) is a monoStearyl alcohol triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/16:1(9Z)/O-18:0), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of Stearyl alcohol at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/O-18:0/16:1(9Z))

C53H102O5 (818.7727)

TG(16:0/O-18:0/16:1(9Z)) is a monoStearyl alcohol triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/O-18:0/16:1(9Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of Stearyl alcohol at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(18:0/15:0/16:1(9Z))

C52H98O6 (818.7363)

TG(18:0/15:0/16:1(9Z)) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:0/15:0/16:1(9Z)), in particular, consists of one chain of stearic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(18:0/14:1(9Z)/O-18:0)

C53H102O5 (818.7727)

TG(18:0/14:1(9Z)/O-18:0) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:0/14:1(9Z)/O-18:0), in particular, consists of one chain of stearic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of Stearyl alcohol at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(18:0/O-18:0/14:1(9Z))

C53H102O5 (818.7727)

TG(18:0/O-18:0/14:1(9Z)) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:0/O-18:0/14:1(9Z)), in particular, consists of one chain of stearic acid at the C-1 position, one chain of Stearyl alcohol at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(20:0/15:0/14:1(9Z))

C52H98O6 (818.7363)

TG(20:0/15:0/14:1(9Z)) is a monoarachidic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(20:0/15:0/14:1(9Z)), in particular, consists of one chain of arachidic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:1(9Z)/18:0/O-18:0)

C53H102O5 (818.7727)

TG(14:1(9Z)/18:0/O-18:0) is a monostearic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/18:0/O-18:0), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of stearic acid at the C-2 position and one chain of Stearyl alcohol at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:1(9Z)/16:0/O-18:0)

C53H102O5 (818.7727)

TG(16:1(9Z)/16:0/O-18:0) is a monoStearyl alcohol triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:1(9Z)/16:0/O-18:0), in particular, consists of one chain of palmitoleic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of Stearyl alcohol at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(18:1(11Z)/14:0/O-18:0)

C53H102O5 (818.7727)

TG(18:1(11Z)/14:0/O-18:0) is a monovaccenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:1(11Z)/14:0/O-18:0), in particular, consists of one chain of vaccenic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of Stearyl alcohol at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(18:1(9Z)/14:0/O-18:0)

C53H102O5 (818.7727)

TG(18:1(9Z)/14:0/O-18:0) is a monooleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(18:1(9Z)/14:0/O-18:0), in particular, consists of one chain of oleic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of Stearyl alcohol at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

Etiroxate

C18H17I4NO4 (818.7337)

D006730 - Hormones, Hormone Substitutes, and Hormone Antagonists > D006728 - Hormones C147908 - Hormone Therapy Agent > C548 - Therapeutic Hormone > C1553 - Thyroid Agent D057847 - Lipid Regulating Agents > D000960 - Hypolipidemic Agents D009676 - Noxae > D000963 - Antimetabolites

Triacylglycerol 15:0-16:0-18:1

C52H98O6 (818.7363)

alpha-Spinasteroloctacosanoate

C57H102O2 (818.7879)

TG 16:0-17:1-16:0-d5

C52H98O6 (818.7363)

TG(14:0/14:1(9Z)/21:0)[iso6]

C52H98O6 (818.7363)

TG(14:0/15:0/20:1(11Z))[iso6]

C52H98O6 (818.7363)

TG(14:0/15:1(9Z)/20:0)[iso6]

C52H98O6 (818.7363)

TG(14:0/16:0/19:1(9Z))[iso6]

C52H98O6 (818.7363)

TG(14:0/16:1(9Z)/19:0)[iso6]

C52H98O6 (818.7363)

TG(14:0/17:0/18:1(9Z))[iso6]

C52H98O6 (818.7363)

TG(14:0/17:1(9Z)/18:0)[iso6]

C52H98O6 (818.7363)

TG(14:1(9Z)/15:0/20:0)[iso6]

C52H98O6 (818.7363)

TG(14:1(9Z)/16:0/19:0)[iso6]

C52H98O6 (818.7363)

TG(14:1(9Z)/17:0/18:0)[iso6]

C52H98O6 (818.7363)

TG(15:0/15:1(9Z)/19:0)[iso6]

C52H98O6 (818.7363)

TG(15:0/16:0/18:1(9Z))[iso6]

C52H98O6 (818.7363)

TG(15:0/16:1(9Z)/18:0)[iso6]

C52H98O6 (818.7363)

TG(15:0/17:0/17:1(9Z))[iso6]

C52H98O6 (818.7363)

TG(15:1(9Z)/16:0/18:0)[iso6]

C52H98O6 (818.7363)

TG(16:0/16:1/17:0)[iso6]

C52H98O6 (818.7363)

TG(16:0/16:0/17:1)[iso3]

C52H98O6 (818.7363)

Triglyceride

C52H98O6 (818.7363)

TG(15:0/15:0/19:1(9Z))[iso3]

C52H98O6 (818.7363)

TG(15:1(9Z)/17:0/17:0)[iso3]

C52H98O6 (818.7363)

TG(12:0/15:0/22:1(11Z))[iso6]

C52H98O6 (818.7363)

TG(12:0/15:1(9Z)/22:0)[iso6]

C52H98O6 (818.7363)

TG(12:0/16:1(9Z)/21:0)[iso6]

C52H98O6 (818.7363)

TG(12:0/17:0/20:1(11Z))[iso6]

C52H98O6 (818.7363)

TG(12:0/17:1(9Z)/20:0)[iso6]

C52H98O6 (818.7363)

TG(12:0/18:0/19:1(9Z))[iso6]

C52H98O6 (818.7363)

TG(12:0/18:1(9Z)/19:0)[iso6]

C52H98O6 (818.7363)

TG(13:0/14:0/22:1(11Z))[iso6]

C52H98O6 (818.7363)

TG(13:0/14:1(9Z)/22:0)[iso6]

C52H98O6 (818.7363)

TG(13:0/15:1(9Z)/21:0)[iso6]

C52H98O6 (818.7363)

TG(13:0/16:0/20:1(11Z))[iso6]

C52H98O6 (818.7363)

TG(13:0/16:1(9Z)/20:0)[iso6]

