Exact Mass: 798.6822572

Levoxyl

C15H10I4NNaO4 (798.6686579999999)

H - Systemic hormonal preparations, excl. sex hormones and insulins > H03 - Thyroid therapy > H03A - Thyroid preparations > H03AA - Thyroid hormones D006730 - Hormones, Hormone Substitutes, and Hormone Antagonists > D006728 - Hormones C147908 - Hormone Therapy Agent > C548 - Therapeutic Hormone > C1553 - Thyroid Agent

SM(d17:1/24:1(15Z))

C46H91N2O6P (798.6614396)

Sphingomyelin (d17:1/24:1(15Z)) or SM(d17:1/24:1(15Z)) is a type of sphingolipid found in animal cell membranes, especially in the membranous myelin sheath which surrounds some nerve cell axons. It usually consists of phosphorylcholine and ceramide. SM(d17:1/24:1(15Z)) consists of a heptadecasphingosine backbone and a nervonic acid chain. In humans, sphingomyelin is the only membrane phospholipid not derived from glycerol. Like all sphingolipids, SM has a ceramide core (sphingosine bonded to a fatty acid via an amide linkage). In addition, it contains one polar head group, which is either phosphocholine or phosphoethanolamine. The plasma membrane of cells is highly enriched in sphingomyelin and is considered largely to be found in the exoplasmic leaflet of the cell membrane. However, there is some evidence that there may also be a sphingomyelin pool in the inner leaflet of the membrane. Moreover, neutral sphingomyelinase-2, an enzyme that breaks down sphingomyelin into ceramide, has been found to localize exclusively to the inner leaflet further suggesting that there may be sphingomyelin present there. Sphingomyelin can accumulate in a rare hereditary disease called Niemann-Pick Disease, types A and B. Niemann-Pick disease is a genetically-inherited disease caused by a deficiency in the enzyme sphingomyelinase, which causes the accumulation of sphingomyelin in spleen, liver, lungs, bone marrow, and the brain, causing irreversible neurological damage. SMs play a role in signal transduction. Sphingomyelins are synthesized by the transfer of phosphorylcholine from phosphatidylcholine to a ceramide in a reaction catalyzed by sphingomyelin synthase. Sphingomyelin SM(d17:1/24:1(15Z)) or SM(d17:1/24:1(15Z)) is a sphingomyelin. Sphingomyelin (SM or SPH) is a type of sphingolipid found in animal cell membranes, especially in the membranous myelin sheath which surrounds some nerve cell axons. It usually consists of phosphorylcholine and ceramide. In humans, sphingomyelin is the only membrane phospholipid not derived from glycerol. Like all sphingolipids, SPH has a ceramide core (sphingosine bonded to a fatty acid via an amide linkage). In addition it contains one polar head group, which is either phosphocholine or phosphoethanolamine. The plasma membrane of cells is highly enriched in sphingomyelin and is considered largely to be found in the exoplasmic leaflet of the cell membrane. However, there is some evidence that there may also be a sphingomyelin pool in the inner leaflet of the membrane. Moreover, neutral sphingomyelinase-2 - an enzyme that breaks down sphingomyelin into ceramide has been found to localise exclusively to the inner leaflet further suggesting that there may be sphingomyelin present there. Sphingomyelin can accumulate in a rare hereditary disease called Niemann-Pick Disease, types A and B. Niemann-Pick disease is a genetically-inherited disease caused by a deficiency in the enzyme Sphingomyelinase, which causes the accumulation of Sphingomyelin in spleen, liver, lungs, bone marrow, and the brain, causing irreversible neurological damage. SMs play a role in signal transduction.

TG(14:0/14:0/20:4(5Z,8Z,11Z,14Z))

C51H90O6 (798.673704)

TG(14:0/14:0/20:4(5Z,8Z,11Z,14Z)) is a dimyristic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/14:0/20:4(5Z,8Z,11Z,14Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of arachidonic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/14:0/20:4(8Z,11Z,14Z,17Z))

C51H90O6 (798.673704)

TG(14:0/14:0/20:4(8Z,11Z,14Z,17Z)) is a dimyristic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/14:0/20:4(8Z,11Z,14Z,17Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of eicosatetraenoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/16:0/18:4(6Z,9Z,12Z,15Z))

C51H90O6 (798.673704)

TG(14:0/16:0/18:4(6Z,9Z,12Z,15Z)) is a monostearidonic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/16:0/18:4(6Z,9Z,12Z,15Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of stearidonic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/14:1(9Z)/20:3(5Z,8Z,11Z))

C51H90O6 (798.673704)

TG(14:0/14:1(9Z)/20:3(5Z,8Z,11Z)) is a monomead acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/14:1(9Z)/20:3(5Z,8Z,11Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of mead acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/14:1(9Z)/20:3n6)

C51H90O6 (798.673704)

TG(14:0/14:1(9Z)/20:3n6) is a monohomo-g-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/14:1(9Z)/20:3n6), in particular, consists of one chain of myristic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of homo-g-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/16:1(9Z)/18:3(6Z,9Z,12Z))

C51H90O6 (798.673704)

TG(14:0/16:1(9Z)/18:3(6Z,9Z,12Z)) is a monog-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/16:1(9Z)/18:3(6Z,9Z,12Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of g-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/16:1(9Z)/18:3(9Z,12Z,15Z))

C51H90O6 (798.673704)

TG(14:0/16:1(9Z)/18:3(9Z,12Z,15Z)) is a monoa-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/16:1(9Z)/18:3(9Z,12Z,15Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of a-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/20:3(5Z,8Z,11Z)/14:1(9Z))

C51H90O6 (798.673704)

TG(14:0/20:3(5Z,8Z,11Z)/14:1(9Z)) is a monomead acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/20:3(5Z,8Z,11Z)/14:1(9Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of mead acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/18:3(6Z,9Z,12Z)/16:1(9Z))

C51H90O6 (798.673704)

TG(14:0/18:3(6Z,9Z,12Z)/16:1(9Z)) is a monog-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/18:3(6Z,9Z,12Z)/16:1(9Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of g-linolenic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/20:3n6/14:1(9Z))

C51H90O6 (798.673704)

TG(14:0/20:3n6/14:1(9Z)) is a monohomo-g-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/20:3n6/14:1(9Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of homo-g-linolenic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/20:4(5Z,8Z,11Z,14Z)/14:0)

C51H90O6 (798.673704)

TG(14:0/20:4(5Z,8Z,11Z,14Z)/14:0) is a dimyristic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/20:4(5Z,8Z,11Z,14Z)/14:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of arachidonic acid at the C-2 position and one chain of myristic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/18:3(9Z,12Z,15Z)/16:1(9Z))

C51H90O6 (798.673704)

TG(14:0/18:3(9Z,12Z,15Z)/16:1(9Z)) is a monoa-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/18:3(9Z,12Z,15Z)/16:1(9Z)), in particular, consists of one chain of myristic acid at the C-1 position, one chain of a-linolenic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/18:4(6Z,9Z,12Z,15Z)/16:0)

C51H90O6 (798.673704)

TG(14:0/18:4(6Z,9Z,12Z,15Z)/16:0) is a monostearidonic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/18:4(6Z,9Z,12Z,15Z)/16:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of stearidonic acid at the C-2 position and one chain of palmitic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:0/20:4(8Z,11Z,14Z,17Z)/14:0)

C51H90O6 (798.673704)

TG(14:0/20:4(8Z,11Z,14Z,17Z)/14:0) is a dimyristic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:0/20:4(8Z,11Z,14Z,17Z)/14:0), in particular, consists of one chain of myristic acid at the C-1 position, one chain of eicosatetraenoic acid at the C-2 position and one chain of myristic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/15:0/18:4(6Z,9Z,12Z,15Z))

C51H90O6 (798.673704)

TG(15:0/15:0/18:4(6Z,9Z,12Z,15Z)) is a dipentadecanoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/15:0/18:4(6Z,9Z,12Z,15Z)), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of pentadecanoic acid at the C-2 position and one chain of stearidonic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(15:0/18:4(6Z,9Z,12Z,15Z)/15:0)

C51H90O6 (798.673704)

TG(15:0/18:4(6Z,9Z,12Z,15Z)/15:0) is a dipentadecanoic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(15:0/18:4(6Z,9Z,12Z,15Z)/15:0), in particular, consists of one chain of pentadecanoic acid at the C-1 position, one chain of stearidonic acid at the C-2 position and one chain of pentadecanoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/14:0/18:4(6Z,9Z,12Z,15Z))

C51H90O6 (798.673704)

TG(16:0/14:0/18:4(6Z,9Z,12Z,15Z)) is a monostearidonic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/14:0/18:4(6Z,9Z,12Z,15Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of stearidonic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/14:1(9Z)/18:3(6Z,9Z,12Z))

C51H90O6 (798.673704)

TG(16:0/14:1(9Z)/18:3(6Z,9Z,12Z)) is a monog-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/14:1(9Z)/18:3(6Z,9Z,12Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of g-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/14:1(9Z)/18:3(9Z,12Z,15Z))

C51H90O6 (798.673704)

TG(16:0/14:1(9Z)/18:3(9Z,12Z,15Z)) is a monoa-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/14:1(9Z)/18:3(9Z,12Z,15Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of a-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/18:3(6Z,9Z,12Z)/14:1(9Z))

C51H90O6 (798.673704)

TG(16:0/18:3(6Z,9Z,12Z)/14:1(9Z)) is a monog-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/18:3(6Z,9Z,12Z)/14:1(9Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of g-linolenic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:0/18:3(9Z,12Z,15Z)/14:1(9Z))

C51H90O6 (798.673704)

TG(16:0/18:3(9Z,12Z,15Z)/14:1(9Z)) is a monoa-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:0/18:3(9Z,12Z,15Z)/14:1(9Z)), in particular, consists of one chain of palmitic acid at the C-1 position, one chain of a-linolenic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:1(9Z)/14:0/20:3(5Z,8Z,11Z))

C51H90O6 (798.673704)

TG(14:1(9Z)/14:0/20:3(5Z,8Z,11Z)) is a monomead acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/14:0/20:3(5Z,8Z,11Z)), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of mead acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:1(9Z)/14:0/20:3n6)

C51H90O6 (798.673704)

TG(14:1(9Z)/14:0/20:3n6) is a monohomo-g-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/14:0/20:3n6), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of homo-g-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:1(9Z)/16:0/18:3(6Z,9Z,12Z))