C52H98O6 (818.7363)

TG(13:0/17:0/19:1(9Z))[iso6]

C52H98O6 (818.7363)

TG(13:0/17:1(9Z)/19:0)[iso6]

C52H98O6 (818.7363)

TG(13:0/18:0/18:1(9Z))[iso6]

C52H98O6 (818.7363)

TG 49:1

C52H98O6 (818.7363)

n,n-bis[2-[(1-oxooctadecyl)amino]ethyl]-decanediamid

C50H98N4O4 (818.7588)

DIOCTYLDODECYL IPDI

C52H102N2O4 (818.7839)

1-Pentadecanoyl-2-oleoyl-3-palmitoyl-glycerol

C52H98O6 (818.7363)

2-[hydroxy-[(2R)-3-icosoxy-2-nonadecanoyloxypropoxy]phosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

1,2-Dipalmitoyl-3-oleyl-rac-glycerol

C53H102O5 (818.7727)

NAOrn 26:1/20:0

C51H98N2O5 (818.7475)

NAOrn 21:1/25:0

C51H98N2O5 (818.7475)

NAOrn 24:0/22:1

C51H98N2O5 (818.7475)

NAOrn 25:0/21:1

C51H98N2O5 (818.7475)

NAOrn 24:1/22:0

C51H98N2O5 (818.7475)

NAOrn 22:0/24:1

C51H98N2O5 (818.7475)

NAOrn 22:1/24:0

C51H98N2O5 (818.7475)

NAOrn 20:1/26:0

C51H98N2O5 (818.7475)

NAOrn 20:0/26:1

C51H98N2O5 (818.7475)

NAOrn 26:0/20:1

C51H98N2O5 (818.7475)

[10,13-dimethyl-17-(6-methylheptan-2-yl)-2,3,4,7,8,9,11,12,14,15,16,17-dodecahydro-1H-cyclopenta[a]phenanthren-3-yl] (Z)-triacont-19-enoate

C57H102O2 (818.7879)

[1-[(8Z,11Z,14Z,17Z,20Z,23Z)-hexacosa-8,11,14,17,20,23-hexaenoxy]-3-hydroxypropan-2-yl] (15Z,18Z)-hexacosa-15,18-dienoate

C55H94O4 (818.7152)

[1-hydroxy-3-[(10Z,13Z,16Z,19Z,22Z,25Z)-octacosa-10,13,16,19,22,25-hexaenoxy]propan-2-yl] (13Z,16Z)-tetracosa-13,16-dienoate

C55H94O4 (818.7152)

[1-hydroxy-3-[(12Z,15Z,18Z,21Z)-tetracosa-12,15,18,21-tetraenoxy]propan-2-yl] (16Z,19Z,22Z,25Z)-octacosa-16,19,22,25-tetraenoate

C55H94O4 (818.7152)

[1-[(14Z,17Z,20Z,23Z)-hexacosa-14,17,20,23-tetraenoxy]-3-hydroxypropan-2-yl] (14Z,17Z,20Z,23Z)-hexacosa-14,17,20,23-tetraenoate

C55H94O4 (818.7152)

[1-hydroxy-3-[(7Z,10Z,13Z,16Z,19Z,22Z,25Z)-octacosa-7,10,13,16,19,22,25-heptaenoxy]propan-2-yl] (Z)-tetracos-13-enoate

C55H94O4 (818.7152)

[1-hydroxy-3-[(17Z,20Z)-octacosa-17,20-dienoxy]propan-2-yl] (6Z,9Z,12Z,15Z,18Z,21Z)-tetracosa-6,9,12,15,18,21-hexaenoate

C55H94O4 (818.7152)

[1-[(12Z,15Z,18Z)-hexacosa-12,15,18-trienoxy]-3-hydroxypropan-2-yl] (11Z,14Z,17Z,20Z,23Z)-hexacosa-11,14,17,20,23-pentaenoate

C55H94O4 (818.7152)

[1-hydroxy-3-[(13Z,16Z)-tetracosa-13,16-dienoxy]propan-2-yl] (10Z,13Z,16Z,19Z,22Z,25Z)-octacosa-10,13,16,19,22,25-hexaenoate

C55H94O4 (818.7152)

[1-hydroxy-3-[(16Z,19Z,22Z,25Z)-octacosa-16,19,22,25-tetraenoxy]propan-2-yl] (12Z,15Z,18Z,21Z)-tetracosa-12,15,18,21-tetraenoate

C55H94O4 (818.7152)

[1-[(Z)-hexacos-15-enoxy]-3-hydroxypropan-2-yl] (5Z,8Z,11Z,14Z,17Z,20Z,23Z)-hexacosa-5,8,11,14,17,20,23-heptaenoate

C55H94O4 (818.7152)

[1-hydroxy-3-[(6Z,9Z,12Z,15Z,18Z,21Z)-tetracosa-6,9,12,15,18,21-hexaenoxy]propan-2-yl] (17Z,20Z)-octacosa-17,20-dienoate

C55H94O4 (818.7152)

[1-hydroxy-3-[(14Z,17Z,20Z)-octacosa-14,17,20-trienoxy]propan-2-yl] (9Z,12Z,15Z,18Z,21Z)-tetracosa-9,12,15,18,21-pentaenoate

C55H94O4 (818.7152)

[1-[(5Z,8Z,11Z,14Z,17Z,20Z,23Z)-hexacosa-5,8,11,14,17,20,23-heptaenoxy]-3-hydroxypropan-2-yl] (Z)-hexacos-15-enoate

C55H94O4 (818.7152)

[1-[(11Z,14Z,17Z,20Z,23Z)-hexacosa-11,14,17,20,23-pentaenoxy]-3-hydroxypropan-2-yl] (12Z,15Z,18Z)-hexacosa-12,15,18-trienoate

C55H94O4 (818.7152)

[1-hydroxy-3-[(9Z,12Z,15Z,18Z,21Z)-tetracosa-9,12,15,18,21-pentaenoxy]propan-2-yl] (14Z,17Z,20Z)-octacosa-14,17,20-trienoate

C55H94O4 (818.7152)