C51H90O6 (798.673704)

TG(14:1(9Z)/16:0/18:3(6Z,9Z,12Z)) is a monog-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/16:0/18:3(6Z,9Z,12Z)), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of g-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:1(9Z)/16:0/18:3(9Z,12Z,15Z))

C51H90O6 (798.673704)

TG(14:1(9Z)/16:0/18:3(9Z,12Z,15Z)) is a monoa-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/16:0/18:3(9Z,12Z,15Z)), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of palmitic acid at the C-2 position and one chain of a-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:1(9Z)/14:1(9Z)/20:2n6)

C51H90O6 (798.673704)

TG(14:1(9Z)/14:1(9Z)/20:2n6) is a dimyristoleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/14:1(9Z)/20:2n6), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of eicosadienoic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:1(9Z)/16:1(9Z)/18:2(9Z,12Z))

C51H90O6 (798.673704)

TG(14:1(9Z)/16:1(9Z)/18:2(9Z,12Z)) is a monolinoleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/16:1(9Z)/18:2(9Z,12Z)), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of palmitoleic acid at the C-2 position and one chain of linoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:1(9Z)/18:2(9Z,12Z)/16:1(9Z))

C51H90O6 (798.673704)

TG(14:1(9Z)/18:2(9Z,12Z)/16:1(9Z)) is a monolinoleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/18:2(9Z,12Z)/16:1(9Z)), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of linoleic acid at the C-2 position and one chain of palmitoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(14:1(9Z)/20:2n6/14:1(9Z))

C51H90O6 (798.673704)

TG(14:1(9Z)/20:2n6/14:1(9Z)) is a dimyristoleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(14:1(9Z)/20:2n6/14:1(9Z)), in particular, consists of one chain of myristoleic acid at the C-1 position, one chain of eicosadienoic acid at the C-2 position and one chain of myristoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:1(9Z)/14:0/18:3(6Z,9Z,12Z))

C51H90O6 (798.673704)

TG(16:1(9Z)/14:0/18:3(6Z,9Z,12Z)) is a monog-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:1(9Z)/14:0/18:3(6Z,9Z,12Z)), in particular, consists of one chain of palmitoleic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of g-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:1(9Z)/14:0/18:3(9Z,12Z,15Z))

C51H90O6 (798.673704)

TG(16:1(9Z)/14:0/18:3(9Z,12Z,15Z)) is a monoa-linolenic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:1(9Z)/14:0/18:3(9Z,12Z,15Z)), in particular, consists of one chain of palmitoleic acid at the C-1 position, one chain of myristic acid at the C-2 position and one chain of a-linolenic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

TG(16:1(9Z)/14:1(9Z)/18:2(9Z,12Z))

C51H90O6 (798.673704)

TG(16:1(9Z)/14:1(9Z)/18:2(9Z,12Z)) is a monolinoleic acid triglyceride. Triglycerides (TGs or TAGs) are also known as triacylglycerols or triacylglycerides, meaning that they are glycerides in which the glycerol is esterified with three fatty acid groups (i.e. fatty acid trimesters of glycerol). TGs may be divided into three general types with respect to their acyl substituents. They are simple or monoacid if they contain only one type of fatty acid, diacid if they contain two types of fatty acids and triacid if three different acyl groups. Chain lengths of the fatty acids in naturally occurring triglycerides can be of varying lengths and saturations but 16, 18 and 20 carbons are the most common. TG(16:1(9Z)/14:1(9Z)/18:2(9Z,12Z)), in particular, consists of one chain of palmitoleic acid at the C-1 position, one chain of myristoleic acid at the C-2 position and one chain of linoleic acid at the C-3 position. TGs are the main constituent of vegetable oil and animal fats. TGs are major components of very low density lipoprotein (VLDL) and chylomicrons, play an important role in metabolism as energy sources and transporters of dietary fat. They contain more than twice the energy (9 kcal/g) of carbohydrates and proteins. In the intestine, triglycerides are split into glycerol and fatty acids (this process is called lipolysis) with the help of lipases and bile secretions, which can then move into blood vessels. The triglycerides are rebuilt in the blood from their fragments and become constituents of lipoproteins, which deliver the fatty acids to and from fat cells among other functions. Various tissues can release the free fatty acids and take them up as a source of energy. Fat cells can synthesize and store triglycerides. When the body requires fatty acids as an energy source, the hormone glucagon signals the breakdown of the triglycerides by hormone-sensitive lipase to release free fatty acids. As the brain cannot utilize fatty acids as an energy source, the glycerol component of triglycerides can be converted into glucose for brain fuel when it is broken down. (www.cyberlipid.org, www.wikipedia.org)
TAGs can serve as fatty acid stores in all cells, but primarily in adipocytes of adipose tissue. The major building block for the synthesis of triacylglycerides, in non-adipose tissue, is glycerol. Adipocytes lack glycerol kinase and so must use another route to TAG synthesis. Specifically, dihydroxyacetone phosphate (DHAP), which is produced during glycolysis, is the precursor for TAG synthesis in adipose tissue. DHAP can also serve as a TAG precursor in non-adipose tissues, but does so to a much lesser extent than glycerol. The use of DHAP for the TAG backbone depends on whether the synthesis of the TAGs occurs in the mitochondria and ER or the ER and the peroxisomes. The ER/mitochondria pathway requires the action of glycerol-3-phosphate dehydrogenase to convert DHAP to glycerol-3-phosphate. Glycerol-3-phosphate acyltransferase then esterifies a fatty acid to glycerol-3-phosphate thereby generating lysophosphatidic acid. The ER/peroxisome reaction pathway uses the peroxisomal enzyme DHAP acyltransferase to acylate DHAP to acyl-DHAP which is then reduced by acyl-DHAP reductase. The fatty acids that are incorporated into TAGs are activated to acyl-CoAs through the action of acyl-CoA synthetases. Two molecules of acyl-CoA are esterified to glycerol-3-phosphate to yield 1,2-diacylglycerol phosphate (also known as phosphatidic acid). The phosphate is then removed by phosphatidic acid phosphatase (PAP1), to generate 1,2-diacylglycerol. This diacylglycerol serves as the substrate for addition of the third fatty acid to make TAG. Intestinal monoacylglycerols, derived from dietary fats, can also serve as substrates for the synthesis of 1,2-diacylglycerols.

SM(d18:1/23:1(9Z))

C46H91N2O6P (798.6614396)

Sphingomyelin (d18:1/23:1(9Z)) or SM(d18:1/23:1(9Z)) is a type of sphingolipid found in animal cell membranes, especially in the membranous myelin sheath which surrounds some nerve cell axons. It usually consists of phosphorylcholine and ceramide. SM(d18:1/23:1(9Z)) consists of a sphingosine backbone and a 14Z-tricosenoic acid chain. In humans, sphingomyelin is the only membrane phospholipid not derived from glycerol. Like all sphingolipids, SM has a ceramide core (sphingosine bonded to a fatty acid via an amide linkage). In addition, it contains one polar head group, which is either phosphocholine or phosphoethanolamine. The plasma membrane of cells is highly enriched in sphingomyelin and is considered largely to be found in the exoplasmic leaflet of the cell membrane. However, there is some evidence that there may also be a sphingomyelin pool in the inner leaflet of the membrane. Moreover, neutral sphingomyelinase-2, an enzyme that breaks down sphingomyelin into ceramide, has been found to localize exclusively to the inner leaflet further suggesting that there may be sphingomyelin present there. Sphingomyelin can accumulate in a rare hereditary disease called Niemann-Pick Disease, types A and B. Niemann-Pick disease is a genetically-inherited disease caused by a deficiency in the enzyme sphingomyelinase, which causes the accumulation of sphingomyelin in spleen, liver, lungs, bone marrow, and the brain, causing irreversible neurological damage. SMs play a role in signal transduction. Sphingomyelins are synthesized by the transfer of phosphorylcholine from phosphatidylcholine to a ceramide in a reaction catalyzed by sphingomyelin synthase.

SM(d18:2(4E,14Z)/23:0)

C46H91N2O6P (798.6614396)

Sphingomyelin (d18:2(4E,14Z)/23:0) or SM(d18:2(4E,14Z)/23:0) is a type of sphingolipid found in animal cell membranes, especially in the membranous myelin sheath which surrounds some nerve cell axons. It usually consists of phosphorylcholine and ceramide. SM(d18:2(4E,14Z)/23:0) consists of a sphinga-4E,14Z-dienine backbone and a tricosylic acid chain. In humans, sphingomyelin is the only membrane phospholipid not derived from glycerol. Like all sphingolipids, SM has a ceramide core (sphingosine bonded to a fatty acid via an amide linkage). In addition, it contains one polar head group, which is either phosphocholine or phosphoethanolamine. The plasma membrane of cells is highly enriched in sphingomyelin and is considered largely to be found in the exoplasmic leaflet of the cell membrane. However, there is some evidence that there may also be a sphingomyelin pool in the inner leaflet of the membrane. Moreover, neutral sphingomyelinase-2, an enzyme that breaks down sphingomyelin into ceramide, has been found to localize exclusively to the inner leaflet further suggesting that there may be sphingomyelin present there. Sphingomyelin can accumulate in a rare hereditary disease called Niemann-Pick Disease, types A and B. Niemann-Pick disease is a genetically-inherited disease caused by a deficiency in the enzyme sphingomyelinase, which causes the accumulation of sphingomyelin in spleen, liver, lungs, bone marrow, and the brain, causing irreversible neurological damage. SMs play a role in signal transduction. Sphingomyelins are synthesized by the transfer of phosphorylcholine from phosphatidylcholine to a ceramide in a reaction catalyzed by sphingomyelin synthase.

1-Tetradecanoyl-2-(9Z-tetradecenoyl)-3-(8Z,11Z,14Z-eicosatrienoyl)-sn-glycerol

C51H90O6 (798.673704)

Triacylglycerol 12:0-18:2-18:2

C51H90O6 (798.673704)

Triacylglycerol 14:0-16:1-18:3

C51H90O6 (798.673704)

Sphingomyelin d18:1-C23:1

C46H91N2O6P (798.6614396)

Sphingomyelin d18:2-C23:0

C46H91N2O6P (798.6614396)

SM(d41:2)

C46H91N2O6P (798.6614396)

Found in mouse kidney; TwoDicalId=1148; MgfFile=160827_Kidney_normal_Neg_03; MgfId=1877 Found in mouse spleen; TwoDicalId=2127; MgfFile=160729_spleen_normal_02_Neg; MgfId=1383

3-O-[6-O-palmitoyl-beta-D-glucosyl]-alpha-spinasterol

C51H90O6 (798.673704)

TG(14:0/14:1(9Z)/20:3(8Z,11Z,14Z))[iso6]

C51H90O6 (798.673704)

TG(14:0/16:0/18:4(6Z,9Z,12Z,15Z))[iso6]

C51H90O6 (798.673704)

TG(14:0/16:1(9Z)/18:3(6Z,9Z,12Z))[iso6]

C51H90O6 (798.673704)

TG(14:0/16:1(9Z)/18:3(9Z,12Z,15Z))[iso6]

C51H90O6 (798.673704)

TG(14:1(9Z)/16:0/18:3(6Z,9Z,12Z))[iso6]

C51H90O6 (798.673704)

TG(14:1(9Z)/16:0/18:3(9Z,12Z,15Z))[iso6]

C51H90O6 (798.673704)

TG(14:1(9Z)/16:1(9Z)/18:2(9Z,12Z))[iso6]

C51H90O6 (798.673704)

TG(14:1(9Z)/17:1(9Z)/17:2(9Z,12Z))[iso6]

C51H90O6 (798.673704)

TG(15:0/15:1(9Z)/18:3(6Z,9Z,12Z))[iso6]

C51H90O6 (798.673704)

TG(15:0/15:1(9Z)/18:3(9Z,12Z,15Z))[iso6]

C51H90O6 (798.673704)

TG(15:1(9Z)/16:1(9Z)/17:2(9Z,12Z))[iso6]

C51H90O6 (798.673704)

SM(d17:1/24:1(15Z))

C46H91N2O6P (798.6614396)

TG(12:0/18:2(9Z,12Z)/18:2(9Z,12Z))[iso3]

C51H90O6 (798.673704)

TG(13:0/13:0/22:4(7Z,10Z,13Z,16Z))[iso3]

C51H90O6 (798.673704)

TG(14:0/14:0/20:4(5Z,8Z,11Z,14Z))[iso3]

C51H90O6 (798.673704)

TG(14:0/17:2(9Z,12Z)/17:2(9Z,12Z))[iso3]

C51H90O6 (798.673704)

TG(14:1(9Z)/14:1(9Z)/20:2(11Z,14Z))[iso3]

C51H90O6 (798.673704)

TG(15:0/15:0/18:4(6Z,9Z,12Z,15Z))[iso3]

C51H90O6 (798.673704)

TG(15:1(9Z)/15:1(9Z)/18:2(9Z,12Z))[iso3]

C51H90O6 (798.673704)

TG(12:0/14:0/22:4(7Z,10Z,13Z,16Z))[iso6]

C51H90O6 (798.673704)

TG(12:0/14:1(9Z)/22:3(10Z,13Z,16Z))[iso6]

C51H90O6 (798.673704)

TG(12:0/16:0/20:4(5Z,8Z,11Z,14Z))[iso6]

C51H90O6 (798.673704)

TG(12:0/16:1(9Z)/20:3(8Z,11Z,14Z))[iso6]

C51H90O6 (798.673704)

TG(12:0/18:0/18:4(6Z,9Z,12Z,15Z))[iso6]

C51H90O6 (798.673704)

TG(12:0/18:1(9Z)/18:3(6Z,9Z,12Z))[iso6]

C51H90O6 (798.673704)

TG(12:0/18:1(9Z)/18:3(9Z,12Z,15Z))[iso6]

C51H90O6 (798.673704)

TG(13:0/15:0/20:4(5Z,8Z,11Z,14Z))[iso6]

C51H90O6 (798.673704)

TG(13:0/15:1(9Z)/20:3(8Z,11Z,14Z))[iso6]

C51H90O6 (798.673704)

TG(13:0/17:0/18:4(6Z,9Z,12Z,15Z))[iso6]

C51H90O6 (798.673704)

TG(13:0/17:1(9Z)/18:3(6Z,9Z,12Z))[iso6]

C51H90O6 (798.673704)

TG(13:0/17:1(9Z)/18:3(9Z,12Z,15Z))[iso6]

C51H90O6 (798.673704)

TG(13:0/17:2(9Z,12Z)/18:2(9Z,12Z))[iso6]

C51H90O6 (798.673704)

TG 48:4

C51H90O6 (798.673704)

SM 41:2;O2

C46H91N2O6P (798.6614396)

Dicholesteryl Carbonate

C55H90O3 (798.688959)

dl-thyroxine sodium salt

C15H10I4NNaO4 (798.6686579999999)

dicamba-potassium

C15H10I4NNaO4 (798.6686579999999)

D-THYROXINE SODIUM SALT

C15H10I4NNaO4 (798.6686579999999)

Sodium thyroxinate

C15H10I4NNaO4 (798.6686579999999)

[(E)-3-hydroxy-2-[[(Z)-tricos-11-enoyl]amino]octadec-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate

C46H91N2O6P (798.6614396)

[(4E,8E)-3-hydroxy-2-(tricosanoylamino)octadeca-4,8-dienyl] 2-(trimethylazaniumyl)ethyl phosphate

C46H91N2O6P (798.6614396)

1-Myristoleoyl-2-myristoyl-3-homo-g-linolenoyl-glycerol

C51H90O6 (798.673704)

[(E,2S,3R)-3-hydroxy-2-[[(Z)-tricos-9-enoyl]amino]octadec-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate

C46H91N2O6P (798.6614396)

[3-tetradecanoyloxy-2-[(E)-tetradec-9-enoyl]oxypropyl] (8E,11E,14E)-icosa-8,11,14-trienoate

C51H90O6 (798.673704)

CID 154573182

C46H91N2O6P (798.6614396)

NAOrn 25:0/20:4

C50H90N2O5 (798.684937)

NAOrn 24:3/21:1

C50H90N2O5 (798.684937)

NAOrn 26:4/19:0

C50H90N2O5 (798.684937)

NAOrn 26:2/19:2

C50H90N2O5 (798.684937)

NAOrn 19:0/26:4

C50H90N2O5 (798.684937)

NAOrn 19:2/26:2

C50H90N2O5 (798.684937)

NAOrn 22:4/23:0

C50H90N2O5 (798.684937)

NAOrn 24:2/21:2

C50H90N2O5 (798.684937)

NAOrn 24:4/21:0

C50H90N2O5 (798.684937)

NAOrn 20:4/25:0

C50H90N2O5 (798.684937)

NAOrn 21:2/24:2

C50H90N2O5 (798.684937)

NAOrn 26:3/19:1

C50H90N2O5 (798.684937)

NAOrn 21:0/24:4

C50H90N2O5 (798.684937)

NAOrn 23:0/22:4

C50H90N2O5 (798.684937)

PE-Cer 20:1;2O/24:1

C46H91N2O6P (798.6614396)

PE-Cer 21:2;2O/23:0

C46H91N2O6P (798.6614396)

PE-Cer 22:1;2O/22:1

C46H91N2O6P (798.6614396)

PE-Cer 19:2;2O/25:0

C46H91N2O6P (798.6614396)

PE-Cer 23:0;2O/21:2

C46H91N2O6P (798.6614396)

PE-Cer 23:2;2O/21:0

C46H91N2O6P (798.6614396)

PE-Cer 22:2;2O/22:0

C46H91N2O6P (798.6614396)

PE-Cer 22:0;2O/22:2

C46H91N2O6P (798.6614396)

PE-Cer 26:2;2O/18:0

C46H91N2O6P (798.6614396)

PE-Cer 18:2;2O/26:0

C46H91N2O6P (798.6614396)

PE-Cer 25:0;2O/19:2

C46H91N2O6P (798.6614396)

PE-Cer 23:1;2O/21:1

C46H91N2O6P (798.6614396)

PE-Cer 24:1;2O/20:1

C46H91N2O6P (798.6614396)

PE-Cer 26:1;2O/18:1

C46H91N2O6P (798.6614396)

PE-Cer 20:2;2O/24:0

C46H91N2O6P (798.6614396)

PE-Cer 24:0;2O/20:2

C46H91N2O6P (798.6614396)

PE-Cer 18:1;2O/26:1

C46H91N2O6P (798.6614396)

PE-Cer 20:0;2O/24:2

C46H91N2O6P (798.6614396)

PE-Cer 25:2;2O/19:0

C46H91N2O6P (798.6614396)

PE-Cer 18:0;2O/26:2

C46H91N2O6P (798.6614396)

PE-Cer 25:1;2O/19:1

C46H91N2O6P (798.6614396)

PE-Cer 24:2;2O/20:0

C46H91N2O6P (798.6614396)

PE-Cer 26:0;2O/18:2

C46H91N2O6P (798.6614396)

[(E)-2-[[(Z)-dotriacont-21-enoyl]amino]-3-hydroxynon-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate

C46H91N2O6P (798.6614396)

[(4E,8E)-2-(heptanoylamino)-3-hydroxytetratriaconta-4,8-dienyl] 2-(trimethylazaniumyl)ethyl phosphate

C46H91N2O6P (798.6614396)

[(4E,8E)-3-hydroxy-2-(pentanoylamino)hexatriaconta-4,8-dienyl] 2-(trimethylazaniumyl)ethyl phosphate

C46H91N2O6P (798.6614396)

[(4E,8E)-2-(hexanoylamino)-3-hydroxypentatriaconta-4,8-dienyl] 2-(trimethylazaniumyl)ethyl phosphate

C46H91N2O6P (798.6614396)

[(4E,8E)-2-(butanoylamino)-3-hydroxyheptatriaconta-4,8-dienyl] 2-(trimethylazaniumyl)ethyl phosphate

C46H91N2O6P (798.6614396)

[(4E,8E)-3-hydroxy-2-(nonanoylamino)dotriaconta-4,8-dienyl] 2-(trimethylazaniumyl)ethyl phosphate

C46H91N2O6P (798.6614396)

[(4E,8E)-3-hydroxy-2-(octanoylamino)tritriaconta-4,8-dienyl] 2-(trimethylazaniumyl)ethyl phosphate

C46H91N2O6P (798.6614396)