[1-hydroxy-3-[(13Z,16Z,19Z,22Z,25Z)-octacosa-13,16,19,22,25-pentaenoxy]propan-2-yl] (10Z,13Z,16Z)-tetracosa-10,13,16-trienoate

C55H94O4 (818.7152)

[1-hydroxy-3-[(Z)-tetracos-13-enoxy]propan-2-yl] (7Z,10Z,13Z,16Z,19Z,22Z,25Z)-octacosa-7,10,13,16,19,22,25-heptaenoate

C55H94O4 (818.7152)

[1-hydroxy-3-[(10Z,13Z,16Z)-tetracosa-10,13,16-trienoxy]propan-2-yl] (13Z,16Z,19Z,22Z,25Z)-octacosa-13,16,19,22,25-pentaenoate

C55H94O4 (818.7152)

[1-[(15Z,18Z)-hexacosa-15,18-dienoxy]-3-hydroxypropan-2-yl] (8Z,11Z,14Z,17Z,20Z,23Z)-hexacosa-8,11,14,17,20,23-hexaenoate

C55H94O4 (818.7152)

(1-hydroxy-3-octanoyloxypropan-2-yl) (Z)-dotetracont-31-enoate

C53H102O5 (818.7727)

(1-docosanoyloxy-3-hydroxypropan-2-yl) (Z)-octacos-17-enoate

C53H102O5 (818.7727)

[3-hydroxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] pentatriacontanoate

C53H102O5 (818.7727)

(1-hydroxy-3-tetradecanoyloxypropan-2-yl) (Z)-hexatriacont-25-enoate

C53H102O5 (818.7727)

[3-hydroxy-2-[(Z)-icos-11-enoyl]oxypropyl] triacontanoate

C53H102O5 (818.7727)

[3-hydroxy-2-[(Z)-nonadec-9-enoyl]oxypropyl] hentriacontanoate

C53H102O5 (818.7727)

(1-hexadecanoyloxy-3-hydroxypropan-2-yl) (Z)-tetratriacont-23-enoate

C53H102O5 (818.7727)

[3-hydroxy-2-[(Z)-tridec-9-enoyl]oxypropyl] heptatriacontanoate

C53H102O5 (818.7727)

(1-hydroxy-3-octadecanoyloxypropan-2-yl) (Z)-dotriacont-21-enoate

C53H102O5 (818.7727)

[3-hydroxy-2-[(Z)-octadec-9-enoyl]oxypropyl] dotriacontanoate

C53H102O5 (818.7727)

(1-dodecanoyloxy-3-hydroxypropan-2-yl) (Z)-octatriacont-27-enoate

C53H102O5 (818.7727)

(1-hydroxy-3-icosanoyloxypropan-2-yl) (Z)-triacont-19-enoate

C53H102O5 (818.7727)

[2-[(Z)-heptadec-9-enoyl]oxy-3-hydroxypropyl] tritriacontanoate

C53H102O5 (818.7727)

[2-[(Z)-docos-13-enoyl]oxy-3-hydroxypropyl] octacosanoate

C53H102O5 (818.7727)

[2-[(Z)-henicos-11-enoyl]oxy-3-hydroxypropyl] nonacosanoate

C53H102O5 (818.7727)

(1-decanoyloxy-3-hydroxypropan-2-yl) (Z)-tetracont-29-enoate

C53H102O5 (818.7727)

[2-[(Z)-hexadec-9-enoyl]oxy-3-hydroxypropyl] tetratriacontanoate

C53H102O5 (818.7727)

[3-hydroxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] hexatriacontanoate

C53H102O5 (818.7727)

(2-nonanoyloxy-3-octanoyloxypropyl) (Z)-dotriacont-21-enoate

C52H98O6 (818.7363)

(3-docosoxy-2-octanoyloxypropyl) (Z)-icos-11-enoate

C53H102O5 (818.7727)

[3-[(Z)-icos-11-enoxy]-2-octanoyloxypropyl] docosanoate

C53H102O5 (818.7727)

(2-icosanoyloxy-3-octoxypropyl) (Z)-docos-13-enoate

C53H102O5 (818.7727)

(3-icosoxy-2-octanoyloxypropyl) (Z)-docos-13-enoate

C53H102O5 (818.7727)

[2-[(Z)-icos-11-enoyl]oxy-3-octoxypropyl] docosanoate

C53H102O5 (818.7727)

[3-[(Z)-docos-13-enoxy]-2-octanoyloxypropyl] icosanoate

C53H102O5 (818.7727)

[2-decanoyloxy-3-[(Z)-icos-11-enoxy]propyl] icosanoate

C53H102O5 (818.7727)

(3-octanoyloxy-2-tridecanoyloxypropyl) (Z)-octacos-17-enoate

C52H98O6 (818.7363)

[3-octadecoxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] octadecanoate

C53H102O5 (818.7727)

[2-[(Z)-heptadec-9-enoyl]oxy-3-nonanoyloxypropyl] tricosanoate

C52H98O6 (818.7363)

[3-[(Z)-icos-11-enoxy]-2-tetradecanoyloxypropyl] hexadecanoate

C53H102O5 (818.7727)

[2-[(Z)-hexadec-9-enoyl]oxy-3-tetradecoxypropyl] icosanoate

C53H102O5 (818.7727)

[2-[(Z)-hexadec-9-enoyl]oxy-3-nonanoyloxypropyl] tetracosanoate

C52H98O6 (818.7363)

(3-nonanoyloxy-2-tetradecanoyloxypropyl) (Z)-hexacos-15-enoate

C52H98O6 (818.7363)

[2-[(Z)-tetradec-9-enoyl]oxy-3-tetradecoxypropyl] docosanoate

C53H102O5 (818.7727)

(3-dodecoxy-2-hexadecanoyloxypropyl) (Z)-docos-13-enoate

C53H102O5 (818.7727)

[2-tetradecanoyloxy-3-[(Z)-tetradec-9-enoxy]propyl] docosanoate

C53H102O5 (818.7727)

(2-hexadecanoyloxy-3-tetradecoxypropyl) (Z)-icos-11-enoate

C53H102O5 (818.7727)