[(4E,8E)-2-(decanoylamino)-3-hydroxyhentriaconta-4,8-dienyl] 2-(trimethylazaniumyl)ethyl phosphate

C46H91N2O6P (798.6614396)

[(E)-3-hydroxy-2-[[(Z)-tetracos-13-enoyl]amino]heptadec-4-enyl] 2-(trimethylazaniumyl)ethyl phosphate

C46H91N2O6P (798.6614396)

[(4E,8E)-3-hydroxy-2-(nonacosanoylamino)dodeca-4,8-dienyl] 2-(trimethylazaniumyl)ethyl phosphate

C46H91N2O6P (798.6614396)

[2-[[(9Z,12Z)-hexadeca-9,12-dienoyl]amino]-3-hydroxypentacosyl] 2-(trimethylazaniumyl)ethyl phosphate

C46H91N2O6P (798.6614396)

2,3-di(octanoyloxy)propyl (20Z,23Z,26Z,29Z)-dotriaconta-20,23,26,29-tetraenoate

C51H90O6 (798.673704)

2,3-di(nonanoyloxy)propyl (18Z,21Z,24Z,27Z)-triaconta-18,21,24,27-tetraenoate

C51H90O6 (798.673704)

(2-octadecanoyloxy-3-octanoyloxypropyl) (10Z,13Z,16Z,19Z)-docosa-10,13,16,19-tetraenoate

C51H90O6 (798.673704)

(2-heptadecanoyloxy-3-nonanoyloxypropyl) (10Z,13Z,16Z,19Z)-docosa-10,13,16,19-tetraenoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-nonadeca-9,12-dienoyl]oxy-3-octanoyloxypropyl] (11Z,14Z)-henicosa-11,14-dienoate

C51H90O6 (798.673704)

(2-decanoyloxy-3-octanoyloxypropyl) (18Z,21Z,24Z,27Z)-triaconta-18,21,24,27-tetraenoate

C51H90O6 (798.673704)

[2-[(6Z,9Z,12Z,15Z)-octadeca-6,9,12,15-tetraenoyl]oxy-3-octanoyloxypropyl] docosanoate

C51H90O6 (798.673704)

[3-nonanoyloxy-2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxypropyl] (11Z,14Z)-henicosa-11,14-dienoate

C51H90O6 (798.673704)

[3-nonanoyloxy-2-[(9Z,12Z,15Z)-octadeca-9,12,15-trienoyl]oxypropyl] (Z)-henicos-11-enoate

C51H90O6 (798.673704)

[3-octanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (12Z,15Z,18Z)-hexacosa-12,15,18-trienoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxy-3-octanoyloxypropyl] (13Z,16Z)-docosa-13,16-dienoate

C51H90O6 (798.673704)

[2-[(Z)-octadec-9-enoyl]oxy-3-octanoyloxypropyl] (10Z,13Z,16Z)-docosa-10,13,16-trienoate

C51H90O6 (798.673704)

[2-[(4Z,7Z,10Z,13Z)-hexadeca-4,7,10,13-tetraenoyl]oxy-3-nonanoyloxypropyl] tricosanoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-nonanoyloxypropyl] (13Z,16Z)-docosa-13,16-dienoate

C51H90O6 (798.673704)

(3-nonanoyloxy-2-pentadecanoyloxypropyl) (12Z,15Z,18Z,21Z)-tetracosa-12,15,18,21-tetraenoate

C51H90O6 (798.673704)

[3-nonanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] (10Z,13Z,16Z)-tetracosa-10,13,16-trienoate

C51H90O6 (798.673704)

[2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxy-3-octanoyloxypropyl] (Z)-tetracos-13-enoate

C51H90O6 (798.673704)

[1-[(8Z,11Z,14Z,17Z)-icosa-8,11,14,17-tetraenoyl]oxy-3-octanoyloxypropan-2-yl] icosanoate

C51H90O6 (798.673704)

(2-nonadecanoyloxy-3-nonanoyloxypropyl) (8Z,11Z,14Z,17Z)-icosa-8,11,14,17-tetraenoate

C51H90O6 (798.673704)

(2-hexadecanoyloxy-3-octanoyloxypropyl) (12Z,15Z,18Z,21Z)-tetracosa-12,15,18,21-tetraenoate

C51H90O6 (798.673704)

[1-[(11Z,14Z,17Z)-icosa-11,14,17-trienoyl]oxy-3-octanoyloxypropan-2-yl] (Z)-icos-11-enoate

C51H90O6 (798.673704)

(3-nonanoyloxy-2-undecanoyloxypropyl) (16Z,19Z,22Z,25Z)-octacosa-16,19,22,25-tetraenoate

C51H90O6 (798.673704)

[2-[(9Z,12Z,15Z)-octadeca-9,12,15-trienoyl]oxy-3-octanoyloxypropyl] (Z)-docos-13-enoate

C51H90O6 (798.673704)

[2-[(Z)-heptadec-9-enoyl]oxy-3-nonanoyloxypropyl] (10Z,13Z,16Z)-docosa-10,13,16-trienoate

C51H90O6 (798.673704)

(2-dodecanoyloxy-3-octanoyloxypropyl) (16Z,19Z,22Z,25Z)-octacosa-16,19,22,25-tetraenoate

C51H90O6 (798.673704)

[3-nonanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] (12Z,15Z,18Z)-hexacosa-12,15,18-trienoate

C51H90O6 (798.673704)

[2-[(Z)-hexadec-9-enoyl]oxy-3-octanoyloxypropyl] (10Z,13Z,16Z)-tetracosa-10,13,16-trienoate

C51H90O6 (798.673704)

(3-nonanoyloxy-2-tridecanoyloxypropyl) (14Z,17Z,20Z,23Z)-hexacosa-14,17,20,23-tetraenoate

C51H90O6 (798.673704)

[2-[(4Z,7Z,10Z,13Z)-hexadeca-4,7,10,13-tetraenoyl]oxy-3-octanoyloxypropyl] tetracosanoate

C51H90O6 (798.673704)

[2-[(11Z,14Z)-icosa-11,14-dienoyl]oxy-3-octanoyloxypropyl] (11Z,14Z)-icosa-11,14-dienoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-octanoyloxypropyl] (13Z,16Z)-tetracosa-13,16-dienoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-nonadeca-9,12-dienoyl]oxy-3-nonanoyloxypropyl] (11Z,14Z)-icosa-11,14-dienoate

C51H90O6 (798.673704)

(3-octanoyloxy-2-tetradecanoyloxypropyl) (14Z,17Z,20Z,23Z)-hexacosa-14,17,20,23-tetraenoate

C51H90O6 (798.673704)

[3-nonanoyloxy-2-[(6Z,9Z,12Z,15Z)-octadeca-6,9,12,15-tetraenoyl]oxypropyl] henicosanoate

C51H90O6 (798.673704)

[2-[(Z)-nonadec-9-enoyl]oxy-3-nonanoyloxypropyl] (11Z,14Z,17Z)-icosa-11,14,17-trienoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxypropyl] (9Z,12Z)-octadeca-9,12-dienoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-tridecanoyloxypropyl] (9Z,12Z)-octadeca-9,12-dienoate

C51H90O6 (798.673704)

[3-tridecanoyloxy-2-[(Z)-tridec-9-enoyl]oxypropyl] (10Z,13Z,16Z)-docosa-10,13,16-trienoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(Z)-hexadec-9-enoyl]oxypropyl] (11Z,14Z,17Z)-icosa-11,14,17-trienoate

C51H90O6 (798.673704)

[2-pentadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (11Z,14Z,17Z)-icosa-11,14,17-trienoate

C51H90O6 (798.673704)

[3-decanoyloxy-2-[(6Z,9Z,12Z,15Z)-octadeca-6,9,12,15-tetraenoyl]oxypropyl] icosanoate

C51H90O6 (798.673704)

[3-tetradecanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (11Z,14Z,17Z)-icosa-11,14,17-trienoate

C51H90O6 (798.673704)

[3-decanoyloxy-2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxypropyl] (13Z,16Z)-docosa-13,16-dienoate

C51H90O6 (798.673704)

[2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxy-3-tetradecanoyloxypropyl] (Z)-octadec-9-enoate

C51H90O6 (798.673704)

[1-dodecanoyloxy-3-[(6Z,9Z,12Z,15Z)-octadeca-6,9,12,15-tetraenoyl]oxypropan-2-yl] octadecanoate

C51H90O6 (798.673704)

2,3-di(decanoyloxy)propyl (16Z,19Z,22Z,25Z)-octacosa-16,19,22,25-tetraenoate

C51H90O6 (798.673704)

2,3-di(tetradecanoyloxy)propyl (8Z,11Z,14Z,17Z)-icosa-8,11,14,17-tetraenoate

C51H90O6 (798.673704)

[3-decanoyloxy-2-[(4Z,7Z,10Z,13Z)-hexadeca-4,7,10,13-tetraenoyl]oxypropyl] docosanoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-tridecanoyloxypropyl] (9Z,12Z)-nonadeca-9,12-dienoate

C51H90O6 (798.673704)

[2-[(Z)-pentadec-9-enoyl]oxy-3-tridecanoyloxypropyl] (11Z,14Z,17Z)-icosa-11,14,17-trienoate

C51H90O6 (798.673704)

[2-[(Z)-heptadec-9-enoyl]oxy-3-undecanoyloxypropyl] (11Z,14Z,17Z)-icosa-11,14,17-trienoate

C51H90O6 (798.673704)

[2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] nonadecanoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-undecanoyloxypropyl] (11Z,14Z)-icosa-11,14-dienoate

C51H90O6 (798.673704)

2,3-bis[[(Z)-tridec-9-enoyl]oxy]propyl (13Z,16Z)-docosa-13,16-dienoate

C51H90O6 (798.673704)

[2-[(4Z,7Z,10Z,13Z)-hexadeca-4,7,10,13-tetraenoyl]oxy-3-tetradecanoyloxypropyl] octadecanoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-tetradecanoyloxypropyl] (9Z,12Z)-octadeca-9,12-dienoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-undecanoyloxypropyl] (11Z,14Z)-henicosa-11,14-dienoate

C51H90O6 (798.673704)

[3-decanoyloxy-2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxypropyl] (Z)-docos-13-enoate

C51H90O6 (798.673704)

[2-[(4Z,7Z,10Z,13Z)-hexadeca-4,7,10,13-tetraenoyl]oxy-3-undecanoyloxypropyl] henicosanoate

C51H90O6 (798.673704)

[2-[(Z)-heptadec-9-enoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (9Z,12Z)-octadeca-9,12-dienoate

C51H90O6 (798.673704)

[3-decanoyloxy-2-[(9Z,12Z)-nonadeca-9,12-dienoyl]oxypropyl] (9Z,12Z)-nonadeca-9,12-dienoate

C51H90O6 (798.673704)

(3-decanoyloxy-2-tetradecanoyloxypropyl) (12Z,15Z,18Z,21Z)-tetracosa-12,15,18,21-tetraenoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-octadec-9-enoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxypropyl] (Z)-icos-11-enoate

C51H90O6 (798.673704)

(2-tridecanoyloxy-3-undecanoyloxypropyl) (12Z,15Z,18Z,21Z)-tetracosa-12,15,18,21-tetraenoate

C51H90O6 (798.673704)

(3-decanoyloxy-2-dodecanoyloxypropyl) (14Z,17Z,20Z,23Z)-hexacosa-14,17,20,23-tetraenoate

C51H90O6 (798.673704)

(3-decanoyloxy-2-hexadecanoyloxypropyl) (10Z,13Z,16Z,19Z)-docosa-10,13,16,19-tetraenoate

C51H90O6 (798.673704)

[2-[(Z)-tridec-9-enoyl]oxy-3-undecanoyloxypropyl] (10Z,13Z,16Z)-tetracosa-10,13,16-trienoate

C51H90O6 (798.673704)

[2-[(6Z,9Z,12Z,15Z)-octadeca-6,9,12,15-tetraenoyl]oxy-3-undecanoyloxypropyl] nonadecanoate

C51H90O6 (798.673704)

(3-dodecanoyloxy-2-hexadecanoyloxypropyl) (8Z,11Z,14Z,17Z)-icosa-8,11,14,17-tetraenoate

C51H90O6 (798.673704)

(2-heptadecanoyloxy-3-undecanoyloxypropyl) (8Z,11Z,14Z,17Z)-icosa-8,11,14,17-tetraenoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxypropyl] (11Z,14Z)-icosa-11,14-dienoate

C51H90O6 (798.673704)

[2-[(Z)-tetradec-9-enoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (11Z,14Z)-henicosa-11,14-dienoate

C51H90O6 (798.673704)

[1-dodecanoyloxy-3-[(9Z,12Z,15Z)-octadeca-9,12,15-trienoyl]oxypropan-2-yl] (Z)-octadec-9-enoate

C51H90O6 (798.673704)

[2-[(Z)-heptadec-9-enoyl]oxy-3-tridecanoyloxypropyl] (9Z,12Z,15Z)-octadeca-9,12,15-trienoate

C51H90O6 (798.673704)

[2-[(9Z,12Z,15Z)-octadeca-9,12,15-trienoyl]oxy-3-undecanoyloxypropyl] (Z)-nonadec-9-enoate

C51H90O6 (798.673704)

(2-pentadecanoyloxy-3-tridecanoyloxypropyl) (8Z,11Z,14Z,17Z)-icosa-8,11,14,17-tetraenoate

C51H90O6 (798.673704)

[2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxy-3-tridecanoyloxypropyl] (Z)-nonadec-9-enoate

C51H90O6 (798.673704)

(2-hexadecanoyloxy-3-tetradecanoyloxypropyl) (6Z,9Z,12Z,15Z)-octadeca-6,9,12,15-tetraenoate

C51H90O6 (798.673704)

(3-decanoyloxy-2-octadecanoyloxypropyl) (8Z,11Z,14Z,17Z)-icosa-8,11,14,17-tetraenoate

C51H90O6 (798.673704)

[2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] octadecanoate

C51H90O6 (798.673704)

[2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxy-3-undecanoyloxypropyl] (Z)-henicos-11-enoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (10Z,13Z,16Z)-docosa-10,13,16-trienoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxypropyl] (9Z,12Z)-nonadeca-9,12-dienoate

C51H90O6 (798.673704)

[1-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropan-2-yl] (Z)-heptadec-9-enoate

C51H90O6 (798.673704)

[3-decanoyloxy-2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxypropyl] (11Z,14Z)-icosa-11,14-dienoate

C51H90O6 (798.673704)

2,3-di(undecanoyloxy)propyl (14Z,17Z,20Z,23Z)-hexacosa-14,17,20,23-tetraenoate

C51H90O6 (798.673704)

[2-heptadecanoyloxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (9Z,12Z,15Z)-octadeca-9,12,15-trienoate

C51H90O6 (798.673704)

[2-[(4Z,7Z,10Z,13Z)-hexadeca-4,7,10,13-tetraenoyl]oxy-3-tridecanoyloxypropyl] nonadecanoate

C51H90O6 (798.673704)

[3-decanoyloxy-2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxypropyl] (11Z,14Z)-henicosa-11,14-dienoate

C51H90O6 (798.673704)

[3-decanoyloxy-2-[(9Z,12Z,15Z)-octadeca-9,12,15-trienoyl]oxypropyl] (Z)-icos-11-enoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-octadeca-9,12-dienoyl]oxy-3-undecanoyloxypropyl] (9Z,12Z)-nonadeca-9,12-dienoate

C51H90O6 (798.673704)

[3-decanoyloxy-2-[(Z)-octadec-9-enoyl]oxypropyl] (11Z,14Z,17Z)-icosa-11,14,17-trienoate

C51H90O6 (798.673704)

[3-decanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (10Z,13Z,16Z)-tetracosa-10,13,16-trienoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (Z)-nonadec-9-enoate

C51H90O6 (798.673704)

(2-heptadecanoyloxy-3-tridecanoyloxypropyl) (6Z,9Z,12Z,15Z)-octadeca-6,9,12,15-tetraenoate

C51H90O6 (798.673704)

[2-[(Z)-pentadec-9-enoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (9Z,12Z)-nonadeca-9,12-dienoate

C51H90O6 (798.673704)

[2-[(Z)-pentadec-9-enoyl]oxy-3-undecanoyloxypropyl] (10Z,13Z,16Z)-docosa-10,13,16-trienoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (Z)-octadec-9-enoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-heptadeca-9,12-dienoyl]oxy-3-tetradecanoyloxypropyl] (9Z,12Z)-heptadeca-9,12-dienoate

C51H90O6 (798.673704)

[2-[(Z)-hexadec-9-enoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (9Z,12Z)-octadeca-9,12-dienoate

C51H90O6 (798.673704)

[3-decanoyloxy-2-[(Z)-hexadec-9-enoyl]oxypropyl] (10Z,13Z,16Z)-docosa-10,13,16-trienoate

C51H90O6 (798.673704)

[2-hexadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (9Z,12Z,15Z)-octadeca-9,12,15-trienoate

C51H90O6 (798.673704)

[2-[(Z)-hexadec-9-enoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (9Z,12Z)-nonadeca-9,12-dienoate

C51H90O6 (798.673704)

[2-[(Z)-pentadec-9-enoyl]oxy-3-[(Z)-tridec-9-enoyl]oxypropyl] (11Z,14Z)-icosa-11,14-dienoate

C51H90O6 (798.673704)

[2-[(Z)-hexadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] (9Z,12Z,15Z)-octadeca-9,12,15-trienoate

C51H90O6 (798.673704)

(2-pentadecanoyloxy-3-undecanoyloxypropyl) (10Z,13Z,16Z,19Z)-docosa-10,13,16,19-tetraenoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(4Z,7Z,10Z,13Z)-hexadeca-4,7,10,13-tetraenoyl]oxypropyl] icosanoate

C51H90O6 (798.673704)

2,3-di(dodecanoyloxy)propyl (12Z,15Z,18Z,21Z)-tetracosa-12,15,18,21-tetraenoate

C51H90O6 (798.673704)

[2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxy-3-pentadecanoyloxypropyl] (Z)-heptadec-9-enoate

C51H90O6 (798.673704)

[2-[(4Z,7Z,10Z,13Z)-hexadeca-4,7,10,13-tetraenoyl]oxy-3-pentadecanoyloxypropyl] heptadecanoate

C51H90O6 (798.673704)

2,3-bis[[(Z)-pentadec-9-enoyl]oxy]propyl (9Z,12Z)-octadeca-9,12-dienoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-pentadecanoyloxypropyl] (9Z,12Z)-heptadeca-9,12-dienoate

C51H90O6 (798.673704)

[3-pentadecanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] (9Z,12Z,15Z)-octadeca-9,12,15-trienoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] (Z)-heptadec-9-enoate

C51H90O6 (798.673704)

2,3-bis[[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy]propyl hexadecanoate

C51H90O6 (798.673704)

[2-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] heptadecanoate

C51H90O6 (798.673704)

[3-[(7Z,10Z,13Z)-hexadeca-7,10,13-trienoyl]oxy-2-[(Z)-hexadec-9-enoyl]oxypropyl] hexadecanoate

C51H90O6 (798.673704)

[3-[(9Z,12Z)-hexadeca-9,12-dienoyl]oxy-2-[(Z)-hexadec-9-enoyl]oxypropyl] (Z)-hexadec-9-enoate

C51H90O6 (798.673704)

2,3-di(pentadecanoyloxy)propyl (6Z,9Z,12Z,15Z)-octadeca-6,9,12,15-tetraenoate

C51H90O6 (798.673704)

[2-[(Z)-hexadec-9-enoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] (9Z,12Z)-heptadeca-9,12-dienoate

C51H90O6 (798.673704)

[2-hexadecanoyloxy-3-[(4Z,7Z,10Z,13Z)-hexadeca-4,7,10,13-tetraenoyl]oxypropyl] hexadecanoate

C51H90O6 (798.673704)

2,3-di(tridecanoyloxy)propyl (10Z,13Z,16Z,19Z)-docosa-10,13,16,19-tetraenoate

C51H90O6 (798.673704)

(3-dodecanoyloxy-2-tetradecanoyloxypropyl) (10Z,13Z,16Z,19Z)-docosa-10,13,16,19-tetraenoate

C51H90O6 (798.673704)

2,3-bis[[(Z)-tetradec-9-enoyl]oxy]propyl (11Z,14Z)-icosa-11,14-dienoate

C51H90O6 (798.673704)