(3-hexadecoxy-2-tetradecanoyloxypropyl) (Z)-icos-11-enoate

C53H102O5 (818.7727)

[2-decanoyloxy-3-[(Z)-octadec-9-enoxy]propyl] docosanoate

C53H102O5 (818.7727)

[3-nonanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] hexacosanoate

C52H98O6 (818.7363)

(3-decoxy-2-octadecanoyloxypropyl) (Z)-docos-13-enoate

C53H102O5 (818.7727)

(2-dodecanoyloxy-3-octadecoxypropyl) (Z)-icos-11-enoate

C53H102O5 (818.7727)

[3-nonanoyloxy-2-[(Z)-octadec-9-enoyl]oxypropyl] docosanoate

C52H98O6 (818.7363)

[2-decanoyloxy-3-[(Z)-docos-13-enoxy]propyl] octadecanoate

C53H102O5 (818.7727)

(3-octadecoxy-2-tetradecanoyloxypropyl) (Z)-octadec-9-enoate

C53H102O5 (818.7727)

[2-[(Z)-nonadec-9-enoyl]oxy-3-nonanoyloxypropyl] henicosanoate

C52H98O6 (818.7363)

[2-dodecanoyloxy-3-[(Z)-hexadec-9-enoxy]propyl] docosanoate

C53H102O5 (818.7727)

[2-hexadecanoyloxy-3-[(Z)-tetradec-9-enoxy]propyl] icosanoate

C53H102O5 (818.7727)

(2-decanoyloxy-3-docosoxypropyl) (Z)-octadec-9-enoate

C53H102O5 (818.7727)

[3-dodecoxy-2-[(Z)-hexadec-9-enoyl]oxypropyl] docosanoate

C53H102O5 (818.7727)

[2-[(Z)-octadec-9-enoyl]oxy-3-octanoyloxypropyl] tricosanoate

C52H98O6 (818.7363)

(3-octanoyloxy-2-undecanoyloxypropyl) (Z)-triacont-19-enoate

C52H98O6 (818.7363)

[2-dodecanoyloxy-3-[(Z)-icos-11-enoxy]propyl] octadecanoate

C53H102O5 (818.7727)

[3-nonanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] heptacosanoate

C52H98O6 (818.7363)

(2-heptadecanoyloxy-3-octanoyloxypropyl) (Z)-tetracos-13-enoate

C52H98O6 (818.7363)

(2-decanoyloxy-3-octadecoxypropyl) (Z)-docos-13-enoate

C53H102O5 (818.7727)

(2-decanoyloxy-3-icosoxypropyl) (Z)-icos-11-enoate

C53H102O5 (818.7727)

(2-hexadecanoyloxy-3-hexadecoxypropyl) (Z)-octadec-9-enoate

C53H102O5 (818.7727)

[1-[(Z)-icos-11-enoyl]oxy-3-nonanoyloxypropan-2-yl] icosanoate

C52H98O6 (818.7363)

[1-docosoxy-3-[(Z)-tetradec-9-enoyl]oxypropan-2-yl] tetradecanoate

C53H102O5 (818.7727)

[3-octanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] octacosanoate

C52H98O6 (818.7363)

(3-dodecoxy-2-octadecanoyloxypropyl) (Z)-icos-11-enoate

C53H102O5 (818.7727)

(2-tetradecanoyloxy-3-tetradecoxypropyl) (Z)-docos-13-enoate

C53H102O5 (818.7727)

(2-icosanoyloxy-3-octanoyloxypropyl) (Z)-henicos-11-enoate

C52H98O6 (818.7363)

[2-dodecanoyloxy-3-[(Z)-octadec-9-enoxy]propyl] icosanoate

C53H102O5 (818.7727)

(3-octanoyloxy-2-pentadecanoyloxypropyl) (Z)-hexacos-15-enoate

C52H98O6 (818.7363)

[3-[(Z)-octadec-9-enoxy]-2-tetradecanoyloxypropyl] octadecanoate

C53H102O5 (818.7727)

[2-[(Z)-heptadec-9-enoyl]oxy-3-octanoyloxypropyl] tetracosanoate

C52H98O6 (818.7363)

[1-[(Z)-hexadec-9-enoyl]oxy-3-octadecoxypropan-2-yl] hexadecanoate

C53H102O5 (818.7727)

[3-octanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] heptacosanoate

C52H98O6 (818.7363)

(3-icosoxy-2-tetradecanoyloxypropyl) (Z)-hexadec-9-enoate

C53H102O5 (818.7727)

[2-hexadecanoyloxy-3-[(Z)-hexadec-9-enoxy]propyl] octadecanoate

C53H102O5 (818.7727)

[2-[(Z)-hexadec-9-enoyl]oxy-3-octanoyloxypropyl] pentacosanoate

C52H98O6 (818.7363)

[3-[(Z)-docos-13-enoxy]-2-tetradecanoyloxypropyl] tetradecanoate

C53H102O5 (818.7727)

[3-hexadecoxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] icosanoate

C53H102O5 (818.7727)

(2-hexadecanoyloxy-3-nonanoyloxypropyl) (Z)-tetracos-13-enoate

C52H98O6 (818.7363)

[2-[(Z)-nonadec-9-enoyl]oxy-3-octanoyloxypropyl] docosanoate

C52H98O6 (818.7363)

[3-decoxy-2-[(Z)-octadec-9-enoyl]oxypropyl] docosanoate

C53H102O5 (818.7727)

(3-nonanoyloxy-2-octadecanoyloxypropyl) (Z)-docos-13-enoate

C52H98O6 (818.7363)

[3-icosoxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] hexadecanoate

C53H102O5 (818.7727)

[3-octanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] hexacosanoate

C52H98O6 (818.7363)

[1-[(Z)-octadec-9-enoyl]oxy-3-tetradecoxypropan-2-yl] octadecanoate

C53H102O5 (818.7727)

[2-[(Z)-icos-11-enoyl]oxy-3-octanoyloxypropyl] henicosanoate

C52H98O6 (818.7363)

(2-dodecanoyloxy-3-hexadecoxypropyl) (Z)-docos-13-enoate

C53H102O5 (818.7727)