[2-[(Z)-tetradec-9-enoyl]oxy-3-tridecanoyloxypropyl] (9Z,11Z,13Z)-henicosa-9,11,13-trienoate

C51H90O6 (798.673704)

[2-[(Z)-pentadec-9-enoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (7Z,9Z)-nonadeca-7,9-dienoate

C51H90O6 (798.673704)

[2-[(Z)-hexadec-7-enoyl]oxy-3-tetradecanoyloxypropyl] (11Z,13Z,15Z)-octadeca-11,13,15-trienoate

C51H90O6 (798.673704)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-[(Z)-octadec-11-enoyl]oxypropyl] (Z)-octadec-11-enoate

C51H90O6 (798.673704)

[3-tridecanoyloxy-2-[(Z)-tridec-8-enoyl]oxypropyl] (13Z,16Z,19Z)-docosa-13,16,19-trienoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] (14Z,16Z)-docosa-14,16-dienoate

C51H90O6 (798.673704)

[2-[(9Z,11Z,13Z)-hexadeca-9,11,13-trienoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] nonadecanoate

C51H90O6 (798.673704)

[2-[(9Z,11Z,13Z)-hexadeca-9,11,13-trienoyl]oxy-3-tridecanoyloxypropyl] (Z)-nonadec-9-enoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (Z)-icos-11-enoate

C51H90O6 (798.673704)

[2-[(Z)-hexadec-7-enoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] (11Z,14Z)-heptadeca-11,14-dienoate

C51H90O6 (798.673704)

[2-[(Z)-heptadec-7-enoyl]oxy-3-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] (Z)-heptadec-7-enoate

C51H90O6 (798.673704)

[2-[(5Z,8Z,11Z)-tetradeca-5,8,11-trienoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] henicosanoate

C51H90O6 (798.673704)

[2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (Z)-nonadec-9-enoate

C51H90O6 (798.673704)

[2-hexadecanoyloxy-3-[(7Z,9Z,11Z,13Z)-hexadeca-7,9,11,13-tetraenoyl]oxypropyl] hexadecanoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(8Z,11Z,14Z)-heptadeca-8,11,14-trienoyl]oxypropyl] (Z)-nonadec-9-enoate

C51H90O6 (798.673704)

[2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxy-3-tridecanoyloxypropyl] (7Z,9Z)-nonadeca-7,9-dienoate

C51H90O6 (798.673704)

[2-hexadecanoyloxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] (8Z,11Z,14Z)-heptadeca-8,11,14-trienoate

C51H90O6 (798.673704)

[1-[(8Z,11Z,14Z)-heptadeca-8,11,14-trienoyl]oxy-3-tetradecanoyloxypropan-2-yl] (Z)-heptadec-7-enoate

C51H90O6 (798.673704)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-tetradecanoyloxypropyl] (14Z,16Z)-docosa-14,16-dienoate

C51H90O6 (798.673704)

2,3-di(pentadecanoyloxy)propyl (9Z,11Z,13Z,15Z)-octadeca-9,11,13,15-tetraenoate

C51H90O6 (798.673704)

[2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxy-3-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxypropyl] heptadecanoate

C51H90O6 (798.673704)

[2-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (Z)-henicos-9-enoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxypropyl] (11Z,14Z)-icosa-11,14-dienoate

C51H90O6 (798.673704)

2,3-bis[[(Z)-pentadec-9-enoyl]oxy]propyl (10Z,12Z)-octadeca-10,12-dienoate

C51H90O6 (798.673704)

[2-[(Z)-heptadec-7-enoyl]oxy-3-tridecanoyloxypropyl] (11Z,13Z,15Z)-octadeca-11,13,15-trienoate

C51H90O6 (798.673704)

[2-heptadecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (11Z,13Z,15Z)-octadeca-11,13,15-trienoate

C51H90O6 (798.673704)

2,3-bis[[(9Z,12Z)-pentadeca-9,12-dienoyl]oxy]propyl octadecanoate

C51H90O6 (798.673704)

[2-pentadecanoyloxy-3-[(5Z,8Z,11Z)-tetradeca-5,8,11-trienoyl]oxypropyl] (Z)-nonadec-9-enoate

C51H90O6 (798.673704)

[1-[(3Z,6Z,9Z)-dodeca-3,6,9-trienoyl]oxy-3-[(Z)-octadec-11-enoyl]oxypropan-2-yl] octadecanoate

C51H90O6 (798.673704)

[2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxy-3-tetradecanoyloxypropyl] (10Z,12Z)-octadeca-10,12-dienoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] (9Z,11Z)-henicosa-9,11-dienoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxypropyl] (Z)-henicos-9-enoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-tetradecanoyloxypropyl] (13Z,16Z,19Z)-docosa-13,16,19-trienoate

C51H90O6 (798.673704)

[2-[(Z)-hexadec-7-enoyl]oxy-3-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] (Z)-octadec-11-enoate

C51H90O6 (798.673704)

[2-pentadecanoyloxy-3-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] (7Z,9Z)-nonadeca-7,9-dienoate

C51H90O6 (798.673704)

[3-[(9Z,11Z,13Z)-hexadeca-9,11,13-trienoyl]oxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] hexadecanoate

C51H90O6 (798.673704)

[2-[(9Z,11Z,13Z)-hexadeca-9,11,13-trienoyl]oxy-3-pentadecanoyloxypropyl] (Z)-heptadec-7-enoate

C51H90O6 (798.673704)

[2-[(Z)-hexadec-7-enoyl]oxy-3-[(5Z,8Z,11Z)-tetradeca-5,8,11-trienoyl]oxypropyl] octadecanoate

C51H90O6 (798.673704)

[3-pentadecanoyloxy-2-[(6Z,9Z,12Z)-pentadeca-6,9,12-trienoyl]oxypropyl] (Z)-octadec-11-enoate

C51H90O6 (798.673704)

[2-[(Z)-pentadec-9-enoyl]oxy-3-tetradecanoyloxypropyl] (10Z,13Z,16Z)-nonadeca-10,13,16-trienoate

C51H90O6 (798.673704)

[2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxy-3-pentadecanoyloxypropyl] (11Z,14Z)-heptadeca-11,14-dienoate

C51H90O6 (798.673704)

(2-tetradecanoyloxy-3-tridecanoyloxypropyl) (9Z,11Z,13Z,15Z)-henicosa-9,11,13,15-tetraenoate

C51H90O6 (798.673704)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxypropyl] icosanoate

C51H90O6 (798.673704)

[2-[(6Z,9Z,12Z)-pentadeca-6,9,12-trienoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] nonadecanoate

C51H90O6 (798.673704)

(2-pentadecanoyloxy-3-tetradecanoyloxypropyl) (7Z,10Z,13Z,16Z)-nonadeca-7,10,13,16-tetraenoate

C51H90O6 (798.673704)

[2-[(Z)-pentadec-9-enoyl]oxy-3-[(5Z,8Z,11Z)-tetradeca-5,8,11-trienoyl]oxypropyl] nonadecanoate

C51H90O6 (798.673704)

[1-dodecanoyloxy-3-[(11Z,13Z,15Z)-octadeca-11,13,15-trienoyl]oxypropan-2-yl] (Z)-octadec-11-enoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxypropyl] (Z)-icos-11-enoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxy-3-tetradecanoyloxypropyl] (7Z,9Z)-nonadeca-7,9-dienoate

C51H90O6 (798.673704)

[2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-octadec-11-enoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-[(5Z,8Z,11Z)-tetradeca-5,8,11-trienoyl]oxypropyl] docosanoate

C51H90O6 (798.673704)

[1-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropan-2-yl] (Z)-heptadec-7-enoate

C51H90O6 (798.673704)

[2-hexadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (11Z,13Z,15Z)-octadeca-11,13,15-trienoate

C51H90O6 (798.673704)

[2-[(7Z,9Z,11Z,13Z)-hexadeca-7,9,11,13-tetraenoyl]oxy-3-tetradecanoyloxypropyl] octadecanoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-[(Z)-heptadec-7-enoyl]oxypropyl] (7Z,9Z)-nonadeca-7,9-dienoate

C51H90O6 (798.673704)

(3-dodecanoyloxy-2-pentadecanoyloxypropyl) (9Z,11Z,13Z,15Z)-henicosa-9,11,13,15-tetraenoate

C51H90O6 (798.673704)

[3-[(3Z,6Z,9Z)-dodeca-3,6,9-trienoyl]oxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] docosanoate

C51H90O6 (798.673704)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-pentadecanoyloxypropyl] (9Z,11Z)-henicosa-9,11-dienoate

C51H90O6 (798.673704)

[2-[(6Z,9Z,12Z)-pentadeca-6,9,12-trienoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] icosanoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (13Z,16Z,19Z)-docosa-13,16,19-trienoate

C51H90O6 (798.673704)

[1-[(Z)-dodec-5-enoyl]oxy-3-[(10Z,12Z)-octadeca-10,12-dienoyl]oxypropan-2-yl] (Z)-octadec-11-enoate

C51H90O6 (798.673704)

2,3-di(tetradecanoyloxy)propyl (5Z,8Z,11Z,14Z)-icosa-5,8,11,14-tetraenoate

C51H90O6 (798.673704)

[3-[(3Z,6Z,9Z)-dodeca-3,6,9-trienoyl]oxy-2-hexadecanoyloxypropyl] (Z)-icos-11-enoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-hexadecanoyloxypropyl] (5Z,8Z,11Z)-icosa-5,8,11-trienoate

C51H90O6 (798.673704)

[3-[(3Z,6Z,9Z)-dodeca-3,6,9-trienoyl]oxy-2-[(Z)-heptadec-7-enoyl]oxypropyl] nonadecanoate

C51H90O6 (798.673704)

[1-[(Z)-dodec-5-enoyl]oxy-3-[(11Z,13Z,15Z)-octadeca-11,13,15-trienoyl]oxypropan-2-yl] octadecanoate

C51H90O6 (798.673704)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxypropyl] nonadecanoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(9Z,11Z,13Z)-hexadeca-9,11,13-trienoyl]oxypropyl] (Z)-icos-11-enoate

C51H90O6 (798.673704)

(2-heptadecanoyloxy-3-tridecanoyloxypropyl) (9Z,11Z,13Z,15Z)-octadeca-9,11,13,15-tetraenoate

C51H90O6 (798.673704)

[2-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxy-3-tridecanoyloxypropyl] (10Z,12Z)-octadeca-10,12-dienoate