(2-dodecanoyloxy-3-icosoxypropyl) (Z)-octadec-9-enoate

C53H102O5 (818.7727)

[2-octadecanoyloxy-3-[(Z)-tetradec-9-enoxy]propyl] octadecanoate

C53H102O5 (818.7727)

[3-dodecoxy-2-[(Z)-octadec-9-enoyl]oxypropyl] icosanoate

C53H102O5 (818.7727)

[1-decoxy-3-[(Z)-icos-11-enoyl]oxypropan-2-yl] icosanoate

C53H102O5 (818.7727)

(2-nonadecanoyloxy-3-octanoyloxypropyl) (Z)-docos-13-enoate

C52H98O6 (818.7363)

(2-dodecanoyloxy-3-nonanoyloxypropyl) (Z)-octacos-17-enoate

C52H98O6 (818.7363)

[3-nonanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] pentacosanoate

C52H98O6 (818.7363)

[2-[(Z)-hexadec-9-enoyl]oxy-3-hexadecoxypropyl] octadecanoate

C53H102O5 (818.7727)

[3-[(Z)-hexadec-9-enoxy]-2-tetradecanoyloxypropyl] icosanoate

C53H102O5 (818.7727)

(3-docosoxy-2-dodecanoyloxypropyl) (Z)-hexadec-9-enoate

C53H102O5 (818.7727)

(2-nonadecanoyloxy-3-nonanoyloxypropyl) (Z)-henicos-11-enoate

C52H98O6 (818.7363)

[3-[(Z)-docos-13-enoxy]-2-dodecanoyloxypropyl] hexadecanoate

C53H102O5 (818.7727)

(2-decanoyloxy-3-nonanoyloxypropyl) (Z)-triacont-19-enoate

C52H98O6 (818.7363)

(3-decanoyloxy-2-heptadecanoyloxypropyl) (Z)-docos-13-enoate

C52H98O6 (818.7363)

(2-heptadecanoyloxy-3-undecanoyloxypropyl) (Z)-henicos-11-enoate

C52H98O6 (818.7363)

[3-dodecanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] tetracosanoate

C52H98O6 (818.7363)

(2-tetradecanoyloxy-3-tridecanoyloxypropyl) (Z)-docos-13-enoate

C52H98O6 (818.7363)

[3-dodecanoyloxy-2-[(Z)-heptadec-9-enoyl]oxypropyl] icosanoate

C52H98O6 (818.7363)

[3-decanoyloxy-2-[(Z)-nonadec-9-enoyl]oxypropyl] icosanoate

C52H98O6 (818.7363)

[2-[(Z)-pentadec-9-enoyl]oxy-3-undecanoyloxypropyl] tricosanoate

C52H98O6 (818.7363)

(3-decanoyloxy-2-nonadecanoyloxypropyl) (Z)-icos-11-enoate

C52H98O6 (818.7363)

[2-[(Z)-heptadec-9-enoyl]oxy-3-undecanoyloxypropyl] henicosanoate

C52H98O6 (818.7363)

(3-dodecanoyloxy-2-pentadecanoyloxypropyl) (Z)-docos-13-enoate

C52H98O6 (818.7363)

[3-decanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] pentacosanoate

C52H98O6 (818.7363)

(3-decanoyloxy-2-octadecanoyloxypropyl) (Z)-henicos-11-enoate

C52H98O6 (818.7363)

[2-tetradecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] docosanoate

C52H98O6 (818.7363)

(3-dodecanoyloxy-2-hexadecanoyloxypropyl) (Z)-henicos-11-enoate

C52H98O6 (818.7363)

(2-dodecanoyloxy-3-undecanoyloxypropyl) (Z)-hexacos-15-enoate

C52H98O6 (818.7363)

(2-heptadecanoyloxy-3-tetradecanoyloxypropyl) (Z)-octadec-9-enoate

C52H98O6 (818.7363)

[2-[(Z)-tridec-9-enoyl]oxy-3-undecanoyloxypropyl] pentacosanoate

C52H98O6 (818.7363)

[2-[(Z)-tetradec-9-enoyl]oxy-3-undecanoyloxypropyl] tetracosanoate

C52H98O6 (818.7363)

[3-decanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] hexacosanoate

C52H98O6 (818.7363)

[3-decanoyloxy-2-[(Z)-octadec-9-enoyl]oxypropyl] henicosanoate

C52H98O6 (818.7363)

(2-tetradecanoyloxy-3-undecanoyloxypropyl) (Z)-tetracos-13-enoate

C52H98O6 (818.7363)

(3-decanoyloxy-2-tridecanoyloxypropyl) (Z)-hexacos-15-enoate

C52H98O6 (818.7363)

[3-dodecanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] tricosanoate

C52H98O6 (818.7363)

[3-dodecanoyloxy-2-[(Z)-hexadec-9-enoyl]oxypropyl] henicosanoate

C52H98O6 (818.7363)

(3-decanoyloxy-2-pentadecanoyloxypropyl) (Z)-tetracos-13-enoate

C52H98O6 (818.7363)

[3-tridecanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] tricosanoate

C52H98O6 (818.7363)

[3-decanoyloxy-2-[(Z)-hexadec-9-enoyl]oxypropyl] tricosanoate

C52H98O6 (818.7363)

[2-[(Z)-hexadec-9-enoyl]oxy-3-undecanoyloxypropyl] docosanoate

C52H98O6 (818.7363)

[1-[(Z)-octadec-9-enoyl]oxy-3-tridecanoyloxypropan-2-yl] octadecanoate

C52H98O6 (818.7363)

[2-pentadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] henicosanoate

C52H98O6 (818.7363)

(2-pentadecanoyloxy-3-tridecanoyloxypropyl) (Z)-henicos-11-enoate

C52H98O6 (818.7363)

[2-[(Z)-hexadec-9-enoyl]oxy-3-tridecanoyloxypropyl] icosanoate

C52H98O6 (818.7363)

[3-decanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] tetracosanoate

C52H98O6 (818.7363)

[2-[(Z)-heptadec-9-enoyl]oxy-3-tridecanoyloxypropyl] nonadecanoate

C52H98O6 (818.7363)