C51H90O6 (798.673704)

[2-pentadecanoyloxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (10Z,13Z,16Z)-nonadeca-10,13,16-trienoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] (5Z,8Z,11Z)-icosa-5,8,11-trienoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(10Z,12Z)-octadeca-10,12-dienoyl]oxypropyl] (10Z,12Z)-octadeca-10,12-dienoate

C51H90O6 (798.673704)

[2-[(9Z,11Z,13Z)-hexadeca-9,11,13-trienoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] heptadecanoate

C51H90O6 (798.673704)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-[(Z)-heptadec-7-enoyl]oxypropyl] (Z)-nonadec-9-enoate

C51H90O6 (798.673704)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxypropyl] henicosanoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(7Z,9Z,11Z,13Z)-hexadeca-7,9,11,13-tetraenoyl]oxypropyl] icosanoate

C51H90O6 (798.673704)

[3-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] (Z)-hexadec-7-enoate

C51H90O6 (798.673704)

[2-[(Z)-hexadec-7-enoyl]oxy-3-tridecanoyloxypropyl] (10Z,13Z,16Z)-nonadeca-10,13,16-trienoate

C51H90O6 (798.673704)

[2-[(6Z,9Z,12Z)-pentadeca-6,9,12-trienoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] octadecanoate

C51H90O6 (798.673704)

[1-[(Z)-heptadec-7-enoyl]oxy-3-[(5Z,8Z,11Z)-tetradeca-5,8,11-trienoyl]oxypropan-2-yl] heptadecanoate

C51H90O6 (798.673704)

[2-[(7Z,9Z,11Z,13Z)-hexadeca-7,9,11,13-tetraenoyl]oxy-3-pentadecanoyloxypropyl] heptadecanoate

C51H90O6 (798.673704)

[2-[(7Z,9Z,11Z,13Z)-hexadeca-7,9,11,13-tetraenoyl]oxy-3-tridecanoyloxypropyl] nonadecanoate

C51H90O6 (798.673704)

[2-[(Z)-hexadec-7-enoyl]oxy-3-[(6Z,9Z,12Z)-pentadeca-6,9,12-trienoyl]oxypropyl] heptadecanoate

C51H90O6 (798.673704)

[3-pentadecanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] (11Z,13Z,15Z)-octadeca-11,13,15-trienoate

C51H90O6 (798.673704)

[2-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxy-3-tetradecanoyloxypropyl] (11Z,14Z)-heptadeca-11,14-dienoate

C51H90O6 (798.673704)

[2-pentadecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (5Z,8Z,11Z)-icosa-5,8,11-trienoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(Z)-heptadec-7-enoyl]oxypropyl] (10Z,13Z,16Z)-nonadeca-10,13,16-trienoate

C51H90O6 (798.673704)

[2-[(5Z,8Z,11Z)-tetradeca-5,8,11-trienoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] icosanoate

C51H90O6 (798.673704)

[2-hexadecanoyloxy-3-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] (10Z,12Z)-octadeca-10,12-dienoate

C51H90O6 (798.673704)

[2-[(6Z,9Z,12Z)-pentadeca-6,9,12-trienoyl]oxy-3-tridecanoyloxypropyl] (Z)-icos-11-enoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxy-3-pentadecanoyloxypropyl] (10Z,12Z)-octadeca-10,12-dienoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (14Z,16Z)-docosa-14,16-dienoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxypropyl] (9Z,11Z)-henicosa-9,11-dienoate

C51H90O6 (798.673704)

[2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] (Z)-heptadec-7-enoate

C51H90O6 (798.673704)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-hexadecanoyloxypropyl] (11Z,14Z)-icosa-11,14-dienoate

C51H90O6 (798.673704)

[2-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (Z)-octadec-11-enoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(5Z,8Z,11Z)-tetradeca-5,8,11-trienoyl]oxypropyl] (Z)-docos-11-enoate

C51H90O6 (798.673704)

[2-[(8Z,11Z,14Z)-heptadeca-8,11,14-trienoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] octadecanoate

C51H90O6 (798.673704)

[2-hexadecanoyloxy-3-[(6Z,9Z,12Z)-pentadeca-6,9,12-trienoyl]oxypropyl] (Z)-heptadec-7-enoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] (11Z,14Z)-icosa-11,14-dienoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxy-3-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] nonadecanoate

C51H90O6 (798.673704)

[1-[(8Z,11Z,14Z)-heptadeca-8,11,14-trienoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropan-2-yl] heptadecanoate

C51H90O6 (798.673704)

2,3-bis[[(7Z,9Z)-tetradeca-7,9-dienoyl]oxy]propyl icosanoate

C51H90O6 (798.673704)

[3-[(3Z,6Z,9Z)-dodeca-3,6,9-trienoyl]oxy-2-pentadecanoyloxypropyl] (Z)-henicos-9-enoate

C51H90O6 (798.673704)

[3-[(3Z,6Z,9Z)-dodeca-3,6,9-trienoyl]oxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] henicosanoate

C51H90O6 (798.673704)

[2-[(Z)-hexadec-7-enoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (7Z,9Z)-nonadeca-7,9-dienoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxy-3-tridecanoyloxypropyl] (11Z,14Z)-icosa-11,14-dienoate

C51H90O6 (798.673704)

[2-[(Z)-heptadec-7-enoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (10Z,12Z)-octadeca-10,12-dienoate

C51H90O6 (798.673704)

[2-hexadecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (10Z,13Z,16Z)-nonadeca-10,13,16-trienoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-[(8Z,11Z,14Z)-heptadeca-8,11,14-trienoyl]oxypropyl] nonadecanoate

C51H90O6 (798.673704)

2,3-bis[[(Z)-tridec-8-enoyl]oxy]propyl (14Z,16Z)-docosa-14,16-dienoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxypropyl] (Z)-nonadec-9-enoate

C51H90O6 (798.673704)

[2-[(Z)-tetradec-9-enoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (9Z,11Z)-henicosa-9,11-dienoate

C51H90O6 (798.673704)

[2-[(Z)-pentadec-9-enoyl]oxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (11Z,14Z)-icosa-11,14-dienoate

C51H90O6 (798.673704)

(3-dodecanoyloxy-2-hexadecanoyloxypropyl) (5Z,8Z,11Z,14Z)-icosa-5,8,11,14-tetraenoate

C51H90O6 (798.673704)

[2-[(9Z,11Z,13Z)-hexadeca-9,11,13-trienoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] octadecanoate

C51H90O6 (798.673704)

(2-pentadecanoyloxy-3-tridecanoyloxypropyl) (5Z,8Z,11Z,14Z)-icosa-5,8,11,14-tetraenoate

C51H90O6 (798.673704)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] (Z)-henicos-9-enoate

C51H90O6 (798.673704)

(3-dodecanoyloxy-2-heptadecanoyloxypropyl) (7Z,10Z,13Z,16Z)-nonadeca-7,10,13,16-tetraenoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(6Z,9Z,12Z)-pentadeca-6,9,12-trienoyl]oxypropyl] (Z)-henicos-9-enoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxy-3-[(Z)-pentadec-9-enoyl]oxypropyl] (Z)-octadec-11-enoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-[(9Z,11Z,13Z)-hexadeca-9,11,13-trienoyl]oxypropyl] icosanoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-heptadecanoyloxypropyl] (10Z,13Z,16Z)-nonadeca-10,13,16-trienoate

C51H90O6 (798.673704)

[3-[(3Z,6Z,9Z)-dodeca-3,6,9-trienoyl]oxy-2-tetradecanoyloxypropyl] (Z)-docos-11-enoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxypropyl] (7Z,9Z)-nonadeca-7,9-dienoate

C51H90O6 (798.673704)

[2-[(5Z,8Z,11Z)-tetradeca-5,8,11-trienoyl]oxy-3-tridecanoyloxypropyl] (Z)-henicos-9-enoate

C51H90O6 (798.673704)

[2-[(Z)-hexadec-7-enoyl]oxy-3-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxypropyl] (Z)-heptadec-7-enoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-pentadecanoyloxypropyl] (9Z,11Z,13Z)-henicosa-9,11,13-trienoate

C51H90O6 (798.673704)

[3-tetradecanoyloxy-2-[(5Z,8Z,11Z)-tetradeca-5,8,11-trienoyl]oxypropyl] (Z)-icos-11-enoate

C51H90O6 (798.673704)

[1-dodecanoyloxy-3-[(9Z,11Z,13Z,15Z)-octadeca-9,11,13,15-tetraenoyl]oxypropan-2-yl] octadecanoate

C51H90O6 (798.673704)

[2-[(4Z,7Z)-hexadeca-4,7-dienoyl]oxy-3-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] octadecanoate

C51H90O6 (798.673704)

[3-[(3Z,6Z,9Z)-dodeca-3,6,9-trienoyl]oxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] icosanoate

C51H90O6 (798.673704)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-[(Z)-hexadec-7-enoyl]oxypropyl] (Z)-icos-11-enoate

C51H90O6 (798.673704)

[3-dodecanoyloxy-2-[(Z)-pentadec-9-enoyl]oxypropyl] (9Z,11Z,13Z)-henicosa-9,11,13-trienoate

C51H90O6 (798.673704)

[2-[(Z)-hexadec-7-enoyl]oxy-3-pentadecanoyloxypropyl] (8Z,11Z,14Z)-heptadeca-8,11,14-trienoate

C51H90O6 (798.673704)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-heptadecanoyloxypropyl] (7Z,9Z)-nonadeca-7,9-dienoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-[(6Z,9Z,12Z)-pentadeca-6,9,12-trienoyl]oxypropyl] henicosanoate

C51H90O6 (798.673704)

[2-hexadecanoyloxy-3-[(5Z,8Z,11Z)-tetradeca-5,8,11-trienoyl]oxypropyl] (Z)-octadec-11-enoate

C51H90O6 (798.673704)

[2-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxy-3-tetradecanoyloxypropyl] (11Z,14Z)-icosa-11,14-dienoate

C51H90O6 (798.673704)

(2-hexadecanoyloxy-3-tetradecanoyloxypropyl) (9Z,11Z,13Z,15Z)-octadeca-9,11,13,15-tetraenoate

C51H90O6 (798.673704)

[2-[(Z)-hexadec-7-enoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (10Z,12Z)-octadeca-10,12-dienoate

C51H90O6 (798.673704)

[3-[(3Z,6Z,9Z)-dodeca-3,6,9-trienoyl]oxy-2-heptadecanoyloxypropyl] (Z)-nonadec-9-enoate

C51H90O6 (798.673704)

[2-tetradecanoyloxy-3-[(Z)-tridec-8-enoyl]oxypropyl] (9Z,11Z,13Z)-henicosa-9,11,13-trienoate

C51H90O6 (798.673704)

[2-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxy-3-tridecanoyloxypropyl] (9Z,11Z)-henicosa-9,11-dienoate

C51H90O6 (798.673704)

[2-[(9Z,11Z,13Z)-hexadeca-9,11,13-trienoyl]oxy-3-tetradecanoyloxypropyl] (Z)-octadec-11-enoate

C51H90O6 (798.673704)

[2-[(Z)-pentadec-9-enoyl]oxy-3-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] (Z)-nonadec-9-enoate

C51H90O6 (798.673704)

(2-hexadecanoyloxy-3-tridecanoyloxypropyl) (7Z,10Z,13Z,16Z)-nonadeca-7,10,13,16-tetraenoate

C51H90O6 (798.673704)

[1-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-3-[(10Z,12Z)-octadeca-10,12-dienoyl]oxypropan-2-yl] octadecanoate

C51H90O6 (798.673704)

[3-[(Z)-dodec-5-enoyl]oxy-2-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] (Z)-docos-11-enoate

C51H90O6 (798.673704)

[2-[(8Z,11Z,14Z)-heptadeca-8,11,14-trienoyl]oxy-3-tridecanoyloxypropyl] (Z)-octadec-11-enoate

C51H90O6 (798.673704)

2,3-bis[[(4Z,7Z)-hexadeca-4,7-dienoyl]oxy]propyl hexadecanoate

C51H90O6 (798.673704)

[2-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-icos-11-enoate

C51H90O6 (798.673704)

[2-[(6Z,9Z,12Z)-pentadeca-6,9,12-trienoyl]oxy-3-tetradecanoyloxypropyl] (Z)-nonadec-9-enoate

C51H90O6 (798.673704)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-docos-11-enoate

C51H90O6 (798.673704)

[2-[(Z)-pentadec-9-enoyl]oxy-3-tridecanoyloxypropyl] (5Z,8Z,11Z)-icosa-5,8,11-trienoate

C51H90O6 (798.673704)

[3-tetradecanoyloxy-2-[(Z)-tetradec-9-enoyl]oxypropyl] (5Z,8Z,11Z)-icosa-5,8,11-trienoate

C51H90O6 (798.673704)

[2-hexadecanoyloxy-3-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxypropyl] (11Z,14Z)-heptadeca-11,14-dienoate

C51H90O6 (798.673704)

[3-[(6Z,9Z)-dodeca-6,9-dienoyl]oxy-2-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropyl] docosanoate

C51H90O6 (798.673704)

[1-[(11Z,14Z)-heptadeca-11,14-dienoyl]oxy-3-[(7Z,9Z)-tetradeca-7,9-dienoyl]oxypropan-2-yl] heptadecanoate

C51H90O6 (798.673704)

[2-[(9Z,12Z)-pentadeca-9,12-dienoyl]oxy-3-[(Z)-tetradec-9-enoyl]oxypropyl] (Z)-nonadec-9-enoate

C51H90O6 (798.673704)

[2-hexadecanoyloxy-3-[(7E,9E,11E,13E)-hexadeca-7,9,11,13-tetraenoyl]oxypropyl] hexadecanoate

C51H90O6 (798.673704)

[3-[(9E,11E,13E)-hexadeca-9,11,13-trienoyl]oxy-2-[(E)-hexadec-7-enoyl]oxypropyl] hexadecanoate

C51H90O6 (798.673704)

[3-[(4E,7E)-hexadeca-4,7-dienoyl]oxy-2-[(E)-hexadec-7-enoyl]oxypropyl] (E)-hexadec-7-enoate

C51H90O6 (798.673704)

2,3-bis[[(4E,7E)-hexadeca-4,7-dienoyl]oxy]propyl hexadecanoate

C51H90O6 (798.673704)

2-[Carboxy-(3-tetradecanoyloxy-2-tricosanoyloxypropoxy)methoxy]ethyl-trimethylazanium

C47H92NO8+ (798.6822572)

2-[Carboxy-(3-heptadecanoyloxy-2-icosanoyloxypropoxy)methoxy]ethyl-trimethylazanium

C47H92NO8+ (798.6822572)

2-[Carboxy-(2-henicosanoyloxy-3-hexadecanoyloxypropoxy)methoxy]ethyl-trimethylazanium

C47H92NO8+ (798.6822572)

2-[Carboxy-(3-decanoyloxy-2-heptacosanoyloxypropoxy)methoxy]ethyl-trimethylazanium

C47H92NO8+ (798.6822572)

2-[Carboxy-(2-docosanoyloxy-3-pentadecanoyloxypropoxy)methoxy]ethyl-trimethylazanium

C47H92NO8+ (798.6822572)

2-[Carboxy-(2-nonadecanoyloxy-3-octadecanoyloxypropoxy)methoxy]ethyl-trimethylazanium

C47H92NO8+ (798.6822572)

2-[Carboxy-(2-nonacosanoyloxy-3-octanoyloxypropoxy)methoxy]ethyl-trimethylazanium

C47H92NO8+ (798.6822572)

2-[Carboxy-(2-tetracosanoyloxy-3-tridecanoyloxypropoxy)methoxy]ethyl-trimethylazanium

C47H92NO8+ (798.6822572)

2-[Carboxy-(2-hexacosanoyloxy-3-undecanoyloxypropoxy)methoxy]ethyl-trimethylazanium

C47H92NO8+ (798.6822572)

2-[Carboxy-(3-nonanoyloxy-2-octacosanoyloxypropoxy)methoxy]ethyl-trimethylazanium

C47H92NO8+ (798.6822572)

2-[Carboxy-(3-dodecanoyloxy-2-pentacosanoyloxypropoxy)methoxy]ethyl-trimethylazanium

C47H92NO8+ (798.6822572)

triacylglycerol 48:4

C51H90O6 (798.673704)

A triglyceride in which the three acyl groups contain a total of 48 carbons and 4 double bonds.

NA-Orn 45:4

C50H90N2O5 (798.684937)

TG 10:0_14:0_24:4

C51H90O6 (798.673704)

TG 10:0_16:0_22:4

C51H90O6 (798.673704)

TG 10:0_18:0_20:4

C51H90O6 (798.673704)

TG 10:0_18:1_20:3

C51H90O6 (798.673704)

TG 10:0_18:2_20:2

C51H90O6 (798.673704)

TG 10:0_18:3_20:1

C51H90O6 (798.673704)

TG 10:0_18:4_20:0

C51H90O6 (798.673704)

TG 11:0_13:0_24:4

C51H90O6 (798.673704)

TG 11:0_15:0_22:4

C51H90O6 (798.673704)

TG 11:0_17:0_20:4

C51H90O6 (798.673704)

TG 11:0_17:1_20:3

C51H90O6 (798.673704)

TG 11:0_17:2_20:2

C51H90O6 (798.673704)

TG 11:0_18:4_19:0

C51H90O6 (798.673704)

TG 12:0_12:0_24:4

C51H90O6 (798.673704)

TG 12:0_14:0_22:4

C51H90O6 (798.673704)

TG 12:0_16:0_20:4

C51H90O6 (798.673704)

TG 12:0_16:1_20:3

C51H90O6 (798.673704)

TG 12:0_18:0_18:4

C51H90O6 (798.673704)

TG 12:0_18:1_18:3

C51H90O6 (798.673704)

TG 12:0_18:2_18:2

C51H90O6 (798.673704)

TG 12:1_18:1_18:2

C51H90O6 (798.673704)

TG 13:0_13:0_22:4

C51H90O6 (798.673704)

TG 13:0_15:0_20:4

C51H90O6 (798.673704)

TG 13:0_15:1_20:3

C51H90O6 (798.673704)

TG 13:0_17:0_18:4

C51H90O6 (798.673704)

TG 13:0_17:1_18:3

C51H90O6 (798.673704)

TG 13:0_17:2_18:2

C51H90O6 (798.673704)

TG 14:0_14:0_20:4

C51H90O6 (798.673704)

TG 14:0_14:1_20:3

C51H90O6 (798.673704)

TG 14:0_16:0_18:4

C51H90O6 (798.673704)

TG 14:0_16:1_18:3

C51H90O6 (798.673704)

TG 14:0_17:2_17:2

C51H90O6 (798.673704)

TG 14:1_14:1_20:2

C51H90O6 (798.673704)

TG 14:1_16:0_18:3

C51H90O6 (798.673704)

TG 14:1_16:1_18:2

C51H90O6 (798.673704)

TG 14:1_17:1_17:2

C51H90O6 (798.673704)

TG 14:2_16:0_18:2

C51H90O6 (798.673704)

TG 15:0_15:0_18:4

C51H90O6 (798.673704)

TG 15:0_15:1_18:3

C51H90O6 (798.673704)

TG 15:1_15:1_18:2

C51H90O6 (798.673704)

TG 15:1_16:1_17:2

C51H90O6 (798.673704)

TG 48:4_12:0

C51H90O6 (798.673704)

TG 48:4_14:0

C51H90O6 (798.673704)

TG 48:4_14:1

C51H90O6 (798.673704)

TG 48:4_15:0

C51H90O6 (798.673704)

TG 48:4_16:0

C51H90O6 (798.673704)

TG 48:4_16:1

C51H90O6 (798.673704)

TG 48:4_17:0

C51H90O6 (798.673704)

TG 48:4_18:0

C51H90O6 (798.673704)

TG 48:4_18:1

C51H90O6 (798.673704)

TG 48:4_18:2

C51H90O6 (798.673704)

TG 48:4_18:3

C51H90O6 (798.673704)

TG 48:4_20:0

C51H90O6 (798.673704)

TG 48:4_20:1

C51H90O6 (798.673704)

TG 48:4_20:2

C51H90O6 (798.673704)

TG 48:4_20:3

C51H90O6 (798.673704)

TG 48:4_20:4

C51H90O6 (798.673704)

TG 48:4_22:1

C51H90O6 (798.673704)