[3-decanoyloxy-2-[(Z)-heptadec-9-enoyl]oxypropyl] docosanoate

C52H98O6 (818.7363)

(3-decanoyloxy-2-undecanoyloxypropyl) (Z)-octacos-17-enoate

C52H98O6 (818.7363)

(2-octadecanoyloxy-3-undecanoyloxypropyl) (Z)-icos-11-enoate

C52H98O6 (818.7363)

(2-hexadecanoyloxy-3-undecanoyloxypropyl) (Z)-docos-13-enoate

C52H98O6 (818.7363)

[2-[(Z)-octadec-9-enoyl]oxy-3-undecanoyloxypropyl] icosanoate

C52H98O6 (818.7363)

[2-[(Z)-hexadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] nonadecanoate

C52H98O6 (818.7363)

[2-heptadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] nonadecanoate

C52H98O6 (818.7363)

[2-octadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] octadecanoate

C52H98O6 (818.7363)

[3-dodecanoyloxy-2-[(Z)-octadec-9-enoyl]oxypropyl] nonadecanoate

C52H98O6 (818.7363)

[2-hexadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] icosanoate

C52H98O6 (818.7363)

(3-dodecanoyloxy-2-tridecanoyloxypropyl) (Z)-tetracos-13-enoate

C52H98O6 (818.7363)

[1-[(Z)-nonadec-9-enoyl]oxy-3-undecanoyloxypropan-2-yl] nonadecanoate

C52H98O6 (818.7363)

[2-[(Z)-heptadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] octadecanoate

C52H98O6 (818.7363)

2,3-di(tetradecanoyloxy)propyl (Z)-henicos-11-enoate

C52H98O6 (818.7363)

[1-[(Z)-heptadec-9-enoyl]oxy-3-pentadecanoyloxypropan-2-yl] heptadecanoate

C52H98O6 (818.7363)

[2-[(Z)-hexadec-9-enoyl]oxy-3-pentadecanoyloxypropyl] octadecanoate

C52H98O6 (818.7363)

(2-hexadecanoyloxy-3-pentadecanoyloxypropyl) (Z)-octadec-9-enoate

C52H98O6 (818.7363)

2,3-di(hexadecanoyloxy)propyl (Z)-heptadec-9-enoate

C52H98O6 (818.7363)

[3-hexadecanoyloxy-2-[(Z)-hexadec-9-enoyl]oxypropyl] heptadecanoate

C52H98O6 (818.7363)

(1-hydroxy-3-tetracosanoyloxypropan-2-yl) (Z)-hexacos-15-enoate

C53H102O5 (818.7727)

[3-hydroxy-2-[(Z)-tetracos-13-enoyl]oxypropyl] hexacosanoate

C53H102O5 (818.7727)

[1,1,2,3,3-pentadeuterio-1,3-di(hexadecanoyloxy)propan-2-yl] (Z)-heptadec-10-enoate

C52H98O6 (818.7363)

2,3-di(pentadecanoyloxy)propyl (Z)-nonadec-9-enoate

C52H98O6 (818.7363)

[2-hexadecanoyloxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] octadecanoate

C52H98O6 (818.7363)

[2-[(Z)-pentadec-9-enoyl]oxy-3-tridecanoyloxypropyl] henicosanoate

C52H98O6 (818.7363)

[2-[(Z)-tetradec-9-enoyl]oxy-3-tridecanoyloxypropyl] docosanoate

C52H98O6 (818.7363)

[3-tetradecanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] henicosanoate

C52H98O6 (818.7363)

[2-heptadecanoyloxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] heptadecanoate

C52H98O6 (818.7363)

(2-pentadecanoyloxy-3-tetradecanoyloxypropyl) (Z)-icos-11-enoate

C52H98O6 (818.7363)

[2-hexadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] nonadecanoate

C52H98O6 (818.7363)

[3-dodecanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] docosanoate

C52H98O6 (818.7363)

(3-dodecanoyloxy-2-octadecanoyloxypropyl) (Z)-nonadec-9-enoate

C52H98O6 (818.7363)

(3-dodecanoyloxy-2-heptadecanoyloxypropyl) (Z)-icos-11-enoate

C52H98O6 (818.7363)

(2-hexadecanoyloxy-3-tetradecanoyloxypropyl) (Z)-nonadec-9-enoate

C52H98O6 (818.7363)

[3-pentadecanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] nonadecanoate

C52H98O6 (818.7363)

[2-heptadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] octadecanoate

C52H98O6 (818.7363)

(2-heptadecanoyloxy-3-tridecanoyloxypropyl) (Z)-nonadec-9-enoate

C52H98O6 (818.7363)

[2-[(Z)-pentadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] icosanoate

C52H98O6 (818.7363)

[2-pentadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] icosanoate

C52H98O6 (818.7363)

(2-hexadecanoyloxy-3-tridecanoyloxypropyl) (Z)-icos-11-enoate

C52H98O6 (818.7363)

[1-[(Z)-heptadec-7-enoyl]oxy-3-pentadecanoyloxypropan-2-yl] heptadecanoate

C52H98O6 (818.7363)

[3-dodecanoyloxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] henicosanoate

C52H98O6 (818.7363)

[2-[(Z)-heptadec-7-enoyl]oxy-3-tridecanoyloxypropyl] nonadecanoate

C52H98O6 (818.7363)

2,3-di(hexadecanoyloxy)propyl (Z)-heptadec-7-enoate

C52H98O6 (818.7363)

2,3-di(tetradecanoyloxy)propyl (Z)-henicos-9-enoate

C52H98O6 (818.7363)

[3-[(Z)-dodec-5-enoyl]oxy-2-heptadecanoyloxypropyl] icosanoate

C52H98O6 (818.7363)

[2-[(Z)-hexadec-7-enoyl]oxy-3-tetradecanoyloxypropyl] nonadecanoate

C52H98O6 (818.7363)

[3-[(Z)-dodec-5-enoyl]oxy-2-octadecanoyloxypropyl] nonadecanoate

C52H98O6 (818.7363)

(2-pentadecanoyloxy-3-tridecanoyloxypropyl) (Z)-henicos-9-enoate

C52H98O6 (818.7363)

[2-hexadecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] icosanoate

C52H98O6 (818.7363)

[2-[(Z)-hexadec-7-enoyl]oxy-3-tridecanoyloxypropyl] icosanoate

C52H98O6 (818.7363)

(2-hexadecanoyloxy-3-pentadecanoyloxypropyl) (Z)-octadec-11-enoate

C52H98O6 (818.7363)

[2-pentadecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] henicosanoate

C52H98O6 (818.7363)

(3-dodecanoyloxy-2-pentadecanoyloxypropyl) (Z)-docos-11-enoate

C52H98O6 (818.7363)

[2-[(Z)-heptadec-7-enoyl]oxy-3-tetradecanoyloxypropyl] octadecanoate

C52H98O6 (818.7363)

(2-tetradecanoyloxy-3-tridecanoyloxypropyl) (Z)-docos-11-enoate

C52H98O6 (818.7363)

[2-octadecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] octadecanoate

C52H98O6 (818.7363)

[3-[(Z)-dodec-5-enoyl]oxy-2-hexadecanoyloxypropyl] henicosanoate

C52H98O6 (818.7363)

[1-[(Z)-octadec-11-enoyl]oxy-3-tridecanoyloxypropan-2-yl] octadecanoate

C52H98O6 (818.7363)

[2-heptadecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] nonadecanoate

C52H98O6 (818.7363)

(3-dodecanoyloxy-2-hexadecanoyloxypropyl) (Z)-henicos-9-enoate

C52H98O6 (818.7363)

[3-dodecanoyloxy-2-[(Z)-octadec-11-enoyl]oxypropyl] nonadecanoate

C52H98O6 (818.7363)

[3-hexadecanoyloxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] heptadecanoate

C52H98O6 (818.7363)

[3-dodecanoyloxy-2-[(Z)-heptadec-7-enoyl]oxypropyl] icosanoate

C52H98O6 (818.7363)

[3-[(Z)-dodec-5-enoyl]oxy-2-pentadecanoyloxypropyl] docosanoate

C52H98O6 (818.7363)

[2-[(Z)-hexadec-7-enoyl]oxy-3-pentadecanoyloxypropyl] octadecanoate

C52H98O6 (818.7363)

[2-tetradecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] docosanoate

C52H98O6 (818.7363)

(2-heptadecanoyloxy-3-tetradecanoyloxypropyl) (Z)-octadec-11-enoate

C52H98O6 (818.7363)

2-[(2-Henicosanoyloxy-3-octadecoxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[Hydroxy-(3-icosoxy-2-nonadecanoyloxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[(2-Docosanoyloxy-3-heptadecoxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

[(2S)-3-hydroxy-2-tetracosanoyloxypropyl] (E)-hexacos-5-enoate

C53H102O5 (818.7727)

[(2S)-1-hydroxy-3-tetracosanoyloxypropan-2-yl] (E)-hexacos-5-enoate

C53H102O5 (818.7727)

[(2S)-3-hydroxy-2-[(E)-tetracos-15-enoyl]oxypropyl] hexacosanoate

C53H102O5 (818.7727)

[(2S)-1-hydroxy-3-[(E)-tetracos-15-enoyl]oxypropan-2-yl] hexacosanoate

C53H102O5 (818.7727)

[(2R)-2,3-di(hexadecanoyloxy)propyl] (E)-heptadec-9-enoate

C52H98O6 (818.7363)

[(2R)-3-hexadecanoyloxy-2-[(E)-hexadec-9-enoyl]oxypropyl] heptadecanoate

C52H98O6 (818.7363)

2-[(3-Hexadecoxy-2-tricosanoyloxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[Hydroxy-(2-pentacosanoyloxy-3-tetradecoxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[Hydroxy-(2-hydroxy-3-nonatriacontanoyloxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[Hydroxy-(2-octacosanoyloxy-3-undecoxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[Hydroxy-(2-pentadecanoyloxy-3-tetracosoxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[(2-Dodecanoyloxy-3-heptacosoxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[Hydroxy-(3-pentacosoxy-2-tetradecanoyloxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[(3-Henicosoxy-2-octadecanoyloxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[(2-Hexacosanoyloxy-3-tridecoxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[Hydroxy-(2-icosanoyloxy-3-nonadecoxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[Hydroxy-(3-octacosoxy-2-undecanoyloxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[Hydroxy-(3-pentadecoxy-2-tetracosanoyloxypropoxy)phosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[(2-Hexadecanoyloxy-3-tricosoxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[(3-Hexacosoxy-2-tridecanoyloxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[(3-Docosoxy-2-heptadecanoyloxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

2-[(3-Dodecoxy-2-heptacosanoyloxypropoxy)-hydroxyphosphoryl]oxyethyl-trimethylazanium

C47H97NO7P+ (818.7002)

ETIROXATE

C18H17I4NO4 (818.7337)

D006730 - Hormones, Hormone Substitutes, and Hormone Antagonists > D006728 - Hormones C147908 - Hormone Therapy Agent > C548 - Therapeutic Hormone > C1553 - Thyroid Agent D057847 - Lipid Regulating Agents > D000960 - Hypolipidemic Agents D009676 - Noxae > D000963 - Antimetabolites

1-Stearoyl-2-pentadecanoyl-3-palmitoleoyl-glycerol

C52H98O6 (818.7363)

1-pentadecanoyl-2-arachidonyl-3-myristoleoyl-glycerol

C52H98O6 (818.7363)

1-Myristoyl-2-vaccenoyl-3-stearyl-glycerol

C53H102O5 (818.7727)

triacylglycerol 49:1

C52H98O6 (818.7363)

A triglyceride in which the three acyl groups contain a total of 49 carbons and 1 double bond.

DG(50:1)

C53H102O5 (818.7727)

Provides by LipidSearch Vendor. © Copyright 2006-2024 Thermo Fisher Scientific Inc. All rights reserved

ZyE(30:0)

C57H102O2 (818.7879)

Provides by LipidSearch Vendor. © Copyright 2006-2024 Thermo Fisher Scientific Inc. All rights reserved

ChE(30:1)

C57H102O2 (818.7879)

Provides by LipidSearch Vendor. © Copyright 2006-2024 Thermo Fisher Scientific Inc. All rights reserved

NA-Orn 46:1

C51H98N2O5 (818.7475)

FAHFA 55:7;O

C55H94O4 (818.7152)

DG 14:1_36:0

C53H102O5 (818.7727)

DG 15:1_35:0

C53H102O5 (818.7727)

DG 16:1_34:0

C53H102O5 (818.7727)

DG 17:1_33:0

C53H102O5 (818.7727)

DG 18:1_32:0

C53H102O5 (818.7727)

DG 20:1_30:0

C53H102O5 (818.7727)

DG 22:1_28:0

C53H102O5 (818.7727)

DG 24:0_26:1

C53H102O5 (818.7727)

DG 24:1_26:0

C53H102O5 (818.7727)

DG 50:1

C53H102O5 (818.7727)

TG O-16:0_16:0_18:1

C53H102O5 (818.7727)

TG O-50:1

C53H102O5 (818.7727)

TG P-50:1

C53H102O5 (818.7727)

MeDAG 50:1

C53H102O5 (818.7727)

TG O-18:1_16:0_16:0

C53H102O5 (818.7727)

TG 10:0_13:0_26:1

C52H98O6 (818.7363)

TG 10:0_14:1_25:0

C52H98O6 (818.7363)

TG 10:0_15:0_24:1

C52H98O6 (818.7363)

TG 10:0_15:1_24:0

C52H98O6 (818.7363)

TG 10:0_16:1_23:0

C52H98O6 (818.7363)

TG 10:0_17:0_22:1

C52H98O6 (818.7363)

TG 10:0_17:1_22:0

C52H98O6 (818.7363)

TG 10:0_18:1_21:0

C52H98O6 (818.7363)

TG 10:0_19:0_20:1

C52H98O6 (818.7363)

TG 11:0_12:0_26:1

C52H98O6 (818.7363)

TG 11:0_14:0_24:1

C52H98O6 (818.7363)

TG 11:0_14:1_24:0

C52H98O6 (818.7363)

TG 11:0_15:1_23:0

C52H98O6 (818.7363)

TG 11:0_16:0_22:1

C52H98O6 (818.7363)

TG 11:0_16:1_22:0

C52H98O6 (818.7363)

TG 11:0_17:1_21:0

C52H98O6 (818.7363)

TG 11:0_18:0_20:1

C52H98O6 (818.7363)

TG 11:0_18:1_20:0

C52H98O6 (818.7363)

TG 12:0_13:0_24:1

C52H98O6 (818.7363)

TG 12:0_14:1_23:0

C52H98O6 (818.7363)

TG 12:0_15:0_22:1

C52H98O6 (818.7363)

TG 12:0_15:1_22:0

C52H98O6 (818.7363)

TG 12:0_16:1_21:0

C52H98O6 (818.7363)

TG 12:0_17:0_20:1

C52H98O6 (818.7363)

TG 12:0_17:1_20:0

C52H98O6 (818.7363)

TG 12:0_18:1_19:0

C52H98O6 (818.7363)

TG 13:0_14:0_22:1

C52H98O6 (818.7363)

TG 13:0_14:1_22:0

C52H98O6 (818.7363)

TG 13:0_15:1_21:0

C52H98O6 (818.7363)

TG 13:0_16:0_20:1

C52H98O6 (818.7363)

TG 13:0_16:1_20:0

C52H98O6 (818.7363)

TG 13:0_17:1_19:0

C52H98O6 (818.7363)

TG 13:0_18:0_18:1

C52H98O6 (818.7363)

TG 14:0_14:1_21:0

C52H98O6 (818.7363)

TG 14:0_15:0_20:1

C52H98O6 (818.7363)

TG 14:0_15:1_20:0

C52H98O6 (818.7363)

TG 14:0_16:0_19:1

C52H98O6 (818.7363)

TG 14:0_16:1_19:0

C52H98O6 (818.7363)

TG 14:0_17:0_18:1

C52H98O6 (818.7363)

TG 14:0_17:1_18:0

C52H98O6 (818.7363)

TG 14:1_15:0_20:0

C52H98O6 (818.7363)

TG 14:1_16:0_19:0

C52H98O6 (818.7363)

TG 14:1_17:0_18:0

C52H98O6 (818.7363)

TG 15:0_15:0_19:1

C52H98O6 (818.7363)

TG 15:0_15:1_19:0

C52H98O6 (818.7363)

TG 15:0_16:0_18:1

C52H98O6 (818.7363)

TG 15:0/16:0/18:1

C52H98O6 (818.7363)

TG 15:0_16:1_18:0

C52H98O6 (818.7363)

TG 15:0_17:0_17:1

C52H98O6 (818.7363)

TG 15:1_16:0_18:0

C52H98O6 (818.7363)

TG 15:1_17:0_17:0

C52H98O6 (818.7363)

TG 16:0_16:0_17:1

C52H98O6 (818.7363)

TG 16:0_16:1_17:0

C52H98O6 (818.7363)

TG 49:1_12:0

C52H98O6 (818.7363)

TG 49:1_14:0

C52H98O6 (818.7363)

TG 49:1_14:1

C52H98O6 (818.7363)

TG 49:1_15:0

C52H98O6 (818.7363)

TG 49:1_16:0

C52H98O6 (818.7363)

TG 49:1_16:1

C52H98O6 (818.7363)

TG 49:1_17:0

C52H98O6 (818.7363)

TG 49:1_18:0

C52H98O6 (818.7363)

TG 49:1_18:1

C52H98O6 (818.7363)

TG 49:1_20:0

C52H98O6 (818.7363)

TG 49:1_20:1

C52H98O6 (818.7363)

TG 49:1_22:1

C52H98O6 (818.7363)

(1s,3as,5ar,7s,9as,11as)-3a,6,6,9a,11a-pentamethyl-1-[(2s)-6-methyl-5-methylideneheptan-2-yl]-1h,2h,3h,4h,5h,5ah,7h,8h,9h,10h,11h-cyclopenta[a]phenanthren-7-yl hexacosanoate

C57H102O2 (818.7879)